early HRA extrastimulation(passive) caused bythe conduction delay
early premature atrial stimulation at the HRA(passive) caused bythe conduction delay
structural damage to the white - matter fasciculi(passive) caused byconduction delays
disease distal to the His bundle(passive) caused byconduction delays
the right bundle branchcausinga delay in conduction
the left bundle branch block(passive) caused bythe interventricular conduction delay
Scar tissue , infiltration of abnormal tissue , electrolyte abnormality , ischemia , infarction and lots of thingscan causeintraventricular conduction delay
The depolarization wave can not access the right ventricle via the bundle branch , so it travels cell - to - cell across the right ventriclecausinga conduction delay
The depolarization wave can not access the right ventricle via the bundle branch , so the right ventricle travels cell - to - cell across the right ventriclecausinga conduction delay
extrinsic ( toxic ) factors that slow ventricular conduction , particularly hyperkalemia or drugs ( e.g. , class 1 antiarrhythmic agents , tricyclic antidepressants , phenothiazines(passive) also can be caused byIntraventricular conduction delays
Elevation of intracellular Ca2 +may causeconduction delays
These cable setscreateconduction timing delays
Vagal stimulation has also been shownto causeconduction delays ( 43,44
the defect itselfcausingdelayed conduction
Expansion of the extracellular matrix between cardiomyocytesmay causeconduction delays
in a uniform random distribution between 1 and 25 ms for long range connections and to 1 ms for all other connections(passive) were setConduction delays
all the tracts to neurons in affected spheres(passive) contributed byconduction delays
any number of things(passive) can be caused byDelayed conduction
the LVH(passive) caused bya conduction delay
preprocessing in the afferent visual pathway(passive) caused byconduction delays
disease distal to the Deshmukh et al(passive) caused byconduction delays
an anatomical block(passive) caused bydelayed conduction
Implantation of TAVR valves can cause pressure from the valve against the septal wall of the heartcausingconduction delays
interruption of a branch of the conduction system , which may alter the initial electrical vectors leading(passive) caused bya conduction delay
an infarct related scar and that functional conduction block does not play a major role in determining ventricular tachycardia circuits(passive) are caused bydelays in conduction
this siteresultingin conduction delay
Desmosomal dysfunction may lead to mechanical and thereafter electrical uncouplingultimately resultingin conduction delay
a demyelinating lesion affecting the cuneate tract of the spinal cord at cervical level(passive) caused byconduction delay
hemodynamic lesions or arrhythmiacausingconduction delay
In cases in which the stimulation CL is shorter than the shortest functional refractory period , a stimulus will reach the reentrant circuit when it is relatively refractoryresultingin conduction delay
to asynchronous contraction patterns and impaired hemodynamic performancecontributeto asynchronous contraction patterns and impaired hemodynamic performance
from said stimulus to a second location in the bodyresultingfrom said stimulus to a second location in the body
The prolonged PR - interval(passive) is caused byThe prolonged PR - interval
in the PR interval prolongationresultingin the PR interval prolongation
the wide QRS complex occurs in the mid to terminal part of the QRScausingthe wide QRS complex occurs in the mid to terminal part of the QRS
pipeline forex wide QRS complex , Teaching , and Learning Learning and Pursuit of Knowledge Students are expected to learn questioning the avail- able wisdom and the established ways for problem solving and to be prepared for indeterminate work and other life tasksusually causespipeline forex wide QRS complex , Teaching , and Learning Learning and Pursuit of Knowledge Students are expected to learn questioning the avail- able wisdom and the established ways for problem solving and to be prepared for indeterminate work and other life tasks
in a postpacing pause longer than basal flutter CLresultedin a postpacing pause longer than basal flutter CL
= 120 ms(passive) caused by= 120 ms
a substrate for re - entrant arrhythmias such as ventricular tachycardia ( 2can also createa substrate for re - entrant arrhythmias such as ventricular tachycardia ( 2
to dispersion of refractory periodsleadto dispersion of refractory periods
to the detection of temporal delays important for direction selectivity in the visual systemcould contributeto the detection of temporal delays important for direction selectivity in the visual system
to a slight phase shift between electrical signals from two neurons , denoted by different colored circlesleadingto a slight phase shift between electrical signals from two neurons , denoted by different colored circles
to delayed activation of the lateral wallwill leadto delayed activation of the lateral wall
abnormal ventricular contraction causing dyssynchrony b / t the rt and lt ventricles resulting in reduced systolic functioncausingabnormal ventricular contraction causing dyssynchrony b / t the rt and lt ventricles resulting in reduced systolic function
PR prolongation and high - degree A - V blockage in EKGcan also causePR prolongation and high - degree A - V blockage in EKG
from sinus or atrioventricular node dysfunction which causes impaired transmission of the electrical impulse and can [ ncbi.nlm.nih.gov ] Abnormal heart rhythms ( ventricular tachycardia , Wolff - Parkinson - White syndrome , long QT syndrome , sinus node dysfunction , atrioventricular block , catecholemanergic polymorphic [ cincinnatichildrens.orgresultingfrom sinus or atrioventricular node dysfunction which causes impaired transmission of the electrical impulse and can [ ncbi.nlm.nih.gov ] Abnormal heart rhythms ( ventricular tachycardia , Wolff - Parkinson - White syndrome , long QT syndrome , sinus node dysfunction , atrioventricular block , catecholemanergic polymorphic [ cincinnatichildrens.org
the conditions needed for arrhythmias.12,13may createthe conditions needed for arrhythmias.12,13
from sinus or atrioventricular node dysfunction which causes impaired transmission of the electrical impulse and can [ ceemjournal.org ] Significant bradycardia ( such as from sinus node dysfunction or atrioventricular ( AV ) nodal conduction disease ) and significant tachycardia ( from either a supraventricular [ clevelandclinicmeded.comresultingfrom sinus or atrioventricular node dysfunction which causes impaired transmission of the electrical impulse and can [ ceemjournal.org ] Significant bradycardia ( such as from sinus node dysfunction or atrioventricular ( AV ) nodal conduction disease ) and significant tachycardia ( from either a supraventricular [ clevelandclinicmeded.com
to ventricular tachycardia caused by macroreentry in the His - Purkinje systemmay leadto ventricular tachycardia caused by macroreentry in the His - Purkinje system
a slow heart ratecan causea slow heart rate
in abnormal electrical depolarisation of the heart and mechanical asynchrony of the ventricles.8–14 Dataresultin abnormal electrical depolarisation of the heart and mechanical asynchrony of the ventricles.8–14 Data
in a wide variety of AV conduction blockscan resultin a wide variety of AV conduction blocks
from reduced sodium current in the region that is activated intrinsically late in the activation sequence at baseline , i.e. , the lateral regionresultingfrom reduced sodium current in the region that is activated intrinsically late in the activation sequence at baseline , i.e. , the lateral region
from the more caudal recording positions of dlas compared with CPG neuronsresultingfrom the more caudal recording positions of dlas compared with CPG neurons
from sinus or atrioventricular node dysfunction which causes impaired transmission of the electrical impulse and can result in heart blocksresultingfrom sinus or atrioventricular node dysfunction which causes impaired transmission of the electrical impulse and can result in heart blocks
in propagated mEC gamma occurring at a different theta phase in CA1 ... as is the case in vivowould resultin propagated mEC gamma occurring at a different theta phase in CA1 ... as is the case in vivo
to wave break and enable rotor formation or anchoring ( 22might leadto wave break and enable rotor formation or anchoring ( 22
a dyssynchronous contraction pattern(passive) caused bya dyssynchronous contraction pattern
in ventricular dysrhythmiaresultingin ventricular dysrhythmia
to lines of functional block , which can produce arrhythmias through initiation of re - entrant circuitsleadsto lines of functional block , which can produce arrhythmias through initiation of re - entrant circuits
the LV free wall(passive) caused bythe LV free wall
disparities in tissue excitability and recovery thereby increasing the risk ofalso createsdisparities in tissue excitability and recovery thereby increasing the risk of
to dyssynchrony of cardiac contraction andmay leadto dyssynchrony of cardiac contraction and