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clamping the muscular bundle of fibers that connectscauseda significant conduction delay
clamping the muscular bundle of fibers that connects the atriacauseda significant conduction delay
by bundle branch block(passive) caused bythe delay in electrical conduction
Tissue death in bundle branchesleadsto permanent conduction delay
Intravenous adenosine , procainamide and verapamilcausedconduction delay or block
by the elevated level of extracellular potassium(passive) caused bycardiac conduction delay
by a myocardial scar(passive) caused bya ventricular conduction delay
AV block - fingolimodmay causeAV conduction delays
potential durationresultingin myocardial conduction delay
by the AV - node D.(passive) caused byC. Conduction delay
tissue discontinuities(passive) can also be caused byConduction delay in infarcted myocardium
disease distal to the His bundle(passive) caused byconduction delays
by the early activation(passive) is caused bythe conduction delay
insufficientto leadto conduction delay
Expansion of the extracellular matrix between cardiomyocytesmay causeconduction delays
by pacing(passive) caused bya conduction delay
Rapid pacingcausedconduction delay
the refractory periodcausingconduction delay
Sometimes medicationscan causeconduction delay
by structural damage to the white - matter fasciculi(passive) caused byconduction delays
structural damage to the white - matter fasciculi(passive) caused byconduction delays
the zener(passive) caused byThe delay in conduction
by the zener(passive) caused byThe delay in conduction
it may be induced by surgery through hemodynamic lesions orcausingconduction delay
by the AV - node D. Wave of depolarization(passive) caused byConduction delay
With a small AP , a slow CV at the vicinity of the stimulation site was observed ,resultingin a conduction delay
preprocessing in the afferent visual pathway(passive) caused byconduction delays
various factorsinfluencingconduction time
hemodynamic lesions or arrhythmiacausingconduction delay
it travels cell - to - cell across the right ventriclecausinga conduction delay
The depolarization wave can not access the right ventricle via the bundle branch , so it travels cell - to - cell across the right ventriclecausinga conduction delay
a demyelinating lesion affecting the cuneate tract of the spinal cord at cervical level(passive) caused byconduction delay
body temperatureinfluencesconduction speed
tissue anisotropy(passive) is caused byslow conduction
the LA fibrosiscausedslow conduction
INa ( that is , residual INacausingslow conduction
the defect itselfcausingdelayed conduction
the His ventricular bundlecausingdelayed conduction
class I antiarrhythmic drugs : antiarrhythmic and proarrhythmic effects(passive) caused byconduction slowing
by class I antiarrhythmic drugs : antiarrhythmic and proarrhythmic(passive) caused byconduction slowing
to heart block in patientsleadingto heart block in patients
to arrhythmiascan leadto arrhythmias
to heart blockleadingto heart block
to myocardial infarction and strokeleadingto myocardial infarction and stroke
to myocardial infarction and strokeleadingto myocardial infarction and stroke
inhibition rather than excitation to favor synchrony in hybrid neuronal networks of the entorhinal cortexcauseinhibition rather than excitation to favor synchrony in hybrid neuronal networks of the entorhinal cortex
delayed activation of the lateral wall of the left ventricle ( LVcausedelayed activation of the lateral wall of the left ventricle ( LV
in prolongation of the QRS complex and the PR / QT intervalsresultingin prolongation of the QRS complex and the PR / QT intervals
in asynchronousresultingin asynchronous
to conduction blockeven leadingto conduction block
to wave break andmight leadto wave break and
a slow heart ratecan causea slow heart rate
to dyssynchronyleadsto dyssynchrony
heart failure and dyssynchrony(passive) caused byheart failure and dyssynchrony
bradycardia and heart losscausingbradycardia and heart loss
PR prolongation and high - degree A - V blockage in EKGcan also causePR prolongation and high - degree A - V blockage in EKG
to prolonged PR intervalsleadingto prolonged PR intervals
regional wall motion abnormalities in the absence of primary myocardial dysfunctioncan causeregional wall motion abnormalities in the absence of primary myocardial dysfunction
the delay between the P - wave and the QRScausingthe delay between the P - wave and the QRS
to abnormal heart rhythms called arrhythmiascan leadto abnormal heart rhythms called arrhythmias
to heart block.3sometimes leadingto heart block.3
to the perpetuation of the macroreentrant circuitcontributingto the perpetuation of the macroreentrant circuit
wavelength shortening , which in turn , promotes the stability of AF circuitscauseswavelength shortening , which in turn , promotes the stability of AF circuits
to the increasing stability of AF in structurally remodeled atriacontributesto the increasing stability of AF in structurally remodeled atria
early commencement of ventricular depolarizationcausesearly commencement of ventricular depolarization
complete heart blockscausingcomplete heart blocks
to a reentrant supraventricular tachycardia , which usually occurs along with enhanced atrioventricular conductioncan leadto a reentrant supraventricular tachycardia , which usually occurs along with enhanced atrioventricular conduction
in the ventricular myocardium versus conduction pathwaysoriginatingin the ventricular myocardium versus conduction pathways
to increased heatleadingto increased heat
to heartleadingto heart
This heat loss ... the transfer of heat through direct contact(passive) is caused byThis heat loss ... the transfer of heat through direct contact
the heatcausedthe heat
heatcan causeheat
irregular beatcausesirregular beat
heating and cooling loss(passive) caused byheating and cooling loss
from the atrium with variable P wave morphologyoriginatesfrom the atrium with variable P wave morphology
alsocan ... resultalso
alsocan ... leadalso
alsomay ... resultalso
depolarizationresultingdepolarization