Loading ...
of more than 25 different proteins produced by • c3b and c4b act as opsonins on the surface of microorganisms and promote(passive) is composedComplement system
of more than 25 different proteins produced by hepatocytes , macrophages and bacteria and subsequently interacts with masp and masp2(passive) is composedComplement system
of more than 25 different proteins produced by different tissues and cells including hepatocytes , macrophages and gut epithelial cells(passive) is composedComplement system
of more than 25 different proteins ( table 1 ) , produced by different tissues and cells including hepatocytes , macrophages and gut epithelial cells(passive) is composedcomplement system
of three different pathways(passive) is composedComplement system
of a series of soluble and membrane - associated proteins present in the blood , which play a role in host defence through the identification , opsonisation , and lysis of pathogenic targets [ 1–3(passive) is composedIntroductionThe complement system
Antigen - antibody complexescan triggercomplement system
nanomaterials in contact with human serum(passive) triggered bycomplement system
of over 20 different proteins produced by hepatocytes , macrophages and intestinal epithelial cells(passive) is composedComplement system
of over 30 proteins(passive) is composedComplement system
the nephrotic deposition of circulating immune complexes ( CIC ) formed by autoantibodies in SLE [ 2(passive) triggered bycomplement system
specific carbohydrate groups on microorganisms(passive) is triggered bycomplement system
primarily ... cell - bound immune complexes(passive) is triggered ... bycomplement system
A United States study ... is knownto causecomplement system overactivation
Serum Antibodies react to Foreign MHCtriggeringcomplement system
as the heat - sensitive effector component of serum in the late 19th century(passive) was first discoveredComplement system
first(passive) was ... discoveredComplement system
transport(passive) caused bycomplement system
virus and other factors(passive) caused bycomplement system
cytotoxicity because they lack the Fc regiontriggeredcytotoxicity because they lack the Fc region
to cell lysis and enhances phagocytosis and inflammationleadsto cell lysis and enhances phagocytosis and inflammation
to modulate the infectivity of susceptible TcI strains of Trypanosoma cruzi BACKGROUND Trypanosoma cruzicontributesto modulate the infectivity of susceptible TcI strains of Trypanosoma cruzi BACKGROUND Trypanosoma cruzi
tissue injury as a result of alteration of blood flow in kidney transplantationcausingtissue injury as a result of alteration of blood flow in kidney transplantation
RBC lysis , damaging kidneys , etccausingRBC lysis , damaging kidneys , etc
processes which increase susceptibility of cell to phagocytosis or lysis(passive) caused byprocesses which increase susceptibility of cell to phagocytosis or lysis
to inflammation by enhancing cytokine and chemokine production , leukocyte recruitment , edema , neuronal cell apoptosis , and BBB degradation.[95],[96 ] Complement proteinscould contributeto inflammation by enhancing cytokine and chemokine production , leukocyte recruitment , edema , neuronal cell apoptosis , and BBB degradation.[95],[96 ] Complement proteins
protein block ... called the membrane attack complex , capable of piercing the membranes into cells by certain bacteria ( lysiscreatesprotein block ... called the membrane attack complex , capable of piercing the membranes into cells by certain bacteria ( lysis
vasodiation and tissue damagecausingvasodiation and tissue damage
in autoimmune responseresultingin autoimmune response
in excess production of bradykinin and kallikreinresultingin excess production of bradykinin and kallikrein
Formation of antigen - antibody complexesresultFormation of antigen - antibody complexes
to the elimination of invading microorganisms , immune complexes and transformed cells from tissues and biologic fluidscontributesto the elimination of invading microorganisms , immune complexes and transformed cells from tissues and biologic fluids
three protease cascades known as the classical ... mannose binding lectin ( MBL ) and alternative pathwayscan triggerthree protease cascades known as the classical ... mannose binding lectin ( MBL ) and alternative pathways
of more than 25 different proteins produced by hepatocytes , macrophages and where the binding of one protein promotes the binding of the next protein in the cascadeis composedof more than 25 different proteins produced by hepatocytes , macrophages and where the binding of one protein promotes the binding of the next protein in the cascade
peritubular andcausingperitubular and
to vascular permeabilityleadsto vascular permeability
to their damage by membrane attack complex [ 171leadsto their damage by membrane attack complex [ 171
in hemolysis in vessels ( intra - vesselsresultin hemolysis in vessels ( intra - vessels
to lysisin all pathways by bindingmembrane - C5b recruits C6 , 7 , 8 , & 9leadsto lysisin all pathways by bindingmembrane - C5b recruits C6 , 7 , 8 , & 9
into the complement cascade which cause hole in the cells and leading to cell deathresultinginto the complement cascade which cause hole in the cells and leading to cell death
the release of numerous toxic free radicals that stop cell growthcausingthe release of numerous toxic free radicals that stop cell growth
to intravascular hemolysisleadingto intravascular hemolysis
to vascular injury and extravasation of erythrocytes.[1leadingto vascular injury and extravasation of erythrocytes.[1
a cytotoxicity activity(passive) caused bya cytotoxicity activity
recruitment of inflammatory cellscausesrecruitment of inflammatory cells
opsonization of microorganisms ( which makes their absorption by phagocytes easier ) , activation of leukocytes and their transferal to inflammatory tissuecausesopsonization of microorganisms ( which makes their absorption by phagocytes easier ) , activation of leukocytes and their transferal to inflammatory tissue
to allograft rejectioncontributesto allograft rejection
to the generation of chemotactic factors that attracts polymorphonuclear leukocytes into the cellular cavityleadingto the generation of chemotactic factors that attracts polymorphonuclear leukocytes into the cellular cavity
to endothelial damage and HUS ( 10 ) Journal of the American Society of Nephrology 2014leadingto endothelial damage and HUS ( 10 ) Journal of the American Society of Nephrology 2014
to uncontrolled complement activation and thrombotic microangiopathyleadingto uncontrolled complement activation and thrombotic microangiopathy
the functions of NMO patientsinfluencesthe functions of NMO patients
skin microbiomeinfluencesskin microbiome
recurrent infections refers to repeated infections that are due to disorders of the complement system , a part of the body 's immune systemcausingrecurrent infections refers to repeated infections that are due to disorders of the complement system , a part of the body 's immune system
to the production of C4 and its ultimate product , C4dleadsto the production of C4 and its ultimate product , C4d
recurrent Causes of Disorders of complement systemcausingrecurrent Causes of Disorders of complement system