They attract phagocytes to both foreign material and self cells d ) Binding of MBLs to a bacterial capsuletriggersthe complement cascade
during meningococcal infec- tion , Volume 3 , Third edition Chemical and Biochemical Reaction Engineering , and Control ( edited by J. 1998 , 42 , 211216(passive) is triggeredThe complement cascade
the binding of C1q to the antibodytriggeringthe complement cascade
the interaction of antibody with specific antigen(passive) is triggered classically byThe complement cascade
either the classical or alternative complement pathways(passive) triggered bycomplement cascade
IgG and IgM antibodies that have bound antigencan triggerthe complement cascade
the complement system ... antibody - mediated rejection ( AMR ) and formation of donor specific antibodies ( DSAtriggerthe complement cascade
although immune complexes formed by these antibodies failto triggerthe complement cascade
in which antibodies against red blood cell membranes are usedto triggerthe complement cascade
Sera from MMN patients with IgM GM1 antibodiestriggerthe complement cascade
Three distinct pathways - the classical , the lectin and the alternative pathwaytriggerthe complement cascade
the antibody formation to reach the zone of equivalence with the circulating antigensthus triggeringthe complement cascade
endothelial cells of recipient xenoreactive natural antibodies ... in turntriggersthe complement cascade
via three basic mecha - nisms : the classical , lectin , and alterna - tive pathways(passive) can be triggeredThe complement cascade
three activation pathways , the classical pathway ( CP ) , the lectin pathway ( LP ) and the alternate pathway ( AP(passive) is triggered byThe complement cascade
one of the three canonical activation pathways , the lectin ( LP ) , the classical ( CP ) , or the alternative pathway ( AP(passive) can be triggered byThe complement cascade
via three distinct pathways , the classical , lectin and alternative pathways , which converge at the third component of complement ( C3(passive) can be triggeredThe complement cascade
after adding a fresh and undiluted serum sample to an assay , with subsequent binding of complement components to mammalian antibodies(passive) can be triggeredThe complement cascade
three activation pathways , the classic pathway ( CP ) , the lectin pathway ( LP ) , and the alternative pathway ( AP ) , which converge in the central and most important step of complement activation : the formation of unstable protease complexes , called C3 convertases ( C3bBb in the AP and C4b2a in the CP / LP ) , that cleave C3 to generate the activated fragment , C3b(passive) is triggered byThe complement cascade
complementary recognition specificities and answer to danger signals through the activation of associated proteasestriggerthe complement cascade
the latter being able to cleave complement components C4 and C2thereby triggeringthe complement cascade
of proteins that are activated through cleavage by an upstream enzyme(passive) is composedThe complement cascade
of proteins that are activated through the partial cleavage by an upstream enzyme(passive) is composedThe complement cascade
CScan influencethe complement cascade
in motion(passive) is setThe complement cascade
One set of thesetriggersthe complement cascade
a chain reactiontriggersthe complement cascade
these changestriggerthe complement cascade
the capacityto triggerthe complement cascade
increased numbers of circulating tumor cells or amount of tumor cell debris in advanced - stage patientstriggerthe complement cascade
the abilities of CRP and L - ficolinto triggersubsequent complement cascade
an antigen ... a cell walltriggersthe " complement cascade
to formation of the membrane attack complex ( MAC , C5b - C9leadingto formation of the membrane attack complex ( MAC , C5b - C9
inflammation and formation of the membrane attack complexcausesinflammation and formation of the membrane attack complex
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in the formation of the membrane - attack complex ( C5b-9resultingin the formation of the membrane - attack complex ( C5b-9
in the formation of the membrane attack complex and subsequent target cell deathresultingin the formation of the membrane attack complex and subsequent target cell death
to cell lysis through CDCleadingto cell lysis through CDC
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in cell lysis 3resultingin cell lysis 3
target cell lysiscausestarget cell lysis
in the formation of a membrane attack complex that makes a hole within the cell membrane ... causing cell lysis and deathresultsin the formation of a membrane attack complex that makes a hole within the cell membrane ... causing cell lysis and death
to the direct killing of a pathogen via the formation of the membrane - attack complex on the pathogen surfacecan leadto the direct killing of a pathogen via the formation of the membrane - attack complex on the pathogen surface
to inflammation and bullaeleadingto inflammation and bullae
to membrane attack complex deposition on the bacterial surface [ 12 , 13leadingto membrane attack complex deposition on the bacterial surface [ 12 , 13
in the deposition of large numbers of C3b molecules on the surface of activating particles , enabling opsonisation and acute local inflammation.[3resultingin the deposition of large numbers of C3b molecules on the surface of activating particles , enabling opsonisation and acute local inflammation.[3
assembly of the membrane attack complex and ... subsequently , osmotic cell lysistriggersassembly of the membrane attack complex and ... subsequently , osmotic cell lysis
to the production of a membrane attack complexleadingto the production of a membrane attack complex
to inflammation and tissue damageleadsto inflammation and tissue damage
opsonization and agglutination , as well as cell lysiscausesopsonization and agglutination , as well as cell lysis
to a localized inflammation and destruction of the muscle membrane at the neuromuscular junction ( NMJleadsto a localized inflammation and destruction of the muscle membrane at the neuromuscular junction ( NMJ
in the formation of soluble membrane attack complex ( sMACresultingin the formation of soluble membrane attack complex ( sMAC
to the formation of a “ membrane attack complex ” that forms a pore in the membrane of the pathogen allowing water and ions to enter into the cell , causing the cell to lyseleadsto the formation of a “ membrane attack complex ” that forms a pore in the membrane of the pathogen allowing water and ions to enter into the cell , causing the cell to lyse
opsonizationand agglutination , as well as cell lysis by the formationofcausesopsonizationand agglutination , as well as cell lysis by the formationof
in a pro - inflammatory , pro - thrombotic and cytolytic cell lysisresultsin a pro - inflammatory , pro - thrombotic and cytolytic cell lysis
to the formation of membrane attack complexes and subsequent tumor cell lysisleadsto the formation of membrane attack complexes and subsequent tumor cell lysis
to inflammation with potential neurotoxic consequences ( Rogers et al . , Procleadingto inflammation with potential neurotoxic consequences ( Rogers et al . , Proc
to an activation of endothelial cells with increased expression of adhesion moleculesleadsto an activation of endothelial cells with increased expression of adhesion molecules
elimination of the " alien " cell by lysis and/or phagocytosis ( Sahu and Lambris 2001causingelimination of the " alien " cell by lysis and/or phagocytosis ( Sahu and Lambris 2001
to the formation binary options youtube hotel the cytolytic membrane attack complexleadingto the formation binary options youtube hotel the cytolytic membrane attack complex
to acute local inflammation at the site of depositionleadsto acute local inflammation at the site of deposition
up to the generation of membrane attack complex and C3a and C5a fragments that cause cellular infiltrationleadingup to the generation of membrane attack complex and C3a and C5a fragments that cause cellular infiltration
in acute intravascular haemolysisresultingin acute intravascular haemolysis
in intravascular haemolysis and haemoglobinuriaresultingin intravascular haemolysis and haemoglobinuria
a series of reactions that results in inflammation and cell deathsetsa series of reactions that results in inflammation and cell death
in inflammation ( 34–36 ) and interstitial fibrosis ( 37resultingin inflammation ( 34–36 ) and interstitial fibrosis ( 37
chronic inflammation and provide a 98 % solution for crippling joint pain and stiffness in just weekstriggerschronic inflammation and provide a 98 % solution for crippling joint pain and stiffness in just weeks
acute perivascular inflammation and symp- toms of small - vessel vasculitiscausingacute perivascular inflammation and symp- toms of small - vessel vasculitis
lung sterile inflammation and COPD progression are not completely understoodto causelung sterile inflammation and COPD progression are not completely understood
in cell lysis with the consequent release of proteolytic enzymes promoting the release of vasoactive effectors from platelets ( e.g. : bradykinine and histamineresultsin cell lysis with the consequent release of proteolytic enzymes promoting the release of vasoactive effectors from platelets ( e.g. : bradykinine and histamine
to intravascular hemolysisleadsto intravascular hemolysis
in extravascular hemolysisresultingin extravascular hemolysis