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chondroitin sulfate released by thrombin receptor - activated platelets J Thromb Haemost(passive) triggered byComplement activation
that CL-11 detects stress - induced L - fucose pattern on renal tubulesto triggercomplement activation
This antibody - antigen interactioncan causecomplement activation
the polysaccharide characteristics.\nCONCLUSIONS(passive) was influenced byComplement activation
Biomaterial Surfaces Mechanisms and Regulation 5(passive) Triggered byComplement Activation
The anti - C1s antibody TNT003preventscomplement activation
new insights into the major factorsinfluencecomplement activation
other substances , such as the surface components of a microorganism alonecould triggercomplement activation
The monoclonal antibody added to control serumpreventscomplement activation
at least two soluble aqueous humor factors(passive) is also prevented byComplement activation
antibody complexes , complexes are deposited in vessel walls and tissuescausingcomplement activation
via three pathways , the classical pathway ( CP ) , the lectin pathway ( LP ) , and the alternative pathway ( AP ) , each culminating in the formation of the surface - bound C3 convertases and C3b opsonization(passive) can be triggeredComplement activation
many factors , including how blood is collected and processed , the format in which the virus is presented ( fixed or soluble(passive) can be influenced byComplement activation
addition of purified CS to plasmatriggeredcomplement activation
Another proteininfluencescomplement activation
both HPG and PEGtriggeredcomplement activation
MBP.MASP-2 complexes(passive) triggered bycomplement activation
mannose - binding protein - MASP-2 complexes(passive) triggered bycomplement activation
the classical , lectin , and alternative pathways(passive) can be triggered byComplement activation
mannan binding lectin ( MBL(passive) triggered byComplement activation
The resulting immune complexescould triggercomplement activation
MASP-2 alone ... sufficientto triggercomplement activation
spontaneous alternative pathway hydrolysis(passive) caused bycomplement activation
the initial binding of the globular domains to the Fc(passive) is triggered byComplement activation
The underlying mechanismstriggercomplement activation
anti - PEG IgM(passive) caused bycomplement activation ,
Under conditions in which overwhelming complement activation takes place , these complement regulators may not sufficeto ... preventcomplement activation from occurring
This reaction ... thoseprovokedcomplement activation
heavy chain deposition ... sufficientto causecomplement activation
neutral dimyristoyl - phosphocholine ( DMPC ) liposomes in human plasma and whole - blood systems(passive) triggered bycomplement activation
the adaptive immune response with particlestriggercomplement activation
317 - 325 , http://dx.doi.org/10.1159/000125048 Li M ; Peake PW ; Charlesworth JA ; Tracey DJ ; Moalem - Taylor G , 2007contributesComplement activation
hemodialysis biomaterials(passive) triggered bycomplement activation
certain batchescausedcomplement activation
cobra venom ( 29preventcomplement activation
removal of the negative charge by methylation of the phosphate oxygen of lipid - PEG conjugatespreventedcomplement activation
this heparin coatingpreventscomplement activation
A ) Fluid regulatory protein C1-inhpreventscomplement activation
which was supplemented with EDTAto preventcomplement activation
the abilityto triggercomplement activation
to Severe Acute Respiratory Syndrome Coronavirus Pathogenesis 3Contributesto Severe Acute Respiratory Syndrome Coronavirus Pathogenesis 3
in an accelerated clearance via complement receptors in the liver ( extravascular hemolysis ) or leading to intravascular lysis of RBCsresultingin an accelerated clearance via complement receptors in the liver ( extravascular hemolysis ) or leading to intravascular lysis of RBCs
to glomerular injury and sclerosis in some patients with FSGScontributesto glomerular injury and sclerosis in some patients with FSGS
to glomerular and tubular injury in experimental FSGScontributesto glomerular and tubular injury in experimental FSGS
in the release of soluble bioactive fragments ( e.g. , C3a , C4a , C5a ) that stimulate mast cells , macrophages , and neutrophils during the inflammatory responsealso resultsin the release of soluble bioactive fragments ( e.g. , C3a , C4a , C5a ) that stimulate mast cells , macrophages , and neutrophils during the inflammatory response
to AAA via a similar mechanismcontributesto AAA via a similar mechanism
to the generation of a series of activation peptides that are capable of producing inflammationalso leadsto the generation of a series of activation peptides that are capable of producing inflammation
to cell lysis ( complement - dependent cytotoxicity [ CDCultimately leadingto cell lysis ( complement - dependent cytotoxicity [ CDC
in the formation of many biologically active complement fragments that act as anaphylatoxins , opsonins , or chemotactic factorsalso resultsin the formation of many biologically active complement fragments that act as anaphylatoxins , opsonins , or chemotactic factors
in a sequential triggering of subsequent componentsresultsin a sequential triggering of subsequent components
to the pathogenesis of acute PUUV infectionmay contributeto the pathogenesis of acute PUUV infection
to production of the membrane attack complex ( MAC ) , which forms pores in the cellular membrane resulting in cytolysisalso leadsto production of the membrane attack complex ( MAC ) , which forms pores in the cellular membrane resulting in cytolysis
to tumor growth and metastasis through different mechanismscan contributeto tumor growth and metastasis through different mechanisms
to this processcontributesto this process
xenograft rejectionto preventxenograft rejection
to loss of dopaminergic neurons in some individuals with PDmay contributeto loss of dopaminergic neurons in some individuals with PD
a simple integrated responsetriggersa simple integrated response
in the masking and removal of apoptotic cells exposing nuclear componentsresultsin the masking and removal of apoptotic cells exposing nuclear components
an inflammatory statecausingan inflammatory state
to opsonization and phagocytosisleadsto opsonization and phagocytosis
to a prothrombotic state through endothelial activation and apoptosis mediated by the release of tissue factor and other prothrombotic substancesto contributeto a prothrombotic state through endothelial activation and apoptosis mediated by the release of tissue factor and other prothrombotic substances
the release of inflammatory cytokines and therefore the activation of effector cells ( 44 , 45to triggerthe release of inflammatory cytokines and therefore the activation of effector cells ( 44 , 45
leakage of intravascular fluid in to serous spacecausingleakage of intravascular fluid in to serous space
in a loss of TER , which required transient membrane attack complex formation , activation of the alternative pathway , and VEGF secretion and signalingresultedin a loss of TER , which required transient membrane attack complex formation , activation of the alternative pathway , and VEGF secretion and signaling
in significant increase of C4a split productresultingin significant increase of C4a split product
in the influx of inflammatory cellsresultingin the influx of inflammatory cells
in cell lysis and inflammationresultingin cell lysis and inflammation
to cardiac autophagy and represents a centralized nexus for pro - fibrotic mediator production including RAGEleadsto cardiac autophagy and represents a centralized nexus for pro - fibrotic mediator production including RAGE
to hypoxic - ischemic brain injury in neonatal ratscontributesto hypoxic - ischemic brain injury in neonatal rats
in vesicle recognition by macrophage complement receptorsresultingin vesicle recognition by macrophage complement receptors
to podocyte damage inmay contributeto podocyte damage in
to angiogenesis with formation of new blood vesselsleadingto angiogenesis with formation of new blood vessels
to pre - eclamptic symptomscontributingto pre - eclamptic symptoms
to hypotensionleadsto hypotension
to formation of a peptide bond between the radioactive cysteine and the labile binding site on the α′ chain of C3bhas ledto formation of a peptide bond between the radioactive cysteine and the labile binding site on the α′ chain of C3b
to assembly of C5b-9leadsto assembly of C5b-9
to the injury and outcome of kidneycontributesto the injury and outcome of kidney
to podocyte injury in IgANmay contributeto podocyte injury in IgAN
to the ability of S. aureus to evade or inactivate host immune responses ( 28can contributeto the ability of S. aureus to evade or inactivate host immune responses ( 28
to opsonization of bacilli 25leadingto opsonization of bacilli 25