pro - inflammatory effects on the endothelial cellscausesatherosclerosis in type 2 diabetes mellitus ( T2DM
significant developments in antidiabetic treatmenthave influencedthe pharmacological treatment of type 2 diabetes mellitus ( T2DM
defects in the insulin secretory response from pancreatic β - cells and in insulin action on skeletal muscles , adipose tissues , and the liver(passive) is caused byHyperglycemia in type 2 diabetes mellitus ( T2DM
rising prevalence of the disease(passive) caused bytype 2 diabetes mellitus ( T2DM ) therapeutics
hypothalamic insulin resistancecan contributeto hyperglycemia in type 2 diabetes mellitus
Two specific point mutations and some deletions and duplications in the mitochondrial genomecan causetype 2 diabetes treatment of type 2 diabetes mellitus
hypothalamic resistin actioncan contributeto hyperglycemia in type 2 diabetes mellitus
either inadequate production of the hormone insulin or a lack of response to insulin by(passive) is caused byMellitus Type 2 Pathophysiology Type 2 diabetes
Can stresscausepharmacological treatment for type 2 diabetes
to FGF21 resistance in pancreatic islets ... probably through reduction of PPARγ expression , which provides a novel mechanism for glucose - mediated islet dysfunctionmay leadto FGF21 resistance in pancreatic islets ... probably through reduction of PPARγ expression , which provides a novel mechanism for glucose - mediated islet dysfunction
to increased Reactive Oxygen Species ( ROS ) generation which lowers mitochondrial biogenesis and causes mitochondrial dysfunction [ 1leadsto increased Reactive Oxygen Species ( ROS ) generation which lowers mitochondrial biogenesis and causes mitochondrial dysfunction [ 1
using our proprietary NuMimetic technologydiscoveredusing our proprietary NuMimetic technology
how to schedule the type 2 diabetes mellitus thesis fooddiscoverhow to schedule the type 2 diabetes mellitus thesis food
blindness riddle Difference between type1 and typecausesblindness riddle Difference between type1 and type
to the cardiovascular complications that account for much of the morbidity and mortality in the diseasecontribute significantlyto the cardiovascular complications that account for much of the morbidity and mortality in the disease
to a stronger stimulation of the IRs than NPH insulinleadsto a stronger stimulation of the IRs than NPH insulin
to bone fragility and predisposes to increased risk of fracture , poor bone healing and other skeletal complicationsleadsto bone fragility and predisposes to increased risk of fracture , poor bone healing and other skeletal complications