Loading ...
bone marrow failure(passive) caused byplatelets
pathology in the bone marrow(passive) caused byplatelets
damage to bone marrow(passive) caused byplatelets
low production of platelets in the bone marrow or increased destruction of platelets in blood vessels , spleen and liver hemosiderosis(passive) caused byplatelets
produced within the bone marrow break apartto createplatelets
other conditions such as tuberculosis or chemical suppression of bone marrow etc(passive) caused byplatelets
Growth Factors Growth factors enable the bone marrowto createplatelets
the mice ... anticoagulant drugs , aspirin and clopidogrelinfluenceplatelets
antiplatelet drugspreventplatelets
certain drugs(passive) can be caused byplatelets
platelet drugs like aspirinwill preventplatelets
Drugs like Aspirin and clopidogrel ( plavixpreventplatelets
your own body ’s blood(passive) created byplatelets
his body so it will startcreatingplatelets
that enables the bodyto createplatelets
Eptifibatide ... a platelet aggregation inhibitorpreventsplatelets
250 mg tabletpreventsplatelets
300 mg tabletpreventsplatelets
Aspirin and anti - inflammatoriespreventplatelets
Strengths Available for Plavix | PharmaWebCanada.to Plavix ( clopidogrelpreventsplatelets
too many cellscreateplatelets
a platelet aggregation inhibitor mainly used during and after coronary artery procedures like angioplastyto preventplatelets
a technology that mimics the way the human bodycreatesplatelets
diseases such as leukemia , viral infections as well as toxic chemicals and chemotherapy drugs(passive) can be caused byplatelets
Ginger , similar to garliccan preventplatelets
Harvard Heart Letter : January 2014 - Harvard Health Aspirinpreventsplatelets
Put patients on meds ( like Plavixto preventplatelets
• Flavanoids – antioxidants which protect from damage to the arteries , function like a low dose aspirinpreventingclumping platelets
a subset of those stem cells in mice that is particularly primedto createplatelets
DiarrhoeaClopidogrel(75 mg)UsesClopidogrel is used in the treatment of heart attackHow it worksClopidogrelpreventsplatelets
the UL VWF , which in turn leads to the development of microthrombi.2 mutations in the ADAMTS13 gene , which is located on chromosome 9q34 and codes for the metalloprotease ( Congenital TTP ) autoantibodies against ADAMTS13 , autoimmune diseases ( systemic lupus erythematosus , rheumatoid arthritis ) , infections ( particularly HIV ) , drugs ( ticlopidine , clopidogrel , cyclosporine A , interferon α , statins , mitomycin C , cisplatin , gemcitabine(passive) caused byplatelets
Involvement of other cell types , such as sinusoidal endothelial cells , Kupffer cells , and hepatic stellate cells , and recruitment of inflammatory cells as well asleadplatelets
blurred imagesresultingfrom clumped platelets
Antiplateletspreventplatelets
Antiplatelet agentspreventplatelets
the soluble crosslinked collagen - related peptide CRP - XL(passive) can ... be triggered byplatelets
• Anti - clotting medicinespreventplatelets
These treatmentspreventplatelets
parasites or immune diseases(passive) caused byplatelets
with depth features that are 60 % of the thickness of the platelet stock(passive) are typically designedPlatelets
to the formation of blood clots , which can cause a heart attackcan leadto the formation of blood clots , which can cause a heart attack
to the formation of blood clots , which in turn can cause a heart attackcan leadto the formation of blood clots , which in turn can cause a heart attack
IFN‐alpha / beta‐induced lethal hemorrhage promoting CTL‐dependent clearance of lymphocytic choriomeningitis viruspreventIFN‐alpha / beta‐induced lethal hemorrhage promoting CTL‐dependent clearance of lymphocytic choriomeningitis virus
to blood clots which can cause a heart attack.6.Chocolate also lowers your bad cholesterolcan leadto blood clots which can cause a heart attack.6.Chocolate also lowers your bad cholesterol
to the inflammatory response in the effector phase of EAEsignificantly contributeto the inflammatory response in the effector phase of EAE
to the formation of blood clots , which can cause all kinds of problems including an increased risk of a heart attackcan leadto the formation of blood clots , which can cause all kinds of problems including an increased risk of a heart attack
to hemostasis ( the process of stopping the bleeding of the wound or infected area of our bodycontributeto hemostasis ( the process of stopping the bleeding of the wound or infected area of our body
to aiding repair any kind of signs of destroyed body musclecontributeto aiding repair any kind of signs of destroyed body muscle
bleeding and form plugs that help stop bleeding after injury to a blood vesselpreventbleeding and form plugs that help stop bleeding after injury to a blood vessel
to the Pathogenesis of Experimental Autoimmune EncephalomyelitisNovelty and Significance Harald F. Langer , Eun Young Choi , Hong Zhou , Rebecca Schleicher , Kyoung - Jin Chung , Zhongshu Tang , Kerstin Göbel , Khalil Bdeir , Antonios Chatzigeorgiou , Connie Wong , Sumeena Bhatia , Michael J. Kruhlak , John W. Rose , James B. Burns , Kenneth E. Hill , Hongchang Qu , Yongqing Zhang , Elin Lehrmann , Kevin G. Becker , Yunmei Wang , Daniel I. Simon , Bernhard Nieswandt , John D. Lambris , Xuri Li , Sven G. Meuth , Paul Kubes and Triantafyllos Chavakis Circulation ResearchContributeto the Pathogenesis of Experimental Autoimmune EncephalomyelitisNovelty and Significance Harald F. Langer , Eun Young Choi , Hong Zhou , Rebecca Schleicher , Kyoung - Jin Chung , Zhongshu Tang , Kerstin Göbel , Khalil Bdeir , Antonios Chatzigeorgiou , Connie Wong , Sumeena Bhatia , Michael J. Kruhlak , John W. Rose , James B. Burns , Kenneth E. Hill , Hongchang Qu , Yongqing Zhang , Elin Lehrmann , Kevin G. Becker , Yunmei Wang , Daniel I. Simon , Bernhard Nieswandt , John D. Lambris , Xuri Li , Sven G. Meuth , Paul Kubes and Triantafyllos Chavakis Circulation Research
clottingcauseclotting
to cardiovascular events like heart attacksleadto cardiovascular events like heart attacks
in decreased number of platelets and depletion of its contents affecting platelet aggregationresultingin decreased number of platelets and depletion of its contents affecting platelet aggregation
strokesmay causestrokes
to cellular or organ injurycan contributeto cellular or organ injury
the increase of platelet derived growth factor , vasoendothelial growth factor and epithelial growth factor to the site of injurycausethe increase of platelet derived growth factor , vasoendothelial growth factor and epithelial growth factor to the site of injury
to amyloid - β aggregation in cerebral vesselscontributeto amyloid - β aggregation in cerebral vessels
excessive blood losspreventexcessive blood loss
to both coagulation and thrombolysiscontributeto both coagulation and thrombolysis
to vessel formationcontributeto vessel formation
to the pathogenesis of EAEcontributeto the pathogenesis of EAE
to the pathogenesis of experimental autoimmune encephalomyelitiscontributeto the pathogenesis of experimental autoimmune encephalomyelitis
to processes beyond thrombus formationcontributeto processes beyond thrombus formation
the plug(passive) created bythe plug
to postnatal occlusion of the ductus arteriosuscontributeto postnatal occlusion of the ductus arteriosus
to the thromboinflammatory responsecontributeto the thromboinflammatory response
IFN - α / β - induced lethal hemorrhage promoting CTL - dependent clearance of lymphocytic choriomeningitis viruspreventIFN - α / β - induced lethal hemorrhage promoting CTL - dependent clearance of lymphocytic choriomeningitis virus
to this pathologycontributeto this pathology
to histone - induced mortalitycontributeto histone - induced mortality
to the reduction of liver fibrosis in micecontributeto the reduction of liver fibrosis in mice
disruption of viral integrity.[174may then causedisruption of viral integrity.[174
to the development of vascular spasm through thromboxane A2 productionmay also contributeto the development of vascular spasm through thromboxane A2 production
to host defense by multiple mechanisms , including forming immune complexes and aggregates , shedding their granular content , and internalizing pathogens and subsequently being marked for removalgreatly contributeto host defense by multiple mechanisms , including forming immune complexes and aggregates , shedding their granular content , and internalizing pathogens and subsequently being marked for removal
tissue damagecan causetissue damage
marrow from megakaryocytes and are discoid anuclear cellsoriginatemarrow from megakaryocytes and are discoid anuclear cells
Ib deficiency adherence to exposed collagen von Willebrand factorsetIb deficiency adherence to exposed collagen von Willebrand factor