Loading ...

Blob

Smart Reasoning:

C&E

See more*

Qaagi - Book of Why

Causes

Effects

a main variant primes a person to develop a complex disorder such as autismsetsthe ' clinical features

mutations in the different genes(passive) caused bythe clinical features

various diseased factors , which mainly behaves as glomerular damage with increased permeability of glomerular basement membrane as well as(passive) caused byclinical features

variablesinfluencingclinical characteristics

Although acute butyrylcholinesterase inhibition does not seemto causeclinical features

various factors , which mainly behaves as increased glomerular basement membrane as well as declined GFR that patients suffer from increasingly serious diseased condition with four significant characters include heavy proteinuria , edema , hypoproteinemia and hyperlipemia(passive) caused byclinical features

the missense mutation and those by the nonsense mutation(passive) caused bythe clinical features

numerous diseased factors , which mainly behaves as glomerular damage of kidneys with increased permeability of glomerular basement membrane as well as declined GFR that patients suffer from increasingly deteriorative internal and external lesions from slight pathological and physiological insufficiency to severe parenchymal and functional dysfunctions(passive) caused byclinical features

paired t - test to nominal disturbed datais composedof clinical features

the missense mutation Y528D and the delection F646fs708X(passive) caused byThe clinical features

various diseased factors , which mainly behaves as glomerular damage of kidneys with increased permeability of glomerular basement membrane as well as declined GFR that patients suffer from increasingly serious internal and external lesions from slight pathological and physiological insufficiency to severe parenchymal and functional dysfunction attribute to varying diseased course(passive) caused byclinical features

various diseased factors , which mainly behaves as glomerular damage of increased glomerular basement membrane as well as declined GFR that patients suffer from four significant characteristics of heavy proteinuria , edema , hyperlipemia and hypoproteinemia(passive) caused byclinical features

various diseased factors , which mainly behaves as glomerular damage of kidneys with increased permeability of glomerular basement membrane as well as declined GFR that patients suffer from increasingly deteriorative internal and external damage from slight pathological and physiological changes to severe parenchymal and functional lesions according to numerous diseased causes , regions and degrees(passive) caused byclinical features

Gly222Glu mutation(passive) caused bythe clinical features

numerous factors , which behaves as pathological damage of increased permeability of glomerular basement membrane and declined GFR(passive) caused byclinical features

different pathophysiological effects(passive) caused byClinical features

various diseased factors , which mainly behaves as glomerular damage of increased glomerular basement membrane as well as declined GFR that patients suffer from increasingly serious internal and external lesions from slight pathological and physiological changes to severe parenchymal and functional damage according to varying diseased causes , regions and degrees(passive) caused byclinical features

repetitive episodes of obstructive apneas and hypopneas what we are usually talking about when we say a patient " has sleep apnea(passive) caused byclinical features

venoms of different species of snake “ Syndromic Approach(passive) caused byclinical features

mutations in a single gene , called autoimmune regulator ( AIRE(passive) is caused byClinical Features APS-1

these two neisserial species(passive) caused bythe clinical features

various diseased causes , which mainly behaves as glomerular lesions of kidneys with increased permeability of glomerular basement membrane as well as declined GFR(passive) caused byclinical features

various diseased factors , which mainly behaves as glomerular insufficiency and dysfunction with increased permeability of glomerular basement membrane as well as declined GFR(passive) caused byclinical features

loss of different segments of 14q(passive) caused byclinical features

different YE bio / serotypes(passive) caused bythe clinical features

mutations in CACNA1A.Objective To test the role of the SLC1A3 gene in EA.Design Genetic and functional studies(passive) is caused byclinical features

dominant negative mutations in STAT3(passive) caused byclinical features

hidden ) trisomy 16 mosaicism than by upd(16)mat itself(passive) are rather caused byclinical features

allergy to certain fruits and vegetables(passive) caused byclinical characteristics

injury and cell death usually of both upper motor neurons in the brain and lower motor neuron groups in the brainstem and spinal cord(passive) are caused byThe clinical features

either raised intracranial tension or infarction(passive) caused byclinical features

various diseased causes , which mainly behaves as glomerular damage of increased permeability of glomerular basement membrane as well as declined GFR that patients suffer from increasingly deteriorative symptoms and complications of pathological changes to systemic damage attribute to varying diseased causes , regions and degrees(passive) caused byclinical features

not just likely diseases but also causative drugs ( drugsmight causethe clinical features

various diseased factors , which mainly behaves as increased permeability of glomerular basement membrane as well as declined GFR that patients suffer from increasingly serious symptoms and complications of slight pathological changes to severe parenchymal and functional damage according to distinct diseased causes , regions and degrees(passive) caused byclinical features

numerous diseased causes , which mainly behaves as glomerular damage of kidneys with increased permeability of glomerular basement membrane as well as decreased GFR that patients suffer from increasingly serious internal and external lesions from slight pathological and physiological insufficiency to severe parenchymal and functional dysfunction according to the progression of diseased course(passive) caused byclinical features

various factors , which mainly attributes to glomerular damage of increased glomerular basement membrane and declined GFR of kidneys(passive) caused byclinical features

various diseased factors , which mainly behaves as renal glomerular lesions with increased permeability of glomerular basement membrane as well as declined GFR that patients suffer from increasingly serious internal and external damage from slight pathological and physiological insufficiency to severe parenchymal and functional dysfunction according to varying diseased causes , regions and degrees(passive) caused byclinical features

various factors , which mainly behaves as increased glomerular basement membrane and declined GFR that patients mainly suffer from four significant symptoms(passive) caused byclinical features

various factors , which mainly behaves as glomerular damage of increased glomerular basement membrane and declined GFR that patients suffer from four significant characters(passive) caused byclinical features

the significantly different serum IFN - g concentrations remains to be determined between T - LAHS and B - LAHS(passive) caused byclinical features

to earlier diagnosiscould leadto earlier diagnosis

further investigation of IS as a diagnosispromptfurther investigation of IS as a diagnosis

to first diagnosisleadingto first diagnosis

to the diagnosis of asthmaleadto the diagnosis of asthma

to positive diagnosiscan leadto positive diagnosis

to a delayed diagnosiscontributeto a delayed diagnosis

to a diagnosis of ADleadingto a diagnosis of AD

to an imprecise diagnosismay leadto an imprecise diagnosis

in a diagnosis of AOSresultedin a diagnosis of AOS

to the preoperative diagnosisleadto the preoperative diagnosis

the diagnosis of SCIDpromptingthe diagnosis of SCID

to a diagnosis of AGSwould have ledto a diagnosis of AGS

to a diagnosis of ALGSwould leadto a diagnosis of ALGS

to the diagnosis of XLAleadingto the diagnosis of XLA

to the diagnosis of iMCDledto the diagnosis of iMCD

the Treatment Plan 7Influencingthe Treatment Plan 7

to the diagnosis of central stenosiscontributeto the diagnosis of central stenosis

to suspicion and implications of this diagnosisleadingto suspicion and implications of this diagnosis

to the specific diagnosis in this caseledto the specific diagnosis in this case

the possible diagnosis of hyperparathyroidismpromptedthe possible diagnosis of hyperparathyroidism

to a clinical diagnosis of bronchopneumonialeadingto a clinical diagnosis of bronchopneumonia

to a diagnosis of neum [ 26leadingto a diagnosis of neum [ 26

to conflation of diagnosis and phenotypeleadingto conflation of diagnosis and phenotype

in a confusing clinical diagnosisresultingin a confusing clinical diagnosis

to false outcome prediction of the Status Epilepticus Severity Score ( STESS ) , a validated and broadly used clinical scoring system for outcome prediction in status epilepticus ( SEleadingto false outcome prediction of the Status Epilepticus Severity Score ( STESS ) , a validated and broadly used clinical scoring system for outcome prediction in status epilepticus ( SE

to the early diagnosis of posterior fossa epidural hematomacould leadto the early diagnosis of posterior fossa epidural hematoma

and recognize the diagnosis approach for HHTcontributeand recognize the diagnosis approach for HHT

to a presumptive diagnosis of lung carcinomaledto a presumptive diagnosis of lung carcinoma

to a diagnosis of a suspected case of measlesleadto a diagnosis of a suspected case of measles

to the correct diagnosis of entrapped embolusleadingto the correct diagnosis of entrapped embolus

to consideration of this diagnosis and recognition of the importance of the cerebrovascular patternsshould leadto consideration of this diagnosis and recognition of the importance of the cerebrovascular patterns

to a diagnosis of DON in EUGOGO centres across Europeleadingto a diagnosis of DON in EUGOGO centres across Europe

in confusion during diagnosis ( Pedersen et al . , 2017could resultin confusion during diagnosis ( Pedersen et al . , 2017

to diagnosis and with present means should assure it with the minimum of delayshould leadto diagnosis and with present means should assure it with the minimum of delay

diagnosis and principles of management of : Haemonthorax , constrictive pericarditiscausesdiagnosis and principles of management of : Haemonthorax , constrictive pericarditis

to the diagnosis of first bite syndrome , the pathophysiology of this syndromeleadingto the diagnosis of first bite syndrome , the pathophysiology of this syndrome

to misleading diagnosis as they closely mimic other diseases like dermatomyositiscan ... leadto misleading diagnosis as they closely mimic other diseases like dermatomyositis

diagnosis and principles of management Laryngeal tumour , esophageal tumourscausesdiagnosis and principles of management Laryngeal tumour , esophageal tumours

referral for diagnostic testing and provided evidence that diagnosis is often missed or delayedshould promptreferral for diagnostic testing and provided evidence that diagnosis is often missed or delayed

acute distress and impairment in one patient may represent another patient ’s usual statuscauseacute distress and impairment in one patient may represent another patient ’s usual status

Blob

Smart Reasoning:

C&E

See more*