formulationresultingin higher loading efficiency of plasmid DNA per ml of formulation
CalTech and UCLAdiscovereda method of manipulating the mitochondria of cells to effectively repair DNA
Bleomycin sulfate treatmentresultedin elongation of E. coli cells and enlargement of HeLa cells through inhibiting DNA and protein syntheses
which of the two groupsinventedthe use of CRISPR / Cas9 for editing DNA in eukaryotic cells
Bifunctional agentsleadto cross - linking of DNA which leads to more efficient cell death
talksresultingfrom the use of HeLa cell genome data
that suggestedcould leadto mitochondrial DNA breakage in a series of mammalian cells
The researchersalso discoveredcircular pieces of DNA enduring mcr-3 , which were derived from IncP plasmids
this elementdoes causecells to release DNA into the medium
These structuresleadto cooperative binding of cGAS to DNA as a function of DNA length
this complex pathwaycontributesto DNA damage induced mutagenesis in eukaryotic cells
a stable TOP2-DPC complexleadsto the accumulation of severed DNA that kills cells
Proteinscan resultfrom the expression of recombinant DNA within living cells
Proteinsresultfrom the expression of recombinant DNA within living cells
Proteins Proteinsresultfrom the expression of recombinant DNA within living cells
which inventorsinventedthe use of CRISPR / Cas9 in eukaryotic cells
complex 1causedcellular DNA damage in HeLa cells
In the case of Thalidomide , it was discoveredcausedmalformations by interacting with the DNA of the dividing embryonic cells
Working with P H Huang , he proposedcausedmalformations by interacting with the DNA of the dividing embryonic cells
All the tested combinationsledto a synergistic decline in cell viability of HeLa cells
The topology ( linear or supercoiled ) and the size of the vector construct , the qualityof the plasmid DNA , and the promoter choice ... major factorsinfluencethe efficiency of plasmid DNA transfection
With the use of gel electrophoresis , the cells can be observedcausesDNA fragmentation in HeLa cells
Working with P H Huang , William McBride proposedcausedmalformations by interacting with the DNA of the dividing embryonic cells
and tau co - transfectionfurther resultedin extended DNA disorganization in N2a cells
With the use of gel electrophoresis , it can be observedcausesDNA fragmentation in HeLa cells
A defect in N terminal cleavage of CTMP manifestationleadsto inflamed mitochondria HeLa cells transfected with full - length CTMP
the pathwayscontributeto genome stability in eukaryotic cells
SFPQ dislocation was found enhanced with tau co - transfection and tau co - transfectionfurther resultedin extended DNA disorganization in N2a cells
to mediate cooperative DNA binding by DNMT3A andto contributeto the efficient DNA methylation by DNMT3A in cells
reactive oxygen species ( ROSoriginatingfrom mitochondria accumulates in cells over an animal 's lifespan
an agentmay causeintrastrand crosslinks in the DNA of tumor cells
This product(passive) is designedThis product
These stains(passive) have been designedThese stains
to a measurable reduction in your body 's cells ability to reproduceleadingto a measurable reduction in your body 's cells ability to reproduce
cell cycle progression and apoptosis(passive) caused bycell cycle progression and apoptosis
to elevation of DNA with long fragments in plasma or serummay leadto elevation of DNA with long fragments in plasma or serum
to overall premature agingleadsto overall premature aging
in precancerous lesions that progress to cancercan resultin precancerous lesions that progress to cancer
in animalscan causein animals
in the chromosome butoriginatein the chromosome but
Buccal Cells kit(passive) is designedBuccal Cells kit
The REPLI - g Single Cell Kit(passive) is specially designedThe REPLI - g Single Cell Kit
to reduced FMRP , which is important for normal cognitive development ( 15leadsto reduced FMRP , which is important for normal cognitive development ( 15
Purification Kit(passive) is designedPurification Kit
to dysfunctional telomeres , arrest of cell growth and cell deatheventually leadingto dysfunctional telomeres , arrest of cell growth and cell death
The agreement , which does not provide the family with the right to potential earnings from future research on Ms. Lacks 's genome(passive) was prompted byThe agreement , which does not provide the family with the right to potential earnings from future research on Ms. Lacks 's genome
to formation of cancer cellsleadto formation of cancer cells
in a strong reduction of RAMA1 mRNA levels ( Fig . 4Aresultedin a strong reduction of RAMA1 mRNA levels ( Fig . 4A
to a chronic activation of the innate immune response that could result in the development of systemic lupus erythematosus ( SLEleadingto a chronic activation of the innate immune response that could result in the development of systemic lupus erythematosus ( SLE
to apoptosis and cell deathledto apoptosis and cell death
to the disintegration of the nucleusleadsto the disintegration of the nucleus
Lipofectamine RNAiMAX(passive) is specifically designedLipofectamine RNAiMAX