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2016 - 06 - 22 SIRT5preventscigarette smoke extract - induced apoptosis in lung epithelial cells
Wang Y , Zhu Y , Xing S , Ma P , Lin D. SIRT5preventscigarette smoke extract - induced apoptosis in lung epithelial cells
NAC may helppreventchanges caused by smoking in cells lining the respiratory passage
Highly Influential CitationsCitationsAccelerationVelocityRecencyLearn MoreSIRT5preventscigarette smoke extract - induced apoptosis in lung epithelial cells
ableto createa better understanding of cigarette smoke effects on lung epithelial cells
the antioxidant effect of the flavonoids in licoricemight preventinjury to the lung epithelial cell from exposure to cigarette smoke
The characteristic changes in airway morphology , inflammatory cell infiltration and mediator expression in COPDmay resultfrom direct effects of cigarette smoke on airway cells
The humectantsmay influencethe deposition of cigarette smoke in the human respiratory tract
N - acetylcysteine , an inhibitor of reactive oxygen species , nitric oxide , and reactive aldehydespreventedthe effects of cigarette smoke on epithelial cells
Gravitational sedimentation , inertial impaction and Brownian motion ( diffusion ... the three basic mechanismsinfluenceon the behaviour of particles in cigarette smoke within the respiratory tract
damage(passive) caused bydamage
to T cell activation [ 21leadingto T cell activation [ 21
to cell toxicityleadsto cell toxicity
its potential protection against cell death(passive) caused byits potential protection against cell death
to the exhaustion of the pool of reduced GSHleadsto the exhaustion of the pool of reduced GSH
production of inflammatory mediators ( tumour necrosis factor ( TNF)-α , interleukin ( IL)-1β , granulocyte - macrophage colony - stimulating factor , IL-8 and leukotriene ( LT)B4 ) , which are responsible for activating and/or recruiting alveolar macrophages and neutrophilscausingproduction of inflammatory mediators ( tumour necrosis factor ( TNF)-α , interleukin ( IL)-1β , granulocyte - macrophage colony - stimulating factor , IL-8 and leukotriene ( LT)B4 ) , which are responsible for activating and/or recruiting alveolar macrophages and neutrophils
to COPD pathology ( Rusznak et al . 2000 ; Schulz et al . 2004 ... Heijink et al . 2012 ; Amatngalim et al .contributesto COPD pathology ( Rusznak et al . 2000 ; Schulz et al . 2004 ... Heijink et al . 2012 ; Amatngalim et al .
irritation and eventually mucus or phlegm will appear as a form of defense of the bodycan causeirritation and eventually mucus or phlegm will appear as a form of defense of the body