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to protect the cholinergic system , which is essential for memory and learning and is progressively destroyed in Alzheimer 's(passive) are designedCholinesterase inhibitors
with the aim of developing analgesic and antihypertensive agents that act by preventing neprilysin 's activity against signaling peptides such as enkephalins , substance P , endothelin , and atrial natriuretic peptide.[13][14(passive) have been designedInhibitors
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R. How can we best use structural information on P - glycoproteinto designinhibitors
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High throughput screening effortsto discoverinhibitors
how this monitoring system ... improvedto preventinhibitors
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C.C. Reengineering an azaphilone biosynthesis pathway in Aspergillus nidulansto createlipoxygenase inhibitors
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with the aim of developing analgesic and antihypertensive agents that act by preventing neprilysin 's activity against signaling peptides such as enkephalins , substance P , endothelin , and atrial natriuretic factor.[10][11(passive) have been designedInhibitors
with the aim of developing analgesic and antihypertensive agents that act by preventing neprilysin 's activity against signaling peptides such as enkephalins , substance P , endothelin , and atrial natriuretic factor.[8][9(passive) have been designedInhibitors
with the aim of developing analgesic and antihypertensive agents that act by preventing neprilysin 's activity against signaling peptides such as enkephalins , substance P , endothelin , and atrial natriuretic factor.[11][12(passive) have been designedInhibitors
with the aim of developing analgesic and antihypertensive agents that act by preventing neprilysin 's activity against signaling peptides such as enkephalins , substance P , endothelin , and atrial natriuretic factor.[12][13(passive) have been designedInhibitors
recently(passive) were ... discovered/ AKT inhibitors
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varying widely from acceleratorsto setinhibitors
to mimic or inhibit specific miRNAs for artificial upregulation and downregulation of target mRNA translation , respectively(passive) are designedInhibitors
it difficultto designinhibitors
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Chem . , an international group of collaborators led by Kristaps Jaudzems and Aigars Jirgensons at the Latvian Institute of Organic Synthesis describediscoveredinhibitors
coadministration with outer membrane permeability enhancers profoundly sensitizes Escherichia colito designedLpxH inhibitors
to increase levels of high - density lipoprotein ( HDL ) cholesterol - which is sometimes described as ' good ' cholesterol with a protective cardiovascular effect although that hypothesis has been dented by the CETP class failures(passive) are designedCETP inhibitors
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orderto successfully designinhibitors
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to break down endocannabinoids , including anandamide , in the brain and are also being investigated for use in the treatment of chronic pain(passive) are designedFAAH inhibitors
29697.69 Strategyto designinhibitors
to prevent the formation of new blood vessels , thereby stopping or slowing the growth or spread of tumors(passive) are designedAngiogenesis inhibitors
to stop the proteasomes from recycling proteins inside cancer cells , causing a toxic protein overload that may kill the cell(passive) are designedProteasome inhibitors
We applied virtual screeningto discoverprimase inhibitors
Grinding aids and packsetinhibitors
to break down endocannabinoids , including anandamide , in the brain and are being investigated for use in the treatment of chronic pain(passive) are designedFAAH inhibitors
from natural sources , including tree bark , fungi , shark muscle and cartilage , sea coral , green tea , and herbs ( licorice , ginseng , cumin , garlic(passive) have been discoveredAngiogenesis inhibitors
in an accumulation from the neurotransmitter acetylcholineresultin an accumulation from the neurotransmitter acetylcholine
the breakdown of acetylcholine Neonicotinoidspreventthe breakdown of acetylcholine Neonicotinoids
the breakdown of acetylcholine by inhibiting acetylcholinesterasepreventthe breakdown of acetylcholine by inhibiting acetylcholinesterase
the breakdown of acetylcholine in the neuromuscular junctionpreventthe breakdown of acetylcholine in the neuromuscular junction
the breakdown of acetylcholine , and memantine , which controls glutamatepreventthe breakdown of acetylcholine , and memantine , which controls glutamate
the breakdown of acetylcholine , a chemical that nerves use to communicate with each otherpreventthe breakdown of acetylcholine , a chemical that nerves use to communicate with each other
the breakdown of acetylcholine ( an essential chemical for learning and memorypreventthe breakdown of acetylcholine ( an essential chemical for learning and memory
the breakdown of acetylcholine – a chemical that functions as a neurotransmitter in the braincan preventthe breakdown of acetylcholine – a chemical that functions as a neurotransmitter in the brain
the breakdown of acetylcholine , an important chemical for learning and memorypreventthe breakdown of acetylcholine , an important chemical for learning and memory
to decreased enzyme activity in the bodycontributeto decreased enzyme activity in the body
the breakdown of acetylcholine , a neurotransmitter that plays a role in memory and thinkingpreventthe breakdown of acetylcholine , a neurotransmitter that plays a role in memory and thinking
the breakdown of acetylcholine a chemical messenger important for learning and memorypreventthe breakdown of acetylcholine a chemical messenger important for learning and memory
the breakdown of acetylcholine , a chemical messenger that is key to learning and memorypreventthe breakdown of acetylcholine , a chemical messenger that is key to learning and memory
or reduce amplification and cause false - negative NAA results ( 8,9preventor reduce amplification and cause false - negative NAA results ( 8,9
the breakdown of acetylcholine in the brain Used to treat mild to moderate Alzheimer 's Disease Hard[quizlet.comto preventthe breakdown of acetylcholine in the brain Used to treat mild to moderate Alzheimer 's Disease Hard[quizlet.com
the breakdown of acetylcholine that results in the partial restoration of memory and decreased confusioncan preventthe breakdown of acetylcholine that results in the partial restoration of memory and decreased confusion
the breakdown of acetylcholine , which is a chemical messenger that is essential for learning and memory functionpreventthe breakdown of acetylcholine , which is a chemical messenger that is essential for learning and memory function
the breakdown of acetylcholine , which is a chemical messenger vital for learning and memory in the brainpreventthe breakdown of acetylcholine , which is a chemical messenger vital for learning and memory in the brain
the breakdown of acetylcholine ( by acetyl cholinesterase ) which increases the availability of Ach at cholinergic synapsespreventthe breakdown of acetylcholine ( by acetyl cholinesterase ) which increases the availability of Ach at cholinergic synapses
in 50 % inhibition of enzyme activity ( IC50resultingin 50 % inhibition of enzyme activity ( IC50
the AChEi enzymepreventthe AChEi enzyme
the enzyme from being harmfulpreventthe enzyme from being harmful
enzyme catalysiscan preventenzyme catalysis
Yasunaga H. Cholinergic crisis(passive) caused byYasunaga H. Cholinergic crisis
the breakdown of acetylcholine ( a - SEA - til - KOHlean ) , a chemical messenger important for memory and learningpreventthe breakdown of acetylcholine ( a - SEA - til - KOHlean ) , a chemical messenger important for memory and learning
the reaction of the enzymes with substrates ... the enzyme is relatively specificpreventthe reaction of the enzymes with substrates ... the enzyme is relatively specific
the body to work against the factor replacement therapy , neutralizing its effect and preventing an individual 's blood from appropriate clotting.6 Individuals who have inhibitorscausethe body to work against the factor replacement therapy , neutralizing its effect and preventing an individual 's blood from appropriate clotting.6 Individuals who have inhibitors
metabolism of acetylcholinepreventmetabolism of acetylcholine
to knock out PLpro and stop the replication of the virusdesignedto knock out PLpro and stop the replication of the virus
the binding of calcium to calmodulinepreventthe binding of calcium to calmoduline
the binding of calcium to calmodulinpreventthe binding of calcium to calmodulin
the action of the enzyme cholinesterase , which breaks down acetylcholinepreventthe action of the enzyme cholinesterase , which breaks down acetylcholine
to target one enzyme from a particular bacterial speciesdesignedto target one enzyme from a particular bacterial species
gastrointestinal side effects(passive) caused bygastrointestinal side effects
poisonings(passive) caused bypoisonings
the binding of white blood cells to VCAM-1 and fibronectinwould preventthe binding of white blood cells to VCAM-1 and fibronectin
falls in Parkinson ’s diseaseto preventfalls in Parkinson ’s disease
APP metabolism in Alzheimer disease patients , NeurobiolinfluenceAPP metabolism in Alzheimer disease patients , Neurobiol
the breakdown of acetylcholine ( a - SEA - til - KOH - lean ) , a chemical compound that works as a neurotransmitter in the Central Nervous Systempreventthe breakdown of acetylcholine ( a - SEA - til - KOH - lean ) , a chemical compound that works as a neurotransmitter in the Central Nervous System
against Mycobacterium tuberculosis thymidylate kinase : new insights into the phosphoryl transfer mechanism , ” Journal of Biological Chemistrydesignedagainst Mycobacterium tuberculosis thymidylate kinase : new insights into the phosphoryl transfer mechanism , ” Journal of Biological Chemistry