disease recurrence after chemotherapycausedisease recurrence after chemotherapy
to a potent immunogenic phenotype with induction of antigen - specific T - cell responses both in vitro and in vivoleadsto a potent immunogenic phenotype with induction of antigen - specific T - cell responses both in vitro and in vivo
to decreased proliferation and tumor growthleadingto decreased proliferation and tumor growth
to a decreased proliferation and tumor growthleadingto a decreased proliferation and tumor growth
to an increased infiltration of various immune cells into the tumorleadingto an increased infiltration of various immune cells into the tumor
to a decreased proliferation and tumor growth ... 94leadsto a decreased proliferation and tumor growth ... 94
to tumor initiation , growth and metastasiscan ... contributeto tumor initiation , growth and metastasis
to tumor volumecan ... contributeto tumor volume
significantlyinfluencesignificantly
to general inhibition of NK cell cytotoxicityleadsto general inhibition of NK cell cytotoxicity
in an interconnected fashion to malignant progressioncontributein an interconnected fashion to malignant progression
to its established effectiveness against various tumor typesmay contributeto its established effectiveness against various tumor types
damagewill causedamage
damagemay causedamage
either in growth proliferation or arrest of different human glioblastoma cell linesresultingeither in growth proliferation or arrest of different human glioblastoma cell lines
to tumor angiogenesiscontributeto tumor angiogenesis
in the induction of apoptosisresultingin the induction of apoptosis
to therapeutic antitumor effects in animalsleadingto therapeutic antitumor effects in animals
from a variety of tissuesoriginatingfrom a variety of tissues
the samples(passive) were composedthe samples
to the established effectiveness of this dietary indole against various tumors typesmay contributeto the established effectiveness of this dietary indole against various tumors types
to tumor necrosisleadsto tumor necrosis
to tumor metastasiscontributeto tumor metastasis
in a tumor cell - directed inflammatory response and rapid tumor destructionmay resultin a tumor cell - directed inflammatory response and rapid tumor destruction
in immune escape by tumor cellsalso resultin immune escape by tumor cells
the hypercalcemia(passive) caused bythe hypercalcemia
to enhanced metastasisleadsto enhanced metastasis
from the primary or the metastasis lesions and disseminating to the peripheral blood circulationoriginatingfrom the primary or the metastasis lesions and disseminating to the peripheral blood circulation
from a primary tumororiginatefrom a primary tumor
from mammary gland , prostate , colon , liver , lung and skin ( Sundaram and Milner 1996aoriginatingfrom mammary gland , prostate , colon , liver , lung and skin ( Sundaram and Milner 1996a
to increased cell invasion and tumor growthcan leadto increased cell invasion and tumor growth
in tumor dormancy , drug resistant , and subsequently leading to tumor regrowth and processioncould ... resultedin tumor dormancy , drug resistant , and subsequently leading to tumor regrowth and procession
in tumor dormancy , drug resistant , and subsequently leading to tumor regrowth and processioncould therefore resultedin tumor dormancy , drug resistant , and subsequently leading to tumor regrowth and procession
cancer to spreadcausescancer to spread
in less impact on healthy tissues and organs and less side effectsresultingin less impact on healthy tissues and organs and less side effects
to the spreadcontributeto the spread
the growth of tumor cellsinfluencethe growth of tumor cells