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to block the channel where the calcium is leaking into the cell(passive) are designedchannel blocker drugs
Extracellular Cd2 + blocked release , but no single selective Ca2 +preventedchannel blocker
01/04/2002 - " A novel voltage - sensitive Na(+ ) and Ca(2 +preventschannel blocker , NS-7
B - receptor antagonists ... patients on other medicationsmight causebradycardia(ie.calcium channel blockers
Amiodarone kcauseschannel blocker miacalcin
using acetylcholineto causeCa2þ channel blocker
also(passive) can ... be influencedChannel blockers
the criteriasetfor open channel blockers
Diltiazem , VerapamilcausesCa channel blockers
that includes structure - based drug design , medicinal chemistry , electrophysiology , and virologyto designM2-S31N channel blockers
the development of a substrate for atrial fibrillation by tachycardia - induced atrial remodeling in dogs , Circulation , 1999;100:2191–7preventsthe development of a substrate for atrial fibrillation by tachycardia - induced atrial remodeling in dogs , Circulation , 1999;100:2191–7
physical dependence and the enhancement of protein kinase C activity by opioid infusion in rats , European Journal of Pharmacology |preventsphysical dependence and the enhancement of protein kinase C activity by opioid infusion in rats , European Journal of Pharmacology |
ischemic preconditioningpreventischemic preconditioning
the development of a substrate for atrial fibrillation by tachycardia - induced atrial remodeling in dogs S. Fareh et al .preventsthe development of a substrate for atrial fibrillation by tachycardia - induced atrial remodeling in dogs S. Fareh et al .
the development of a substrate for atrial fibrillation by tachycardia - induced atrial remodeling in dogs " , CIRCULATION , 100(21 ) , 1999 , pppreventsthe development of a substrate for atrial fibrillation by tachycardia - induced atrial remodeling in dogs " , CIRCULATION , 100(21 ) , 1999 , pp
cardiac cell hypertrophy through an inhibition of calcineurin - nfat3 activation as well as L - type Ca2 + channel blockerspreventcardiac cell hypertrophy through an inhibition of calcineurin - nfat3 activation as well as L - type Ca2 + channel blockers
oxaliplatin - induced cold hyperalgesia and TRPM8 overexpression in rats ... abstract =preventoxaliplatin - induced cold hyperalgesia and TRPM8 overexpression in rats ... abstract =
the Development of a Substrate for Atrial Fibrillation by Tachycardia - Induced Atrial Remodeling in Dogs Samir Fareh , Agnès Bénardeau , Bernard Thibault and Stanley Nattel CirculationPreventsthe Development of a Substrate for Atrial Fibrillation by Tachycardia - Induced Atrial Remodeling in Dogs Samir Fareh , Agnès Bénardeau , Bernard Thibault and Stanley Nattel Circulation
oxaliplatin - induced cold hyperalgesia and TRPM8 overexpression in rats — 九州大学 Backgroundpreventoxaliplatin - induced cold hyperalgesia and TRPM8 overexpression in rats — 九州大学 Background
the Development of a Substrate for Atrial Fibrillation by Tachycardia - Induced Atrial Remodeling in Dogs | Circulation http://dx.doi.org/10.1161/01.CIR.100.21.2191 CirculationPreventsthe Development of a Substrate for Atrial Fibrillation by Tachycardia - Induced Atrial Remodeling in Dogs | Circulation http://dx.doi.org/10.1161/01.CIR.100.21.2191 Circulation
suppression of cyclic AMP - dependent protein kinase and reduces infarct area in the acute phase of cerebral ischemia in ratpreventssuppression of cyclic AMP - dependent protein kinase and reduces infarct area in the acute phase of cerebral ischemia in rat
oxaliplatin - induced cold hyperalgesia and TRPM8 overexpression in rats ' , Molecular Pain , 巻. 8 , 7preventoxaliplatin - induced cold hyperalgesia and TRPM8 overexpression in rats ' , Molecular Pain , 巻. 8 , 7
oxaliplatin - induced cold hyperalgesia and TRPM8 overexpression in rats ... MOLECULAR PAIN , 10.1186/1744 - 8069 - 8 - 7 , 8 , 2012.01preventoxaliplatin - induced cold hyperalgesia and TRPM8 overexpression in rats ... MOLECULAR PAIN , 10.1186/1744 - 8069 - 8 - 7 , 8 , 2012.01
oxaliplatin - induced cold hyperalgesia and TRPM8 overexpression in rats Takehiro Kawashiri , Nobuaki Egashira , Kentaro Kurobe , Kuniaki Tsutsumi , Yuji Yamashita , Soichiro Ushio , Takahisa Yano , Ryozo Oishi Molecular Pain , 2012 ... DOI : 10.1186/1744 - 8069 - 8 - 7preventoxaliplatin - induced cold hyperalgesia and TRPM8 overexpression in rats Takehiro Kawashiri , Nobuaki Egashira , Kentaro Kurobe , Kuniaki Tsutsumi , Yuji Yamashita , Soichiro Ushio , Takahisa Yano , Ryozo Oishi Molecular Pain , 2012 ... DOI : 10.1186/1744 - 8069 - 8 - 7
the Development of a Substrate for Atrial Fibrillation by Tachycardia - Induced Atrial Remodeling in DogsIonic mechanisms of electrical remodeling in human atrial fibrillationIonic Determinants of Atrial Fibrillation and Ca2 +Preventsthe Development of a Substrate for Atrial Fibrillation by Tachycardia - Induced Atrial Remodeling in DogsIonic mechanisms of electrical remodeling in human atrial fibrillationIonic Determinants of Atrial Fibrillation and Ca2 +
the Development of a Substrate for Atrial Fibrillation by Tachycardia - Induced Atrial Remodeling in DogsDigoxin Delays Recovery From Tachycardia - Induced Electrical Remodeling of the AtriaAtrial L - Type Ca2 + Currents and Human Atrial FibrillationIonic Determinants of Atrial Fibrillation and Ca2 +Preventsthe Development of a Substrate for Atrial Fibrillation by Tachycardia - Induced Atrial Remodeling in DogsDigoxin Delays Recovery From Tachycardia - Induced Electrical Remodeling of the AtriaAtrial L - Type Ca2 + Currents and Human Atrial FibrillationIonic Determinants of Atrial Fibrillation and Ca2 +
morphological feature changes such as nuclear shrinkage and fragmentation ... suggesting an activation of apoptotic signaling mechanisms ( Abdul and Hoosein , 2002atriggeredmorphological feature changes such as nuclear shrinkage and fragmentation ... suggesting an activation of apoptotic signaling mechanisms ( Abdul and Hoosein , 2002a
Rosaceacould have triggeredRosacea
to total suppression of mRNA accumulationledto total suppression of mRNA accumulation
secretion evoked by 50 mmK+ ( Taylor and Peers , 1998Cd2+completely preventedsecretion evoked by 50 mmK+ ( Taylor and Peers , 1998
CA1 injury 24 hpreventsCA1 injury 24 h
glucose-preventedglucose-
CA1 injury 24 hrpreventsCA1 injury 24 hr
no side effects expected from channel block in other tissuescauseno side effects expected from channel block in other tissues
to slow the HR , as an anti - anginal agentdesignedto slow the HR , as an anti - anginal agent
channel closure ton , B.J. , Carter , D.B. , Mohler , H. , Benson , J.A. , 1996preventchannel closure ton , B.J. , Carter , D.B. , Mohler , H. , Benson , J.A. , 1996
IPCpreventIPC
the activation of GPMBP - reactive T cellsmay preventthe activation of GPMBP - reactive T cells
bursting while allowing individual fast spikes [ 13preventbursting while allowing individual fast spikes [ 13
the entry of Ca2to preventthe entry of Ca2
the covalent reaction(passive) could be prevented bythe covalent reaction
development of cellular hypertrophy and arrhythmiapreventeddevelopment of cellular hypertrophy and arrhythmia
of ω - CTX - GVIA ( 1 μm ) , ω - CTX - MVIIC ( 3 μm ) , Nife ( 3 μm ) , and ωcomposedof ω - CTX - GVIA ( 1 μm ) , ω - CTX - MVIIC ( 3 μm ) , Nife ( 3 μm ) , and ω
heat supplied by a heat source from reaching the film in certain areaspreventheat supplied by a heat source from reaching the film in certain areas
Cl−preventCl−
from interferences on the Ca2+/ cAMP signalling interactionresultedfrom interferences on the Ca2+/ cAMP signalling interaction
the sustained phase of the ET-1-induced Ca2to preventthe sustained phase of the ET-1-induced Ca2
extracellular Ca ( membrane channels ... from entering in the cellCa ... preventextracellular Ca ( membrane channels ... from entering in the cell
granulysin - induced [ Ca2+]i increasepreventgranulysin - induced [ Ca2+]i increase
the development of membrane permeability to large , but not small , molecules following oxidant injurypreventthe development of membrane permeability to large , but not small , molecules following oxidant injury