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Smart Reasoning:

C&E

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Qaagi - Book of Why

Causes

Effects

H / R treatment ( Fig . 2A(passive) caused bycell viability

drug treatment For this compound(passive) caused bycell viability

Vorinostat treatment(passive) caused bycell viability

doxorubicin treatment(passive) caused bycell viability

etanercept treatment(passive) caused bycell viability

rapamycin treatment only(passive) caused bycell viability

the chrysin treatment(passive) caused bycell viability

treatment with the HH - F3 fraction in PLC5 cells(passive) caused bycell viability

treatment with the HH - F3 fraction in Huh7 cells(passive) caused bycell viability

treatment with the HH - F3 fraction in Mahlavu cells(passive) caused bycell viability

treatment with the HH - F3 fraction in HSC - T6 cells(passive) caused bycell viability

No Abstract Tumor necrosis factor α ( TNF - αinfluencesendothelial cell viability

that sugar , inoculum concentration and proportion of breadfruit flourinfluencedcell viability

the combination of hCG and OP treatment(passive) caused bycell viability

treatment with TNF - α(passive) caused bycell viability

co - treatment at 24(passive) caused bycell viability

SUN N8075 treatment(passive) caused bycell viability

embelin + TRAIL treatment(passive) caused bycell viability

3 March 2014 Tumor necrosis factor α ( TNF - αinfluencesendothelial cell viability

the combination of both 100 nm RTX and CH-11 and 1 μm MTA and(passive) caused bycell viability

high concentration cisplatin treatment alone(passive) caused bycell viability

excess vitamin A , and iron - ascorbate systems(passive) caused bycell viability

treatment with EtnK-1 siRNA(passive) caused bycell viability

R(+)-MA treatment ( data not shown(passive) caused bycell viability

both MPP+ and H2O2 exposure , respectively(passive) caused bythe cell viability

Greenseid K , Jindal S , Zapantis A , Nihsen M , Hurwitz J , Pal L. Declining ovarian reserveadversely influencesgranulosa cell viability

LL-37 in HCT116 and LoVo ( Fig .(passive) caused bycell viability

the treatment of MDCK cells with the SILAC media(passive) provoked bycell viability

to ... HG challenge(passive) led ... bycell viability

SREBP-2 than by LXR activity(passive) is ... influenced bycell viability

silica nanoparticles(passive) caused bycell viability

Let-7f-1 - 3p antagomir treatment(passive) caused bycell viability

H2O2 alone(passive) caused bycell viability

exposure to plasma(passive) caused bycell viability

3.56 Fertility and sterility Declining ovarian reserveadversely influencesgranulosa cell viability

the apparent neutralization capacity , such as IFNγ or factorsinfluencingcell viability

RA per secan influencecell viability

tivantinib ( Fig . 4E(passive) caused bycell viability

brief exposure to 10 microM Glu for 1 hr in a dose - dependent manner at concentrations from 0.1 to 10 microM(passive) caused bycellular viability

the high glucose exposure(passive) caused bycell viability

to cell death and lysisleadingto cell death and lysis

in ∼80 % cell deathresultingin ∼80 % cell death

in cell death ... either necrotic or apoptoticcan ... resultin cell death ... either necrotic or apoptotic

from 80 SHV pulsesresultedfrom 80 SHV pulses

from UV Light Exposure Statistically Average Percent Different from Viability � UV IrradiationResultingfrom UV Light Exposure Statistically Average Percent Different from Viability � UV Irradiation

in enzyme inactivation , membrane disruption , and cell death [ 48resultingin enzyme inactivation , membrane disruption , and cell death [ 48

from SHV pulses applied aloneresultedfrom SHV pulses applied alone

to cell death after a 30 h at concentrations as low as 20 nM. Clearlyleadingto cell death after a 30 h at concentrations as low as 20 nM. Clearly

in a higher quality and quantity of stem cells to provide the best service to client ’s customersresultingin a higher quality and quantity of stem cells to provide the best service to client ’s customers

to cell death by its action on the cAMP in the Small cell lung carcinoma cell linesleadingto cell death by its action on the cAMP in the Small cell lung carcinoma cell lines

from OGD(2)/R(24resultingfrom OGD(2)/R(24

to the induction of apoptosisleadsto the induction of apoptosis

after 80 LLV pulses was higher than that after 20 SHV pulses   +resultingafter 80 LLV pulses was higher than that after 20 SHV pulses   +

from current cryopreservation methodsresultingfrom current cryopreservation methods

from 20 SHV   +   60 LLV ( 360   Vresultedfrom 20 SHV   +   60 LLV ( 360   V

from infection with H. capsulatum with the addition of uracil to the culture medium , including the uninfected wells used to establish 100 % viabilityresultingfrom infection with H. capsulatum with the addition of uracil to the culture medium , including the uninfected wells used to establish 100 % viability

to an increased proportion of cells in the G0 / G1 phase and cell apoptosis via suppression of the PI3K / Akt signaling pathwaymay contributeto an increased proportion of cells in the G0 / G1 phase and cell apoptosis via suppression of the PI3K / Akt signaling pathway

to an increased proportion of cells in the G0 / G1 phase and cell apoptosis via suppression of the PI3 K ( Montrer PIK3CAmay contributeto an increased proportion of cells in the G0 / G1 phase and cell apoptosis via suppression of the PI3 K ( Montrer PIK3CA

to an increased proportion of cells in the G0 / G1 phase and cell apoptosis via suppression of the PI3K / Akt signalling pathwaymay contributeto an increased proportion of cells in the G0 / G1 phase and cell apoptosis via suppression of the PI3K / Akt signalling pathway

to an increased proportion of cells in the G0 / G1 phase and cell apoptosis via suppression of the PI3 K ( zeige PIK3CA Antikörper)/Aktmay contributeto an increased proportion of cells in the G0 / G1 phase and cell apoptosis via suppression of the PI3 K ( zeige PIK3CA Antikörper)/Akt

in a 50–60 % cell toxicityresultingin a 50–60 % cell toxicity

expression levels of proteins or genescould influenceexpression levels of proteins or genes

to the suggestion that TG activity is necessary for ensuring cell survival under conditions of differentiation or cellular stresshas ledto the suggestion that TG activity is necessary for ensuring cell survival under conditions of differentiation or cellular stress

to the normal morphogenesis and functionality of human tissuescontributesto the normal morphogenesis and functionality of human tissues

from C - terminal GFP or RFP fusionresultingfrom C - terminal GFP or RFP fusion

to highly blebbed cellsleadingto highly blebbed cells

to reduced success rates in the generation of viable embryos and stem cell lines , respectivelyleadingto reduced success rates in the generation of viable embryos and stem cell lines , respectively

to a signifiwould leadto a signifi

the growth of the stem cells in a negative mannerinfluencethe growth of the stem cells in a negative manner

to a significant decrease of the number of colonies compared with vector and control cellswould leadto a significant decrease of the number of colonies compared with vector and control cells

to an increase in cell survival after implantation44leadingto an increase in cell survival after implantation44

in both cell lines between 80 and 90 % in comparison with untreated cellsresultedin both cell lines between 80 and 90 % in comparison with untreated cells

to the development of new photoinitiators that are sensitive to visible lighthas ledto the development of new photoinitiators that are sensitive to visible light

to cell damageleadingto cell damage

to death of cells with too short telomeres thus reducing the variability of the mean values of cells in high grade dysplasia compared with adenomaleadingto death of cells with too short telomeres thus reducing the variability of the mean values of cells in high grade dysplasia compared with adenoma

to lower yieldleadingto lower yield

from ZnONP20 and Al - ZnONP20resultingfrom ZnONP20 and Al - ZnONP20

to optimal patient outcomesto contributeto optimal patient outcomes

from β1-subunit knockdownresultingfrom β1-subunit knockdown

from changes in 3D multicellular structure changes in term of for example , fractal dimension , 3D culture size , wound size , and number of cell clustersresultingfrom changes in 3D multicellular structure changes in term of for example , fractal dimension , 3D culture size , wound size , and number of cell clusters

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Smart Reasoning:

C&E

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