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to propose that insulin resistance in expanded adipose tissue results from cell growth restrictionto preventcell necrosis
the coldcould have causedcell necrosis
Both the abilityto causecell necrosis
apoptosis , a form of deathpreventscellular necrosis
e ) Subsequent neutrophil degranulationcausesendothelial cell necrosis
damaged skin cellscausingcell necrosis
tumors ... local focal temperature increasescausecell necrosis
indirectly activates the mitochondrial permeability transition porecausingcell necrosis
Photodynamic therapy ( PDT ) involves the action of photons on photosensitive molecules , where atomic oxygen or OH− molecular species are locally released on pathogenic human cells , which are mainly carcinogenicthus causingcell necrosis
physical damage(passive) caused bycell necrosis
micro pockets ... sudden high temperaturescan causecellular necrosis
Another embodiment of the invention can provide a treatment apparatusto createcell necrosis
a method and apparatus ... sufficiently lowto causecell necrosis
receiving sufficient energyto causecell necrosis
A. Inhibitors of PARP ... the first pharmacological meansto preventcell necrosis
amyloid beta protein - resistant cells.15071834 - Interaction of leishmania ( l. ) chagasi with the vero cell line.16895584 - Calpain inhibitors and antioxidants act synergisticallyto preventcell necrosis
Many available cancer drugscan causecell necrosis
Mitochondrial dysfunction and excessive ROS initiate APAP - induced liver injury [ 25subsequently triggeringcell necrosis
This second application of energy may quickly elevate the temperature enoughto causecell necrosis
This mode of application , however , does not generate sufficient heatto causecell necrosis
levels below 6.8 or above 8.0 ... sufficientto causecell necrosis
The temperature to which the tissue of the lesions 138 is heated ... sufficientto causecell necrosis
Inhibitors of parp may represent pamm account hotforex of the first pharmacological meansto preventcell necrosis
A reduction in medullary blood flow therefore creates critical hypoxiacausingcell necrosis
https://link.springer.com/article/10.1186/s12977-016-0243-3 Solamarginetriggerscellular necrosis
The affected tissue shows accumulation of lymphocytes and plasma cells that produce cytotoxic effects on the cell containing the viral antigencausingcell necrosis
an attemptto causecellular necrosis
The resulting acidic and alkaline regionscausecellular necrosis
All these factorscan causecellular necrosis
their cells ... actual human cancercausescellular necrosis
Even higher levels of ROScan causecellular necrosis
In irreversible electroporation , the cellular membranes of the cells between the electrodes are disruptedcausingcellular necrosis
referring to temperatures 8ufficiently lowto causecell necrosis
amanitins , cyclic octopeptides that inhibit protein synthesis by binding with RNA polymerasecausingcellular necrosis
the presence of pathogenscausecellular necrosis
esophagus ... severe enoughto causecellular necrosis
The virulence of fungi derives primarily from enzymes destroying tissues and fungal bodies proliferating to embolize small and midsize blood vesselsthereby causingcellular necrosis
ROS - triggered lipid peroxidation of plasma or mitochondrial membranescausescell necrosis
the probe ... approximately 80 degrees Celsiuscausescellular necrosis
most notably decreased energy productioncausecellular necrosis
This inflammatory reaction(passive) may be triggered byThis inflammatory reaction
the activation of the immune system followed by an inflammatory responsetriggersthe activation of the immune system followed by an inflammatory response
from either acute or chronic inflammationresultsfrom either acute or chronic inflammation
unmyelinated C fibers.9 Inflammation(passive) caused byunmyelinated C fibers.9 Inflammation
chronic lobular inflammation and accumulation of inflammatory cellshad causedchronic lobular inflammation and accumulation of inflammatory cells
to cellular organelle swelling , plasma membrane disruption , and release of intracellular contents ( Choe et al . , 2009leadingto cellular organelle swelling , plasma membrane disruption , and release of intracellular contents ( Choe et al . , 2009
inflammation and may kill healthy cells along with cancer cellsprovokesinflammation and may kill healthy cells along with cancer cells
overactivation of PARP-1 by negative feedback ... thus preventing cell senescencepreventsoveractivation of PARP-1 by negative feedback ... thus preventing cell senescence
the loss of cell membrane integrity and the uncontrolled release of products of cell death into the extracellular space , all of which can initiate an inflammatory response in the surrounding tissue and finally result in advanced tissue injury [ 53can causethe loss of cell membrane integrity and the uncontrolled release of products of cell death into the extracellular space , all of which can initiate an inflammatory response in the surrounding tissue and finally result in advanced tissue injury [ 53
the host 's immune systemtriggersthe host 's immune system
to intracellular content releaseleadsto intracellular content release
to loss of membrane integrityleadsto loss of membrane integrity
from a low oxygen level [ 10resultedfrom a low oxygen level [ 10
to shedding and flakingleadingto shedding and flaking
from a severe cellular insultnormally resultsfrom a severe cellular insult
its release to the extracellular environment , which stimulates the innate immune system to accumulate neutrophils and macrophages in the necrotic tissuecausesits release to the extracellular environment , which stimulates the innate immune system to accumulate neutrophils and macrophages in the necrotic tissue
to exposure of the intracellular contents including proteins such as myosin and thus an autoimmune activation , which may last 3 to 5 daysleadsto exposure of the intracellular contents including proteins such as myosin and thus an autoimmune activation , which may last 3 to 5 days
from a blunt traumaresultingfrom a blunt trauma
from individual or combined stresses , nutrient shortages and/or physiological disorderscan resultfrom individual or combined stresses , nutrient shortages and/or physiological disorders
to generalized disruption of internal homeostasis and eventually to the lysis of the cells , which give rise to an inflammatory response with the release of oxygen free radicals and activation of the microglial cellswill leadto generalized disruption of internal homeostasis and eventually to the lysis of the cells , which give rise to an inflammatory response with the release of oxygen free radicals and activation of the microglial cells
the high reflectivity(passive) caused bythe high reflectivity
several chemokine - inducing pathways which are regulated through free radical generation , nuclear factor- B activation , TNF- release , and complement activation [ 60 , 61triggersseveral chemokine - inducing pathways which are regulated through free radical generation , nuclear factor- B activation , TNF- release , and complement activation [ 60 , 61
to gangrenecan leadto gangrene
to an exacerbation of lung injuryleadingto an exacerbation of lung injury
cell membrane rupture and increase in the extracellular space where gadolinium can distributecausescell membrane rupture and increase in the extracellular space where gadolinium can distribute
in the total collapse of cognitive , muscular and nervous system functionsresultingin the total collapse of cognitive , muscular and nervous system functions
several chemokine - inducing pathways regulated through free radical generation , nuclear factor - κB activation , tumor necrosis factormay triggerseveral chemokine - inducing pathways regulated through free radical generation , nuclear factor - κB activation , tumor necrosis factor
the body 's immune system to be occupied with cleaning up those dead cellscausesthe body 's immune system to be occupied with cleaning up those dead cells
lesionscreatinglesions
from prolonged ischemia in regions of the brainresultingfrom prolonged ischemia in regions of the brain
to the release of harmful hydrolytic enzymesleadsto the release of harmful hydrolytic enzymes
to psammoma body developmentleadingto psammoma body development
to sloughing of debrisleadsto sloughing of debris
to nephron obstruction and stasis of intrarenal urineleadsto nephron obstruction and stasis of intrarenal urine
rate of RBC to fallwill causerate of RBC to fall
to inflammatory responses ( 34leadsto inflammatory responses ( 34
the cell replication(passive) caused bythe cell replication
permanent damage unless reperfusion with thrombolytics ( ‘ clot - busting ’ drugs ) is undertaken urgentlycan causepermanent damage unless reperfusion with thrombolytics ( ‘ clot - busting ’ drugs ) is undertaken urgently
probable CP(passive) caused byprobable CP
to discoloring of the skincould eventually leadto discoloring of the skin