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your cells ... free radicalscausecell injury
cost - free radicalscausecell injury
totally free radicals(passive) caused bycell injury
free radicals ... your physiquetriggercell injury
Oxygen - derived free radicalsmay causecell injury
free radicals , which are sensitive bitscan causecell injury
oxygen reenters ... free radicalscausecell injury
your everyday diet ... free radicalswill causecell injury
unstable molecules called free radicals and reduce inflammation in the body ( 6(passive) caused bycell injury
Mitochondria ... reactive oxygen species ( ROScould causecell injury
Genetic Derangements Physical agentscausingcell injury
something less free radicals ( those hazardous compoundscausecell injury
something less free radicals ( those dangerous compoundscausecell injury
something less free radicals ( those dangerous substancescausecell injury
These by - products can set off the production of free radicals within the physique ,causingcell injury
the production of oxygen free radicals and release of leukotrienes that continueto causecell injury
toxic oxygen free radicals produced by ischemia - reperfusion injury(passive) may be caused byCell injury
More ) Gini Chempakathinal Kuriakose , G. Muraleedhara Kurup Free radicalscausecell injury
Free radicals ( oxyradicals , such as superoxide , hydroxyl ions , and nitric oxidecausecell injury
The exogenous administration of CO reduced the generation of reactive oxygen species ( ROSto preventcell injury
accumulated metaboliteswill causecell injury
chemical or radiation treatment(passive) caused byCell injury
A chemical or other stimulusmay causecell injury
These mighty mites stimulate recovery from tough physical exercise and combat free radicalsto preventcell injury
free radicals occurring in response to excessive sun exposure , pollution , smoking , alcohol consumption and strenuous exercise(passive) caused bycell injury
Extreme temperature conditionscausingcell injury
chemical toxicants , cellular accumulation of Ca2 + and the generation of oxygen free radicals damage cellular components , particularly mitochondrial membranes(passive) caused bycell injury
where they are sequestered in lysosomes and interact with ribosomes , mitochondria , and other intracellular constituentsto causecell injury
Deficiency of these factors may contribute to inhibition of cell function hencecausingcell injury
the uncontrolled release of reactive oxygen species ( Rami 2009(passive) caused bycell injury
the virus infection;(19(passive) caused bycell injury
Oxidative stress and inflammationcausedcell injury
Thus , Edaravone is a free radical scavenger that acts to scavenge various types of free radicals including active oxygen as a typical exampleto preventcell injury
the prolonged ischemia(passive) caused bycell injury
established ischemia(passive) caused bycell injury
radiation - induced ROS formation(passive) caused bycell injury
oxidative stress in cardiac muscle cells(passive) caused bycell injury
reduction of brain swelling ( bruising ) ; stimulation of new blood vessel growth ; activation of idle and injured neurons ; clearance of free radicals and reduction of substancescausecell injury
oxygen ... ROScan causecell injury
signaling pathways by whichmight influencecell injury
in the premature death of cells in living tissue by autolysisresultsin the premature death of cells in living tissue by autolysis
premature cell death(passive) caused bypremature cell death
to inflammation and cell deathleadingto inflammation and cell death
to irreparable cell deathleadsto irreparable cell death
to necrotic cell deathleadingto necrotic cell death
to cell degeneration or cell deathleadingto cell degeneration or cell death
in premature cellular deathresultingin premature cellular death
in the premature death of cells in living tissues by autolysisresultsin the premature death of cells in living tissues by autolysis
the release of cytokines , especially tumor necrosis factor - alpha ( TNF - α ) which generates ROS from the tissues which in turn cause lipid peroxidation and hence higher levels of MDA seen in overweight and obese subjects studied [ 30causesthe release of cytokines , especially tumor necrosis factor - alpha ( TNF - α ) which generates ROS from the tissues which in turn cause lipid peroxidation and hence higher levels of MDA seen in overweight and obese subjects studied [ 30
to the transcriptional and/or post - translational activation of one or more BH3-only proteins that share sequence similarity in a short alpha helix ( ~9–12 a.a .leadsto the transcriptional and/or post - translational activation of one or more BH3-only proteins that share sequence similarity in a short alpha helix ( ~9–12 a.a .
to inflammation and resultant fibrosisleadingto inflammation and resultant fibrosis
to apoptotic cell death ( Berry et al . , 2010 ; Franco et al . , 2010leadingto apoptotic cell death ( Berry et al . , 2010 ; Franco et al . , 2010
to cell death , which means reduced ATP production resulting in illnesses and disease both physical and neurologicalleadsto cell death , which means reduced ATP production resulting in illnesses and disease both physical and neurological
to the premature and unprogrammed cells deathleadsto the premature and unprogrammed cells death
aging and death ... 22,23causesaging and death ... 22,23
in the death of cells in living tissue , such as with gangreneresultingin the death of cells in living tissue , such as with gangrene
to emphysema pathophysiologycontributingto emphysema pathophysiology
2 ) Necrosis : unregulated ,(passive) caused by2 ) Necrosis : unregulated ,
to eventual death and elimination resulting in less fat volumeleadsto eventual death and elimination resulting in less fat volume
in persistent well being issues akin to most cancers and cardiovascular illnessesresultingin persistent well being issues akin to most cancers and cardiovascular illnesses
to the event of heart problems and most cancerswill contributeto the event of heart problems and most cancers
official … 2008Tubulo - interstitial pathology in diabetic nephropathy is thought(passive) to be caused byofficial … 2008Tubulo - interstitial pathology in diabetic nephropathy is thought
in further local damage that increases inflammation and free radicals , eventually presenting as pancreatitisresultingin further local damage that increases inflammation and free radicals , eventually presenting as pancreatitis
Shinong Wang , Grace M Mitu , Raimund Hirschberg Tubulo - interstitial pathology in diabetic nephropathy is thought(passive) to be caused byShinong Wang , Grace M Mitu , Raimund Hirschberg Tubulo - interstitial pathology in diabetic nephropathy is thought
Diabetes News July 4th 2008 Osmotic polyuria : an overlooked mechanism in diabetic nephropathy Tubulo - interstitial pathology in diabetic nephropathy is thought(passive) to be caused byDiabetes News July 4th 2008 Osmotic polyuria : an overlooked mechanism in diabetic nephropathy Tubulo - interstitial pathology in diabetic nephropathy is thought
to permeabilization of the outer mitochondrial membrane and release of cytochrome c that triggers caspase 3 and 7 activation and apoptosis , or opening of the mitochondrial permeability transition pore in the inner membrane leading to membrane depolarization , cessation of ATP synthesis and necrosiscan leadto permeabilization of the outer mitochondrial membrane and release of cytochrome c that triggers caspase 3 and 7 activation and apoptosis , or opening of the mitochondrial permeability transition pore in the inner membrane leading to membrane depolarization , cessation of ATP synthesis and necrosis
predictable changes in cells that can be interpreted by a pathologist to suggest the underlying cause of the damage and how best to treat itcausespredictable changes in cells that can be interpreted by a pathologist to suggest the underlying cause of the damage and how best to treat it
from a disruption of one or more of the cellular components that maintain cell viabilityresultsfrom a disruption of one or more of the cellular components that maintain cell viability
inflammatory cells to migrate to the wound site and generate free radicals via a nonphagocytic NAD(P)H oxidase mechanism [ 15causesinflammatory cells to migrate to the wound site and generate free radicals via a nonphagocytic NAD(P)H oxidase mechanism [ 15
the space(passive) created bythe space
the cells die and lysis of the cell occurswill causethe cells die and lysis of the cell occurs
diverse pathologic changes in the organscausesdiverse pathologic changes in the organs
when the limits of adaptive responses of cells are exceeded or if cells are exposed to injurious agents or stressresultswhen the limits of adaptive responses of cells are exceeded or if cells are exposed to injurious agents or stress
in the release of cellular contents such as uric acid , ATP , and cellular DNAresultingin the release of cellular contents such as uric acid , ATP , and cellular DNA
nosocomial infectionmay causenosocomial infection
from some effects of oxidative stressmay also resultfrom some effects of oxidative stress
substances : causingsubstances :
to impaired fat metabolism---3 gradesleadsto impaired fat metabolism---3 grades
this complex reaction in vascularized connective tissuescan causethis complex reaction in vascularized connective tissues
a variety of vascular , inflammatory , and immune - mediated responses in addition to disturbances of growthprovokesa variety of vascular , inflammatory , and immune - mediated responses in addition to disturbances of growth