Excessive ROS accumulation in cellscan causecell injury
our cells ... free radicalsmay causecell injury
by 2 mechanisms : 1(passive) caused bycell injury
the destructive effect of pancreatic enzymes(passive) caused byacinar cell injury
hypoxic conditions ... further injuryhave causedcell injury
Responsible for more than 90 % of cellular energy , failure of mitochondrial functioncausescell injury
the agent ... likelyto causecell injury
this mechanism ... knownto causecell injury
failure of mitochondrial functioncausescell injury
by free radicals , which harm cells and DNA(passive) caused bycell injury
Harmful cellular stresscausescell injury
the human metabolic processcausecell injury
Form Here Hypoxia Deficiency of oxygencausescell injury
conditionscausingcell injury
Oxidative stress ... ROS ) production and antioxidant systemmay resultin cell injury
in the premature death of cells in living tissueresultsin the premature death of cells in living tissue
alsocausesalso
in the premature death of cellsresultsin the premature death of cells
in the premature death of cellsresultsin the premature death of cells
in the premature death of cells in living tissue by Autolysisresultsin the premature death of cells in living tissue by Autolysis
in the prematureresultsin the premature
an inflammatory responsecausesan inflammatory response
excessive mitochondrial ROS generation , elevated MDA levels , and reductions in the GPx and SOD1 activities and the GSH contentcausedexcessive mitochondrial ROS generation , elevated MDA levels , and reductions in the GPx and SOD1 activities and the GSH content
infiltration of neutrophils , which produce more ROS and cytokines as tumor necrosis factoralso causesinfiltration of neutrophils , which produce more ROS and cytokines as tumor necrosis factor
in the premature death of cellsresultsin the premature death of cells
to cell deathleadsto cell death
in traumatic cell death and apoptosisresultingin traumatic cell death and apoptosis
in the premature death of cells in living tissueresultsin the premature death of cells in living tissue
in the premature death of cells in living tissue by autolysisresultsin the premature death of cells in living tissue by autolysis
to release of ROScan leadto release of ROS
to organ rejectionleadingto organ rejection
to decreasing urine concentration capabilityleadingto decreasing urine concentration capability
to diseasewill ... leadto disease
to progressive glomerular damagecontributeto progressive glomerular damage
to loss of barrier functionleadingto loss of barrier function
in illnessmay ... resultin illness
in illnesswill ... resultin illness
to emphysemaleadingto emphysema
in abnormal glomerular permeabilityresultingin abnormal glomerular permeability
to elevated serum cTnT concentrationsleadingto elevated serum cTnT concentrations
to infiltration regarding lymphocytes as well as neutrophils within the lungleadingto infiltration regarding lymphocytes as well as neutrophils within the lung
in defective CSF dynamicsmay resultin defective CSF dynamics
in autolysis , the premature death of different cells in tissuesresultsin autolysis , the premature death of different cells in tissues
geneticcausesgenetic
in the death of cells in living tissueresultingin the death of cells in living tissue
to death of cells and living tissuesleadsto death of cells and living tissues
to cell death , which means reduced ATP production resulting in illnesses and disease both physical and neurologicalleadsto cell death , which means reduced ATP production resulting in illnesses and disease both physical and neurological
to cell deathleadsto cell death
local inflammatory responsecauseslocal inflammatory response
to cellular deathleadsto cellular death
to inflammation and cell deathleadingto inflammation and cell death