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your cells ... free radicalscausecell injury
Mitochondria ... reactive oxygen species ( ROScould causecell injury
free radicals ... your physiquetriggercell injury
totally free radicals(passive) caused bycell injury
cost - free radicalscausecell injury
scavenging of free radicalscausecellular injury
neutralize free radicals’ll causecellular injury
The production of reactive oxygen species ( ROS ) during oxidative stressmay causecellular injury
two reactive free radicalscausecellular injury
Oxygen - derived free radicalsmay causecell injury
free radicals , which are sensitive bitscan causecell injury
oxygen reenters ... free radicalscausecell injury
your everyday diet ... free radicalswill causecell injury
The exogenous administration of CO reduced the generation of reactive oxygen species ( ROSto preventcell injury
During reperfusion , oxygen free radicals are generatedcausingcellular injury
unstable molecules called free radicals and reduce inflammation in the body ( 6(passive) caused bycell injury
http://clinicaltrials.gov/show/NCT00298441 Iron can catalyze reactive oxygen species ( ROS ) formationcausingcellular injury
H2O2 ... highly reactive oxidants and free radicalscausecellular injury
something less free radicals ( those hazardous compoundscausecell injury
something less free radicals ( those dangerous compoundscausecell injury
something less free radicals ( those dangerous substancescausecell injury
overwhelming increase of free radicals , superoxide , and hydrogen peroxidecausescellular injury
toxic oxygen free radicals produced by ischemia - reperfusion injury(passive) may be caused byCell injury
These by - products can set off the production of free radicals within the physique ,causingcell injury
the production of oxygen free radicals and release of leukotrienes that continueto causecell injury
Oxidative stress and inflammationcausedcell injury
the free radicals as superoxide , hydroxyl radicals and other ROS like H2O2causingcellular injury
Ionizing Radiation Ionizing radiationcausesendothelial cell injury
which later binds with liver proteinsto causecellular injury
Free radicals ( oxyradicals , such as superoxide , hydroxyl ions , and nitric oxidecausecell injury
More ) Gini Chempakathinal Kuriakose , G. Muraleedhara Kurup Free radicalscausecell injury
neutralization ... virusescan causecell injury
The toxic metabolite binds with liver proteinsto causecellular injury
free oxygen radicals formed during reperfusion(passive) caused bycellular injury
These mighty mites stimulate recovery from tough physical exercise and combat free radicalsto preventcell injury
oxidative stress in cardiac muscle cells(passive) caused bycell injury
chemical or radiation treatment(passive) caused byCell injury
free radicals occurring in response to excessive sun exposure , pollution , smoking , alcohol consumption and strenuous exercise(passive) caused bycell injury
Biological free radicals are highly unstable molecules that react with various organic substrates such as lipids , proteinscausingcellular injury
more oxidative stress and DNA damage ... both of whichcan causecellular injury
premature cell death(passive) caused bypremature cell death
to inflammation and cell deathleadingto inflammation and cell death
in cell death ( 13resultingin cell death ( 13
to irreparable cell deathleadsto irreparable cell death
to necrotic cell deathleadingto necrotic cell death
to cell degeneration or cell deathleadingto cell degeneration or cell death
in the premature death of cells in living tissue by autolysisresultsin the premature death of cells in living tissue by autolysis
in programmed cell death within the transplant organcould resultin programmed cell death within the transplant organ
to two distinct types of cell death : apoptosis or necrosis.6 Apoptosismay leadto two distinct types of cell death : apoptosis or necrosis.6 Apoptosis
cell death within high stress level of cell structurescausescell death within high stress level of cell structures
excessive ROS production ... triggering a state of oxidative stress that can lead to neuronal cell deathcan causeexcessive ROS production ... triggering a state of oxidative stress that can lead to neuronal cell death
to apoptotic cell death ( Berry et al . , 2010 ; Franco et al . , 2010leadingto apoptotic cell death ( Berry et al . , 2010 ; Franco et al . , 2010
to cell death , which means reduced ATP production resulting in illnesses and disease both physical and neurologicalleadsto cell death , which means reduced ATP production resulting in illnesses and disease both physical and neurological
in premature cellular deathresultingin premature cellular death
to liver failure and death ( Refs . 4 and 7can leadto liver failure and death ( Refs . 4 and 7
inflammation of the mandiblecausesinflammation of the mandible
inflammation of the mouthcausesinflammation of the mouth
in either necrosis or apoptosismay resultin either necrosis or apoptosis
to a release of the lysosomal enzymes ... causing _ _ _leadsto a release of the lysosomal enzymes ... causing _ _ _
to inflammation and resultant fibrosisleadingto inflammation and resultant fibrosis
the release of cytokines , especially tumor necrosis factor - alpha ( TNF - α ) which generates ROS from the tissues which in turn cause lipid peroxidation and hence higher levels of MDA seen in overweight and obese subjects studied [ 30causesthe release of cytokines , especially tumor necrosis factor - alpha ( TNF - α ) which generates ROS from the tissues which in turn cause lipid peroxidation and hence higher levels of MDA seen in overweight and obese subjects studied [ 30
in the premature death of cells in living tissues by autolysisresultsin the premature death of cells in living tissues by autolysis
2 ) Necrosis : unregulated ,(passive) caused by2 ) Necrosis : unregulated ,
to necrosis ATP depletionleadto necrosis ATP depletion
in hypoxia and subsequently necrosisresultingin hypoxia and subsequently necrosis
the release of mediators of inflammationtriggersthe release of mediators of inflammation
to necrosis especially to the kidneys and heartleadingto necrosis especially to the kidneys and heart
to local or systemic symptomsleadingto local or systemic symptoms
to electromechanical changes causing inflammation and enlargement of internal organs and bone ... leading to mechanical stress on the more superficial structures , which produce symptomsleadsto electromechanical changes causing inflammation and enlargement of internal organs and bone ... leading to mechanical stress on the more superficial structures , which produce symptoms
to inflammation in the blood vessels , which in turn may lead to atherogenesis , which can result in ischemic injuryleadsto inflammation in the blood vessels , which in turn may lead to atherogenesis , which can result in ischemic injury
to the transcriptional and/or post - translational activation of one or more BH3-only proteins that share sequence similarity in a short alpha helix ( ~9–12 a.a .leadsto the transcriptional and/or post - translational activation of one or more BH3-only proteins that share sequence similarity in a short alpha helix ( ~9–12 a.a .
inflammation of the left side ( arrowcausesinflammation of the left side ( arrow
following oxidative stressprovokesfollowing oxidative stress
from some effects of oxidative stressmay also resultfrom some effects of oxidative stress
recovers memory learning injury(passive) caused byrecovers memory learning injury
activity of memory learning injury(passive) caused byactivity of memory learning injury
to the premature and unprogrammed cells deathleadsto the premature and unprogrammed cells death
aging and death ... 22,23causesaging and death ... 22,23
in an imbalance in endothelium - derived mediators , favouring vasoconstrictionmay resultin an imbalance in endothelium - derived mediators , favouring vasoconstriction
to serious health complications such as cancer and chronic heart diseasecan leadto serious health complications such as cancer and chronic heart disease