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Abstract Background Chemoresistance acquisitionmay influencecancer cell biology
Nora Hinsch Background Chemoresistance acquisitionmay influencecancer cell biology
classes ... ap main essaycausescancer biology
authors Jindrich Cinatl Chemoresistance acquisitionmay influencecancer cell biology
of several areas that include determining tumours ’ characteristics and analysing molecules and genomic patterns that relate to the causation of cancer(passive) is composedCancer computational biology
the tumor microenvironment , which impacts disease prognosis and therapeutic interventions(passive) is influenced byCancer biology
Scott W. Lowe Chemical Biology(passive) led byCancer Biology & Genetics –
novel patterns of GalNAc - T glycosylation on tumour - derived proteinscould influencecancer biology
Joseph Roti Roti , PhD(passive) led byCancer Biology scientists
Interactions Via Pan - Cancer Analysis MiRNAs have long been proposedto contributeto cancer biology
to illustrate basic aspects of cancer development(passive) is designedBiology of Cancer
This course(passive) is designedCancer Biology
Attention to the functioning of the cell as a biochemical system and to developmental processes in embryogenesis , cell differentiation , andhas ledcancer biology
The renin - angiotensin system ( RASinfluencescancer biology
the underlying processescontributeto Cancer Biology
Mutational SignaturesLeadto Cancer Biology
Karin Rodland(passive) led bycancer biology
quality experimental and translational studies on the mechanisms by whichmight influencecancer cell biology
Improved understanding of how transcription factors affectmay leadcancer biology
the pancreatic hormone insulinresultsin cancer biology
the individual factorsinfluencingcancer biology
Dr Adam Patterson(passive) was led byCancer biology
Neuroendocrineinfluenceson cancer biology
to identifymay influencecancer cell biology
pathways leading to the modulation of gene expressioncontributesto cancer biology
other metabolic changesmay contributeto cancer biology
to explaincontributesto cancer biology
Cross talk between SR and membrane - initiated signaling events suggests a mechanism for coordinate regulation of gene subsets by mitogenic stimuli in hormonally responsive normal tissues ; such cross talk is suspectedto contributeto cancer biology
changes in the epigenome(passive) is profoundly influenced byCancer biology
Regulatory factors identified in vascular disease and repairmay also contributeto cancer biology
glutamine and glutamatecontributeto cancer biology
Head ofLeadCancer Biology
the physical sciencescontributingto cancer biology
to become one of the largest health and life sciences clusters in the southern hemisphere(passive) is setCancer Biology
hpvdoes ... causecancer biology
Both upstream PAR - mediated signaling and downstream coagulation eventsmay thus contributeto cancer biology
research on p53influencedcancer biology
Pitt faculty and graduate students(passive) led bycancer biology
Agemay also influencecancer biology
deregulation of this pathwayinfluencescancer biology
to Aetiology , which can lead to the screening of it or prevention by therapeutics , or clinical application of GWAScan leadto Aetiology , which can lead to the screening of it or prevention by therapeutics , or clinical application of GWAS
a standard of best practices that maximize reproducibility and facilitate an accurate accumulation of knowledge , enabling potentially impactful findings to be effectively built upon by the scientific communityto seta standard of best practices that maximize reproducibility and facilitate an accurate accumulation of knowledge , enabling potentially impactful findings to be effectively built upon by the scientific community
in the development or improvement of diagnostic techniques that could better and earlier detect melanocytic lesions ... in order to give patients suffering from melanoma the best chance of prolonged survivalmight resultin the development or improvement of diagnostic techniques that could better and earlier detect melanocytic lesions ... in order to give patients suffering from melanoma the best chance of prolonged survival
to selective cancer cell deathleadsto selective cancer cell death
and advances ... including how gene mutations drive the development and spread of cancer cells ... and how tumors communicate with their encompassing conditioncreatesand advances ... including how gene mutations drive the development and spread of cancer cells ... and how tumors communicate with their encompassing condition
to new targets of therapymay leadto new targets of therapy
to the increase in the number of new agents under developmenthas ledto the increase in the number of new agents under development
to a discovery of new mechanisms to understand cancer physiology as well as response against anti - cancer treatmentleadto a discovery of new mechanisms to understand cancer physiology as well as response against anti - cancer treatment
the stage for others to wonder why all cancers looked the samedid setthe stage for others to wonder why all cancers looked the same
to the selective inhibition of rate - limiting targets in the progression of many chemotherapy resistant cancers ( e.g. Cetuximab ( anti - EGFR ) - colorectal cancerhas ledto the selective inhibition of rate - limiting targets in the progression of many chemotherapy resistant cancers ( e.g. Cetuximab ( anti - EGFR ) - colorectal cancer
to the discovery of novel treatmentsmay leadto the discovery of novel treatments
in uncontrolled growth and proliferationresultingin uncontrolled growth and proliferation
about the intricate interactions that take place beforeto discoverabout the intricate interactions that take place before
to potential novel therapeutics for the treatment of cancer patients in various stagescan leadto potential novel therapeutics for the treatment of cancer patients in various stages
in the development of novel treatment strategiesmight resultin the development of novel treatment strategies
to race - based disparitiesmay contributeto race - based disparities
to theories about how aneuploidy drives cancerleadingto theories about how aneuploidy drives cancer
to individualized treatmentsis also leadingto individualized treatments
for High - Density T Cell Culture FDA - cleared use for human ex vivo tissue and cell culture processing applicationsDesignedfor High - Density T Cell Culture FDA - cleared use for human ex vivo tissue and cell culture processing applications
to germline - somatic interactionscan also leadto germline - somatic interactions
to the devel - opment of new viable treatmentscould leadto the devel - opment of new viable treatments
in transformative new therapiesresultingin transformative new therapies
to disparities in cancer incidence and outcomecontributesto disparities in cancer incidence and outcome
up to 90 % of cancer deathscauseup to 90 % of cancer deaths
us to new therapeutic approaches that will be far more effectivewill ... leadus to new therapeutic approaches that will be far more effective
in long - term survival with liver resectionmay resultin long - term survival with liver resection
to safer and more effective therapiescould leadto safer and more effective therapies
the more preciselycan preventthe more precisely
from these various aspectsresultingfrom these various aspects
to new diabetes treatment Researchers at the University of Pennsylvania School of Medicinecould leadto new diabetes treatment Researchers at the University of Pennsylvania School of Medicine
to the creation of a drug development platform for non - toxic multivalent molecularly directed therapiesledto the creation of a drug development platform for non - toxic multivalent molecularly directed therapies
to better detection , treatment and preventionleadingto better detection , treatment and prevention
to new approaches in clinical practiceleadingto new approaches in clinical practice
in several collaborative papers with the Lowengrub groupresultedin several collaborative papers with the Lowengrub group
out to repeat key experiments from 50 high - profile cancer papers , and so assess the extent to which published results can be replicatedsetout to repeat key experiments from 50 high - profile cancer papers , and so assess the extent to which published results can be replicated
offspring 's neural stem cells(passive) caused byoffspring 's neural stem cells
opportunities for scientists to engage our scholars with hands on projects and experiments and discuss concepts of cancer biologycreateopportunities for scientists to engage our scholars with hands on projects and experiments and discuss concepts of cancer biology
me to the Presidents Cancer Panelledme to the Presidents Cancer Panel
out to replicate 50 studiessetout to replicate 50 studies
August 28 , 2014 |resultsAugust 28 , 2014 |