clude segmental hyperinflation and atelectasis(passive) caused byclude segmental hyperinflation and atelectasis

to atelectasis or infectionleadingto atelectasis or infection

recurrent symptoms of dyspnoea and wheezing(passive) caused byrecurrent symptoms of dyspnoea and wheezing

secondary atelectasismay causesecondary atelectasis

in pneumonia or atelectasisresultingin pneumonia or atelectasis

resorptive atelectasiscausingresorptive atelectasis

from aspiration of food particlesresultingfrom aspiration of food particles

to collapse of lung tissue by absorption of air from the distal lungleadsto collapse of lung tissue by absorption of air from the distal lung

usually a laryngeal keelpreventedusually a laryngeal keel

from extrinsic compression as in hilar lymphadenopathy or tumor of neoplastic origincan resultfrom extrinsic compression as in hilar lymphadenopathy or tumor of neoplastic origin

atelectasis Poor prognostic factorcausingatelectasis Poor prognostic factor

to infection with inflammation ( distal to the obstructionleadingto infection with inflammation ( distal to the obstruction

to atelectasis of the lower lobe or complete lungleadingto atelectasis of the lower lobe or complete lung

to a “ collapse - consolidation ” picture on chest radiogram , usually with minimal symptoms or signscan leadto a “ collapse - consolidation ” picture on chest radiogram , usually with minimal symptoms or signs

a transient and usually repeated attack of breathlessness(passive) caused bya transient and usually repeated attack of breathlessness

in atelectasis , impaired lung mechanics and gas exchangeresultingin atelectasis , impaired lung mechanics and gas exchange

to secondary infections which may complicate to abscess formationleadsto secondary infections which may complicate to abscess formation

from alveolar hypocapnia , which will ensure normal gas exchange through intact areas of the lungs and will quickly eliminate significant cellular and arterial hypercapniaresultingfrom alveolar hypocapnia , which will ensure normal gas exchange through intact areas of the lungs and will quickly eliminate significant cellular and arterial hypercapnia

from bronchospasmresultingfrom bronchospasm

to localized hypoxia , thenleadsto localized hypoxia , then

air from reaching alveolipreventsair from reaching alveoli

from a foreign body , plug of tenacious mucopurulent material , tumour and extrabronchial occlusion by lymph nodesmay resultfrom a foreign body , plug of tenacious mucopurulent material , tumour and extrabronchial occlusion by lymph nodes

to air trapping and respiratory distressleadingto air trapping and respiratory distress

to collapse or hyperinflationleadingto collapse or hyperinflation

as a result of non - malignant endobronchial tumorscausedas a result of non - malignant endobronchial tumors

postobstructive pneumonia , which may lead to abscess formationcausespostobstructive pneumonia , which may lead to abscess formation

Recurrent or slow resolving pneumonia(passive) caused byRecurrent or slow resolving pneumonia

to bronchiectasisdoes ... leadto bronchiectasis

clinical manifestationscausingclinical manifestations

the collapse of the lungs which is a ramification of bronchuscan causethe collapse of the lungs which is a ramification of bronchus

to respiratory failureleadingto respiratory failure

the reflux(passive) caused bythe reflux

from inhalation or airborne bacteria and fungi * dresultingfrom inhalation or airborne bacteria and fungi * d

chronic lung inflammatory alteration(passive) caused bychronic lung inflammatory alteration

to gradual asphyxiationmay leadto gradual asphyxiation

a poorly ventilated region(passive) e.g. ... caused bya poorly ventilated region

oxygen to get into the distal portions of the lungswill preventoxygen to get into the distal portions of the lungs

Secondary lung abscesses(passive) are caused bySecondary lung abscesses

to abscess formation distallyleadsto abscess formation distally

in an oxygen deficiencyresultingin an oxygen deficiency