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The N and Q lineagescontributetwo blast cells
any drugcausingBLAST CELLS PRESENT
THERAPYcausesBLAST CELLS PRESENT
other drugscauseBLAST CELLS PRESENT
a possibilitycould causeBlast Cells Present
you evaluatecausesBLAST CELLS PRESENT
you evaluateACETAMINOPHEN causesBLAST CELLS PRESENT
any drugcausingBLAST CELLS
DrugscausedBlast Cells
you evaluatecausesBLAST CELLS
by the GATAT1 mutation in the presence of the extra chromosome 21(passive) caused byblast cells
a rare extra hematopoietic carcinomacomposedof blast cells
by the GATAT1 mutation in the presence of trisomy 21(passive) caused byblast cells
by the GATAT1 mutation in the presence of trisomy 21 ( or the presence of extra chromosome(passive) caused byblast cells
by a reduction in the expression of Th17 cell - related cytokines(passive) may be prompted byTh17 ) cells
by a reduction from the expression of Th17 cell - related cytokines(passive) can be caused byTh17 ) cells
for ease of use and reliable testing results(passive) are designedSquarePak cells
The BCR - ABL genecausesmyeloid cells
either(passive) is causedTh1 cells
the prolonged activation of Th2 andcausesTh17 cells
by acute stress or infection(passive) caused bythymic cells
by the transcription element(passive) caused byTH1 ) cells
the transcription element(passive) caused byTH1 ) cells
by the reported reduction by GSK3 inhibitors of IL-12(passive) could be caused byTh1 cells
by aberrant lymphocyte development(passive) is caused byTh17 cells
by the transcription element ( T - bet(passive) caused byTH1 ) cells
recently(passive) are ... discoveredTh17 cells
Adenosineinfluencesmyeloid cells
Adenosineinfluencesmyeloid cells
Researchershave discoveredautoreactive cells
Researchersdiscoverautoreactive cells
such as non - enzymatic glycationmay ... influencemicrovascular cells
by disturbance in the neural circuits between the limbic system and the cortex of the brain(passive) caused bymicroglial cells
Hcy(passive) caused bymicroglial cells
Failure to repress some or all of these targetsleadsxbp1 cells
Researchers from the University of Copenhagenhave discoveredautoreactive cells
such loopsto influencegranule cells
SSA or SNA(passive) caused byneurosphere cells
SSA and SNA(passive) caused byneurosphere cells
the release of pro - inflammatory cytokines during inflammation or tissue damage(passive) caused byautoreactive cells
to hypertensioncan ... contributeto hypertension
within the bone marrowdiscoveredwithin the bone marrow
in the bone marrowdiscoveredin the bone marrow
accumulation of > 20 % blast cells in the bone marrowcausingaccumulation of > 20 % blast cells in the bone marrow
a type of cancer ( specifically , a tumor(passive) caused bya type of cancer ( specifically , a tumor
muscle contractioncan causemuscle contraction
in a mesenchymal phenotyperesultedin a mesenchymal phenotype
to an overall very bad prognosis , which is also in part due to limited therapeutical optionsleadingto an overall very bad prognosis , which is also in part due to limited therapeutical options
in a significant decrease in the frequencyresultedin a significant decrease in the frequency
tissue damage by extravasation and infiltration of surrounding tissue , for which the formation and expression of matrix metalloproteinases ( MMP ) is necessarycausetissue damage by extravasation and infiltration of surrounding tissue , for which the formation and expression of matrix metalloproteinases ( MMP ) is necessary
to cell detachmentleadingto cell detachment
to direct inhibition of adenylyl cyclaseleadingto direct inhibition of adenylyl cyclase
a reduction of Th2 cellscausinga reduction of Th2 cells
to inflammation and diseaseleadingto inflammation and disease
the activity of Tfh cellsinfluencesthe activity of Tfh cells
the activity of Tfh cellsinfluencethe activity of Tfh cells
to diseasecontributingto disease
inflammationcausedinflammation
to the inflammationmay contributeto the inflammation
to the activation of macrophagesleadto the activation of macrophages
to enhanced activation of CTL immune responsesleadingto enhanced activation of CTL immune responses
to an IFN - productionleadingto an IFN - production
to the development of autoimmune diabetescontributesto the development of autoimmune diabetes
to macrophage activationleadingto macrophage activation
to the development of EAEleadto the development of EAE
to the development of autoimmune diseasescontributeto the development of autoimmune diseases
to Th2leadingto Th2
to an imbalance of Th1 / Th2leadingto an imbalance of Th1 / Th2
to autoimmune chronic GvHDcontributedto autoimmune chronic GvHD
to cell - mediated immunityleadto cell - mediated immunity
alsocontributealso
immunopathology and organ - specific autoimmune diseasemay causeimmunopathology and organ - specific autoimmune disease
to toxic shock - like syndromecontributeto toxic shock - like syndrome
to the creation of a Th2 environmentleadingto the creation of a Th2 environment
from CD4 + T helper precursor cellsoriginatefrom CD4 + T helper precursor cells
asthmacauseasthma
excessive cytokine release , especially IFN - g and TNF - acausingexcessive cytokine release , especially IFN - g and TNF - a
toward GVHD pathogenesiscontributetoward GVHD pathogenesis
in an increased immune responseresultingin an increased immune response
to uveitis and scleritiscontributeto uveitis and scleritis