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Toxicity ... which have been shownto causemyotoxicity in vitro
The sulfonamide compounds have been shownto causehemolysis in vitro
For example , HgCl2 was shownto causea Th2 skewing in vitro
The ability of many saponinsto causehemolysis in vitro
the apparent ability of LSDto causevasospasm in vitro
the replication of SARS - CoV-2 , the viruscausesCOVID-19 ... in vitro
This switch ... sufficientto triggermorphotype interconversion in vitro
activity against the viruscausesCOVID-19 in vitro
Other F - BAR domains have been shownto causeliposome tubulation in vitro
R518X nonsense mutation has been shownto causeKv7.1 haploinsufficiency , in vitro
for convenience cells to implyoriginatedfrom PGCs in vitro
pathwayscreatepluripotency in vitro neag
Leuconostoc oeni Elios 1 were shownto significantly influencethrombolysis in vitro
The ability of CARPs to act as E3 ubiquitinpromptedligases in vitro
Exposure of mesangial cellsleadsto IL-8 in vitro
metabolic inhibition in astrocytes ... sufficientto causeKv2.1 dephosphorylation in vitro
the many factorsinfluenceorganogenesis in vitro
discovery of the problems associated with abnormal pseudocholinesterase activity may have been shownto triggertubulogenesis in vitro
Virología dc.title An analogue of the antibiotic teicoplaninpreventsflavivirus entry in vitro
5 ) the blockade of GLT-1 ... sufficientto causeKv2.1 dephosphorylation in vitro
the ROC1–Cul3–KLHL20 complex ... sufficientto triggerDAPK ubiquitination in vitro
the deletion of PIKE - A per se ... sufficientto preventadipogenesis in vitro
proteins such as the reason for this antiviral activity against salmonellacausessalmonellosis ... in vitro
the use of antiviral agents with activity against salmonellacausessalmonellosis ... in vitro
CONCLUSION DARAcausespanreactivity in vitro
Both 5beta,6beta - epoxycholesterol and its metabolite , cholestan-6-oxo-3,5-diol , have been shownto causecytotoxicity in vitro
Tocris Summary for HPSE Gene Antiangiogenic agents ... compoundspreventangiogenesis in vitro
from early myeloid cells ( CD34 + and CD14 + cells(passive) can be triggeredReactivation of HCMV in vitro
Three major factorsinfluencesporulation in vitro
ISP-1 ... conditionspreventproteolysis in vitro
Peptides derived from the first exon of Tat , including Tat46 - 60 , Tat37 - 72 , and Tat31 - 61 , have been shownto causeneurotoxicity in vitro
the endoplasmic reticulum - Golgi intermediate compartment ( ERGIC ... sufficientto triggerLC3 lipidation in vitro
Antibodies raised against the proteincan preventadherence in vitro
the calculated concentration of peroxynitrite present at the resistance vessel level ... sufficientto causevasorelaxation in vitro
Long - term exposure of freshly isolated human CD4 + T cells tocausedIL-12 in vitro
Treatment of porcine aortic smooth muscle cells with phorbol esterspreventsnodulation in vitro
the effect of GFP - P loop K - A p60 on wild - type p60 ... sufficientto preventmicrotubule severing in vitro
Photo - Inactivation of Fungal PathogensCauseOnychomycosis In Vitro
This hypothesis ... the association of the s1 type of VacAcausesvacuolation in vitro
the biochemical features of the three main pathogenscauseAOM in vitro
these effectspreventedthese effects
rats(passive) caused byrats
neurotoxicity(passive) caused byneurotoxicity
to its degradation by the proteasome ( Probableleadingto its degradation by the proteasome ( Probable
to increased percentages of IL-1β – expressing monocytes and mDCs from seropositive versus seronegative subjectsledto increased percentages of IL-1β – expressing monocytes and mDCs from seropositive versus seronegative subjects
299 - 304 , 1982 Efficacy of some fungicides against maydis leaf blight of maize(passive) caused by299 - 304 , 1982 Efficacy of some fungicides against maydis leaf blight of maize
one form of DNA damage ( DNA strand breaks(passive) caused byone form of DNA damage ( DNA strand breaks
to activation of endothelial cells , platelets and prothrombinleadingto activation of endothelial cells , platelets and prothrombin
spleen lymphocytes ( including NK cells(passive) caused byspleen lymphocytes ( including NK cells
The Endo - Lysosomal System of Brain Endothelial Cells(passive) Is Influenced byThe Endo - Lysosomal System of Brain Endothelial Cells
a concentration - dependent net increase in superoxide anion in mitochondria from Sod2(+/- ... but not Sod2(+/+ ) mice [ 25].Furthermore , the nimesulide - treated mutant mice exhibited increased hepatic cytosolic levels of cytochrome c and caspase-3 activity , as well as increased numbers of apoptotic hepatocytes [ 25causeda concentration - dependent net increase in superoxide anion in mitochondria from Sod2(+/- ... but not Sod2(+/+ ) mice [ 25].Furthermore , the nimesulide - treated mutant mice exhibited increased hepatic cytosolic levels of cytochrome c and caspase-3 activity , as well as increased numbers of apoptotic hepatocytes [ 25
to apoptosis of pulmonary endothelial cells in a dose - dependent mannerledto apoptosis of pulmonary endothelial cells in a dose - dependent manner
from incidental stimulation of nondopaminergic cells or from abnormal synapse formation in cultureresultedfrom incidental stimulation of nondopaminergic cells or from abnormal synapse formation in culture
in high cell mortality with a small fraction of surviving cells showing gene transferoften resultsin high cell mortality with a small fraction of surviving cells showing gene transfer
neural precursor cells , which are then used to repopulate the damaged tissue without risk of immunologic rejectionto createneural precursor cells , which are then used to repopulate the damaged tissue without risk of immunologic rejection
340 - 342 , 1984 Irreversible stem cell damage(passive) caused by340 - 342 , 1984 Irreversible stem cell damage
loss of their ability to undergo apoptosis as well as other abnormal features ... suggesting that accumulation of senescent cells could contribute to aging and age - related diseasescausesloss of their ability to undergo apoptosis as well as other abnormal features ... suggesting that accumulation of senescent cells could contribute to aging and age - related diseases
genotype - specific inhibition of cell proliferation in neuroblastoma cells ... whereas overexpression of BARD1β led to the transformation of non - malignant murine fibroblast [ 51,77causedgenotype - specific inhibition of cell proliferation in neuroblastoma cells ... whereas overexpression of BARD1β led to the transformation of non - malignant murine fibroblast [ 51,77
the split - luciferase complementation(passive) triggered bythe split - luciferase complementation
the innate immune response(passive) triggered bythe innate immune response
immediatelyleadsimmediately
in active virus particles ( 17resultingin active virus particles ( 17
both inactivation of transforming activity and the induction of mutationscausedboth inactivation of transforming activity and the induction of mutations
inhibition of GBM cell growthcausedinhibition of GBM cell growth
up HIV-1 CA - NC complexes through the use of two different assays [ 1,2setup HIV-1 CA - NC complexes through the use of two different assays [ 1,2
probes using the Digoxigenin RNA labeling kit ( Roche # 11175025910to createprobes using the Digoxigenin RNA labeling kit ( Roche # 11175025910
human eosinophils(passive) triggered byhuman eosinophils
in increased mitochondrial ROS production , responsible for reduced cell proliferationresultedin increased mitochondrial ROS production , responsible for reduced cell proliferation
assayresultsassay
cellular stress(passive) caused bycellular stress
the acidic environment(passive) created bythe acidic environment
in decreased bone resorption in vivo by regulating RANKL / OPG production and ephrins mRNAsresultedin decreased bone resorption in vivo by regulating RANKL / OPG production and ephrins mRNAs
the SC cell to dedifferentiatemight causethe SC cell to dedifferentiate
to reduction of the Cl channel activityleadingto reduction of the Cl channel activity
to a fourfoldleadsto a fourfold
rescued angiogenesis(passive) caused byrescued angiogenesis
CYP3A4 metabolism(passive) caused byCYP3A4 metabolism
the biofilms(passive) created bythe biofilms
neuronal injury(passive) caused byneuronal injury
NO synthetase decreases(passive) caused byNO synthetase decreases