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liver conditionsresultin impaired bile acid flow and retention of bile acids in the liver
These diseasesresultin impaired bile acid flow and retention of bile acids in the liver
local pathological damage and inflammationcan resultin impaired synthesis of bile acids and inflammation - induced cholestasis
Cystic fibrosiscausesproblems with the normal secretion and function of bile
Patientscan causeperianal disease of these cells in bile and skin reactions
toxic dosecould causebile acid , fat , amino acid , and energy metabolic disorders
in our mice loss of bile acid transportersresultsin impaired secretion of bile acids leading to hepatocyte necrosis
the genetic factorscontributingto the levels of endogenous bile acids and pharmacokinetics of therapeutic ursodeoxycholic acid
Combined conditionscausingthe reduction of bile flow and the retention of bile acids
an impairment of bile flow in the livercausingretention of bile acids and injury
By the suppression of intestinal FXR / FGF-15 signaling , hepatic bile - acid synthesis gene expression was increasedleadingto an elevation of secondary and hydrophobic bile acids in liver , bile , and intestine
Subnormothermic Machine Perfusion ( SNMPLeadsto Increased Bile Production and Secretion of Hydrophilic Bile Acids
These diseasesresultin impaired bile acid flow and retention of bile
Bile duct destructionleadsto retained bile acids and foamy hepatocyte degeneration ( 2
a single enzyme defectleadingto bile acid synthesis and peroxisomal disorders
sex hormonesadversely influencehepatic bile secretion and gallbladder function
the changes in chromatin structure and the regulatory protein complex at the CYP7A1 promoterleadto its repression by bile acids and bile acid - induced SHP
Female sex hormonesadversely influencehepatic bile secretion and gallbladder function
These abnormalitiesleadto a build - up of bile acids ( components of bile
Factorsinfluencingformation and excretion of bile acids
LSGresultedin enhanced faecal excretion of nonesterified fatty acids and bile acids
the effects of various agentsinfluencegastric acidity and bile acids
various agentsinfluencegastric acidity and bile acids
Damaged or absent bile ductsresultin bile and bile acids being trapped inside the liver
C miceresultedfrom the overproduction and secretion of bile acids
gallstones ( cholelithiasiscomposedof lecithin and bile acids
have gallstones ( cholelithiasiscomposedof lecithin and bile acids
cholelithiasiscomposedof lecithin and bile acids
BAD ... specific itemsmight be causingthe troubles with bile acids
Foodcausesbile acids to be secreted
catanionic templating systemscomposedof bile acids and CTAB
to progressive liver damage that may ultimately result in liver failureleadingto progressive liver damage that may ultimately result in liver failure
to progressive liver damage that may ultimately result in liver failureleadingto progressive liver damage that may ultimately result in liver failure
to progressive liver damage and , ultimately , liver failureleadingto progressive liver damage and , ultimately , liver failure
to diarrhea in patientshave ... leadsto diarrhea in patients
to diarrhea in patientsleadsto diarrhea in patients
to diarrhealeadsto diarrhea
neurological symptomscausesneurological symptoms
damage(passive) caused bydamage
in pancreatic insufficiency andresultin pancreatic insufficiency and
energy and glucose homeostasiscan influenceenergy and glucose homeostasis
intense itchingcausesintense itching
Biliary disease(passive) is caused byBiliary disease
the drug transport across the biological membranescan influencethe drug transport across the biological membranes
to lung injuryto contributeto lung injury
from dysregulated bile acid synthesis or transitresultfrom dysregulated bile acid synthesis or transit
cholestasis and often , liver inflammation and damagecausingcholestasis and often , liver inflammation and damage
from bile acid malabsorptionresultfrom bile acid malabsorption
the intestinal microorganismscan influencethe intestinal microorganisms
bacteria is the resultcausingbacteria is the result
bacteriacausingbacteria
cells from the cardia to travel to the esophaguscausedcells from the cardia to travel to the esophagus
to an increase in the production of bile acids in the liverleadingto an increase in the production of bile acids in the liver
to the upregulation of LDL receptors and a reduction of LDL - cholesterol levelsleadingto the upregulation of LDL receptors and a reduction of LDL - cholesterol levels
to a healthier liver and pancreasmay ... leadto a healthier liver and pancreas
inflammationcauseinflammation
to the symptoms.[4may contributeto the symptoms.[4
ongoing damage as well as lack of symptom reliefcan causeongoing damage as well as lack of symptom relief
an increase in the permeability of the intestinal mucosa to oxalate also contributing to hyperoxaluriacausean increase in the permeability of the intestinal mucosa to oxalate also contributing to hyperoxaluria
to the formation of taurine and glycine conjugatescan leadto the formation of taurine and glycine conjugates
to development of the gallstonescan also contributeto development of the gallstones
to metabolic acidosiscan leadto metabolic acidosis
in damage and scarring of the liver cells ( cirrhosisresultingin damage and scarring of the liver cells ( cirrhosis
to hyperdynamic circulation in cirrhosiscan leadto hyperdynamic circulation in cirrhosis
in damageresultingin damage
in cirrhosisresultingin cirrhosis
to bouts of uncontrollable itchingledto bouts of uncontrollable itching
to regulate glucose and lipid metabolismcontributingto regulate glucose and lipid metabolism
the itchingcausethe itching
to overall bile resistance levelscontributesto overall bile resistance levels
reduced cholesterol elimination ( 7causingreduced cholesterol elimination ( 7