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Lack of oxygen in the brainalso causesATP exhaustion and cellular breakdown from cytoskeleton damage and nitric oxide production
CL 303268 uncouples oxidative phosphorylation at the mitochondriaresultingin disruption of production of ATP , cellular death , and
in turncould leadto cellular ATP depletion and cell injury
while more extensive injurycausesATP depletion results in cell necrosis [ 27
defects in electron transportcausingdeficient ATP production and cellular injury
The caspases ... the proteinscausecell death in the presence of cellular energy ( ATP
more extensive injurycausesATP depletion results in cell necrosis
while more extensive injurycausesATP depletion results in cell necrosis
Ischemiaresultsin cell hypoxia and depletion of cellular adenosine triphosphate ( ATP
It is toxic and acts by interfering with oxidative phosphorylation in mitochondriacausingdepletion of ATP and cell death.[1
acts by interfering with oxidative phosphorylation in mitochondriacausingdepletion of ATP and cell death.[1
interfering with oxidative phosphorylation in mitochondriacausingdepletion of ATP and cell death.[1
This defectleadsto decreased cellular energy ( ATP ) production
High levels of oxidative stresscan causeATP depletion , necrosis , and apoptosis
ROScan causefatal depletion of mitochondrial energy ( ATP
CoQ10resultingin the production of ATP Cellular Energy
alpha lipoic acid ... vital reactionsleadto the production of cellular energy ( ATP
irreversible structural damage to mitochondriaresultingin cytoplasmic ATP depletion and cell death
It is toxic and acts by interfering with oxidative phosphorylation in mitochondria by inhibiting complex Ileadingto the depletion of ATP and cell death
L - Carnitine ( 25 mg ) , assists in the transport of fat through the cell membrane and into the mitochondria within the cell , where the fats are oxidizedresultingin the production of cellular energy ( ATP
It is toxic and acts by interfering with oxidative phosphorylation in mitochondriacausingdepletion of ATP and cell death
Competitive inhibition of this enzyme was foundto causeATP depletion and cell death
complex I of the electron transport chaincausingdepletion of ATP and cell death
renal blood flow ... a levelresultsin severe cellular ATP depletion
aging - dependent changes , thought due to energy depletion , rotenone , an inhibitor of complex Icausesloss of cellular ATP ... in
the toxin(passive) caused byATP depletion and cell death
a factcausesATP release from glial cells
reportedto causeATP depletion in hepatocellular carcinoma cells
by a mechanism that has been described for isolated mitochondria ... after an increase in Ca2 + levels(passive) may be caused byATP - induced cell death
mechanismsinfluenceATP release from cells
a microorganism invades a cellcausingdamage to ATP synthase
lens epithelial cellsleadingto severe depletion of intracellular ATP
increased 2'-5 ' A synthetase and RNAse L activityleadingto a depletion of cellular ATP , the energy
blood glucoseleadingto depletion of Adenosine Triphosphate ( ATP ) in cells
mPTP openingmay causeATP depletion and cellular necrosis
has the highest electronegativity?sulfuroxygenhydrogennitrogencarbon10.Suppose a microorganism invades a cellcausingdamage to ATP synthase
Two critical factors / conditions seem to determine this selectivity of vitamin C - mediated cancer cell killing : ( 1 ) the selective accumulation of DHA in cancer cells via a GLUT1-dependent transport mechanism , leading to intracellular GSH depletion and ROS accumulation , and ( 2 ) ROS - mediate inhibition of the glycolytic enzyme GAPDH in the background of oncogene - induced metabolic reprogramming ( i.e. , dependence on glycolysis for survivalresultingin ATP depletion and cell death
Ischemiacausesdepletion of ATP in cells in the affected area
This defectleadsto decreased production of cellular energy ( ATP
environmental stressescauseATP depletion in cells
necrosis in AP ( 157to causenecrosis in AP ( 157
to cell deathleadsto cell death
to cell deathleadsto cell death
to the formation of reactive astrocytes 11contributedto the formation of reactive astrocytes 11
to a cascade of beneficial effects ... increasing cellular function and healthleadsto a cascade of beneficial effects ... increasing cellular function and health
to the cascade of beneficial effects ... increasing cellular function and healthleadsto the cascade of beneficial effects ... increasing cellular function and health
to a cascade of beneficial effectsleadsto a cascade of beneficial effects
to normalization of cell function , pain relief and healingleadsto normalization of cell function , pain relief and healing
a constellation of cellular alterations culminating in proximal tubule cell injury , cell death , and organ dysfunctioncausesa constellation of cellular alterations culminating in proximal tubule cell injury , cell death , and organ dysfunction
to normalization of cell function and the halting ( or reversal ) of the miniature process of hair due to DHTleadsto normalization of cell function and the halting ( or reversal ) of the miniature process of hair due to DHT
to the actual inhibitory consequencesmay ... contributeto the actual inhibitory consequences
an increase in luminescence over timecausingan increase in luminescence over time
to autoimmune and inflammatory diseasescan contributeto autoimmune and inflammatory diseases
to a healthy production of mucuscontributesto a healthy production of mucus
to cell deathleadingto cell death
an increase in cell metabolismcausesan increase in cell metabolism
in the increase in ADP / ATP ratiowill resultin the increase in ADP / ATP ratio
in failure of the Ca2can resultin failure of the Ca2
to improved healing and repair of the skinleadingto improved healing and repair of the skin
to lysis and cell deathleadsto lysis and cell death
in cell death and ultimately death of the organismresultsin cell death and ultimately death of the organism
in tumor cell deathresultingin tumor cell death
from LPresultingfrom LP
cell death in mouse hepatocytes and in a hepatoma cell line ( HepG2can ... causecell death in mouse hepatocytes and in a hepatoma cell line ( HepG2
to oxidative stress and chronic inflammationleadsto oxidative stress and chronic inflammation
to increased glucose uptakeleadsto increased glucose uptake
to the activation of AMPKleadsto the activation of AMPK
to energy collapse and cell deathleadingto energy collapse and cell death
inhibition of the primary pumpcausinginhibition of the primary pump
to reversible dysfunction of the ATP dependent sodium potassium ATPaseleadsto reversible dysfunction of the ATP dependent sodium potassium ATPase
on the other handleadson the other hand
thusmay ... leadthus
to increased cytoplasmic Ca from Sarcolesmaleadsto increased cytoplasmic Ca from Sarcolesma
widespread musculus fibre hypoxia cell deceasecauseswidespread musculus fibre hypoxia cell decease
to AMPK activation , mTORC1 inhibition , and autophagy induction as the last protective resort143leadsto AMPK activation , mTORC1 inhibition , and autophagy induction as the last protective resort143
to cell membranes that are more prone to oxidative damageleadingto cell membranes that are more prone to oxidative damage
T helper behaviorwould ... influenceT helper behavior
Glucosamine - induced insulin resistance in 3T3-L1 adipocytes(passive) is caused byGlucosamine - induced insulin resistance in 3T3-L1 adipocytes
to impairment of ionic pumps , cell swelling , clearing of the cytosolleadingto impairment of ionic pumps , cell swelling , clearing of the cytosol
in the inhibition of Ca(2resultedin the inhibition of Ca(2