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cells against stressescauseATP depletion
to reverse mitochondrial dysfunctioncausingmarked ATP depletion
of which mitochondrial dysfunction ( KE1leadingto ATP depletion
leads to uncoupling of oxidative phosphorylationresultingin ATP depletion
the Ca2 + -induced mitochondrial dysfunction(passive) caused bythe ATP depletion
the kinase cascade ... cellular stressescauseATP depletion
Leukemia ] Abstract Targeting JMJD1C - mediated metabolism via pharmacologic inhibition of glycolysis and oxidative phosphorylationledto ATP depletion
ATPby oxidative phosphorylationcausingATP depletion
Ischemia , mitochondrial dysfunction , and/or excessive ATP consumptioncausecellular ATP depletion
impaired glucose breakdowncausingATP depletion
Sufficient hypoxiawill causecellular ATP depletion
hypoxia ( 4(passive) caused byATP depletion
hypoxia in cardiomyocytes(passive) caused bythe intracellular ATP depletion
AMPK ... particularly stressescausedepletion of ATP
The observation ... inhibition of Pink1leadsto ATP depletion
the pharmacological inhibition of glycolysis(passive) caused byATP depletion
a highly conserved sensor of increased levels of AMP and ADPoriginatingfrom ATP depletion [ 185–187
The major molecular sensor for AMP level in cells ... cellular stressesresultin ATP depletion
die only after their glucose supply is exhaustedresultingin ATP depletion
the inhibition of mitochondrial respiratory enzymes and glucose uptake(passive) caused byATP depletion
The Dog1p may create a futile cycle of phosphorylation and dephosphorylation of glucoseresultingin ATP depletion
mitochondrial inhibition(passive) caused byATP depletion
metabolic inhibition(passive) caused byATP depletion
inhibition of mitochondriacausesATP depletion
the mitochondrial complex(passive) caused byATP depletion
metabolic switching to utilize glucose ... sufficientto preventATP depletion
347.33 Upregulates CDKN1Acausesdepletion of ATP
In vitro , glycolysis will lower the glucose concentration in erythrocytescausingdepletion of ATP
extramitochondrial production of ATPleadto ATP depletion
Production of ROS and RNSalso causesATP depletion
However , SO2 can also act both in glycolysis and in respiratory chain phosphorylation in yeastcausingATP depletion
hypoxia , which is contrary to what we observe when the cardiomyocytes are subjected to metabolic stress(passive) caused bythe intracellular ATP depletion
the ability of these stimulito eventually causeATP depletion
the conditions that initiated it ( ie , an increase in free Ca2 + and a decrease in mitochondrial membrane potential ,leadsto ATP depletion
On the other hand , TG02 also caused disruption of the mitochondrial respiration complexes and inhibition of the glycolysisresultingin ATP depletion
glucose in a glucosephosphate isomerase - deficient strain ofSaccharomyces cerevisiae(passive) caused byATP depletion
glucose in a glucosephosphate isomerase - deficient strain of Saccharomyces cerevisiae(passive) caused byATP depletion
the mitochondrion and functional changesresultingin ATP depletion
inhibition of glyceraldeyde-3-phosphate dehydrogenase and alcohol dehydrogenases [ 4(passive) caused bydepletion of ATP
Dong et al22 used 1 mg / mL glucoseto preventATP depletion
to rapid cell deathleadsto rapid cell death
necrosis or apoptosis of cultured mouse proximal tubular cells | Renal Physiology Skipcan causenecrosis or apoptosis of cultured mouse proximal tubular cells | Renal Physiology Skip
to cell death by a necrosis - like mechanismleadingto cell death by a necrosis - like mechanism
necrosis or apoptosis of cultured mouse proximal tubular cells ... ” American Journal of Physiology , vol .can causenecrosis or apoptosis of cultured mouse proximal tubular cells ... ” American Journal of Physiology , vol .
apoptosis initiationis preventingapoptosis initiation
to necrotic cell death ( 19leadingto necrotic cell death ( 19
necrosis or apoptosis of cultured mouse proximal tubular cells ... The American Journal of Physiology — Renal Physiology ... vol .can causenecrosis or apoptosis of cultured mouse proximal tubular cells ... The American Journal of Physiology — Renal Physiology ... vol .
to mitochondrial dysfunction culminating in cell death [ 9then leadsto mitochondrial dysfunction culminating in cell death [ 9
to cell necrosisleadingto cell necrosis
widespread muscle fiber hypoxia ( cell death , rhabdomyolysis ) , which manifests clinically as hyperkalemia and myoglobinuria and an increase in creatine kinasecauseswidespread muscle fiber hypoxia ( cell death , rhabdomyolysis ) , which manifests clinically as hyperkalemia and myoglobinuria and an increase in creatine kinase
to necrotic and/or apoptotic cell death [ 2leadingto necrotic and/or apoptotic cell death [ 2
to initiation of cell death [ 6,24may contributeto initiation of cell death [ 6,24
Autophagic and necrotic cell death pathways(passive) are triggeredAutophagic and necrotic cell death pathways
to a switch from apoptotic to necrotic cell deathcontributedto a switch from apoptotic to necrotic cell death
in a necrotic cell death called parthanatosresultsin a necrotic cell death called parthanatos
either necrosis or apoptosis in MPT cellscan causeeither necrosis or apoptosis in MPT cells
in both apoptosis and necrosis , a reduction of ATP levels by 80 % or morecan resultin both apoptosis and necrosis , a reduction of ATP levels by 80 % or more
in necrosis under conditions that would otherwise result in apoptosis ( 25can resultin necrosis under conditions that would otherwise result in apoptosis ( 25
cell membrane pump failure and cell deathcausescell membrane pump failure and cell death
to the generation of the waste product uric acidleadsto the generation of the waste product uric acid
necrosis or apoptosis of cultured mouse proximal tubular cells | Renal Physiology Wilfred Lieberthal , Sarah A. Menza , Jerrold S. Levine American Journal of Physiology - Renal Physiologycan causenecrosis or apoptosis of cultured mouse proximal tubular cells | Renal Physiology Wilfred Lieberthal , Sarah A. Menza , Jerrold S. Levine American Journal of Physiology - Renal Physiology
to necrosis.17 , 18leadsto necrosis.17 , 18
downstream caspase activation and apoptosispreventsdownstream caspase activation and apoptosis
necrotic cell death due to energy depletion [ 189causingnecrotic cell death due to energy depletion [ 189
to µ-calpain - mediated cell death ( NAD+-Keresisleadingto µ-calpain - mediated cell death ( NAD+-Keresis
necrosis or apoptosis of cultured mouse proximal tubular cellsAbstractFulltextPDFLysophosphatidiccan causenecrosis or apoptosis of cultured mouse proximal tubular cellsAbstractFulltextPDFLysophosphatidic
necrosis or apoptosis of cultered mouse proximal tubular cellscan causenecrosis or apoptosis of cultered mouse proximal tubular cells
necrosis or apoptosis of cultured mouse proximal tubular cellcan causenecrosis or apoptosis of cultured mouse proximal tubular cell
apoptosis in tubular epithelial cellscausesapoptosis in tubular epithelial cells
alsomight ... causealso
release of calcium from the sarcoplasmic reticulum in musclescausesrelease of calcium from the sarcoplasmic reticulum in muscles
to necrotic deathleadsto necrotic death
rapid metabolic collapse and necrosiscausesrapid metabolic collapse and necrosis
to cell membranes that are more prone to oxidative damage [ 5leadingto cell membranes that are more prone to oxidative damage [ 5
to end organ failure in shockleadingto end organ failure in shock
the metabolic stress(passive) caused bythe metabolic stress
in a marked decrease of the G - actin fraction of EYFP - actin ( Fig . 6 Aresultedin a marked decrease of the G - actin fraction of EYFP - actin ( Fig . 6 A
AM - induced cytotoxicity and ultimately pulmonary fibrosismight ... preventAM - induced cytotoxicity and ultimately pulmonary fibrosis
Rho inactivation during ischemia and that recovery of normal cellular architecture and function requires RhocausesRho inactivation during ischemia and that recovery of normal cellular architecture and function requires Rho
to mTOR inhibition via activation of AMPKleadingto mTOR inhibition via activation of AMPK