which releases energy used in a culminating oxidative - phosphorylation reactionto createATP
that animal cells useto createATP
Releasing energy along the waySome of that energy is usedto createATP
Mitochondria ... cellscreateATP
a proton gradient across the cellular membrane , the energy from which is usedto createATP
the process ... usedto createATP
Although NAD+ is used by the bodyto createATP
To get at this energy , cells use the process of cellular respirationto createATP
CoQ10 ... usedto createATP
Your mitochondria use oxygento createATP
of ribose ( a sugar ) , adenine ( a nitrogenous base ) , and three phosphate groups 3(passive) is composedATP
how your cells use glucose and oxygento createATP
glucose then glucose is usedto createATP
Your body ’s abilityto createATP
your cells and mitochondriacreateATP
adding the phosphate to ADPto createATP
the carbohydrates ... then used for cellular respirationto createATP
PMF is then used by ATP synthaseto createATP
oxidative phosphorylation by ATP synthaseto createATP
mitochondria not only use the stored energyto createATP
your cells ... using itto createATP
the mitochondria to use more energyto createATP
enters cells ... is usedto createATP
the ATP synthase ... in turnwill createATP
oxidative phosphorylation ... the processcreatesATP
to be added to ADPcreatingATP
work ... is usedto createATP
The cells can use either glucose or keto acidsto createATP
coenzymes , which are then used in oxidative phosphorylationto createATP
An enzyme called ATP synthase is usedto createATP
that transported protons are used by ATP synthaseto createATP
FADH 2 to create a proton gradient across the mitochondrial membrane and that this energy was used by a reversible proton pump , the ATP synthaseto createATP
The use of ATP synthase isto createATP
The role of ATP synthase isto createATP
which is employed by the ATP synthaseto createATP
to the release of adenosineleadingto the release of adenosine
release of intracellular Ca2causesrelease of intracellular Ca2
release of intracellular ca+causesrelease of intracellular ca+
release of interleukincausedrelease of interleukin
a transient increase in the intracellular Ca2causeda transient increase in the intracellular Ca2
The significance level of this study(passive) was setThe significance level of this study
to rapid energy release and muscle contractioncontributesto rapid energy release and muscle contraction
Ca2 + release from intracellular storestriggeredCa2 + release from intracellular stores
a conformational change in the proteincausesa conformational change in the protein
ER Ca2 + release(passive) provoked byER Ca2 + release
Statistical significance for the differences(passive) was setStatistical significance for the differences
Statistical significance levels(passive) were setStatistical significance levels
the vessel walls to relax and dilate so as to promote normal blood flow.[32causesthe vessel walls to relax and dilate so as to promote normal blood flow.[32
the vessel walls to relax and dilate so as to promote normal blood flow.[34causesthe vessel walls to relax and dilate so as to promote normal blood flow.[34
the vessel walls to relax and dilate so as to promote normal blood flow.[13causesthe vessel walls to relax and dilate so as to promote normal blood flow.[13
the vessel walls to relax and dilate so as to promote normal blood flow.[17causesthe vessel walls to relax and dilate so as to promote normal blood flow.[17
the vessel walls to relax and dilate so as to promote normal blood flow.[16causesthe vessel walls to relax and dilate so as to promote normal blood flow.[16
to NO release from endothelial cellscould leadto NO release from endothelial cells
the release and accumulation of BDNF(passive) caused bythe release and accumulation of BDNF
in the first conformational change in IVAresultsin the first conformational change in IVA
The threshold for statistical significance(passive) was setThe threshold for statistical significance
activation of PLA2 and kallikrein releasecausesactivation of PLA2 and kallikrein release
energy and can be used for muscle contractions and thus cyclingcreatesenergy and can be used for muscle contractions and thus cycling
energy for the cell to functioncreatesenergy for the cell to function
a living memorial to General Dro and his victory in 1918 near the monument and final resting place of the national herois creatinga living memorial to General Dro and his victory in 1918 near the monument and final resting place of the national hero
the maximum contraction(passive) caused bythe maximum contraction
the efflux of K+ from the cell , followed by Ca2 + influx and activation of three phospholipases : phosphatidylcholine - specific phospholipase C and calcium - independent and -dependent phospholipase A2triggersthe efflux of K+ from the cell , followed by Ca2 + influx and activation of three phospholipases : phosphatidylcholine - specific phospholipase C and calcium - independent and -dependent phospholipase A2
to overproduction of reactive oxygen species ( ROSledto overproduction of reactive oxygen species ( ROS
the release of BDNF evoked by ATPpreventedthe release of BDNF evoked by ATP
the release of an unfolded polypeptide from Ssa1p but not fromtriggerthe release of an unfolded polypeptide from Ssa1p but not from
to release of ions of Ca 2leadsto release of ions of Ca 2
the release of leukemia inhibitory factor ( LIF ) , which then stimulates myelination by oligodendrocytes ( Ishibashi et al . , 2006causesthe release of leukemia inhibitory factor ( LIF ) , which then stimulates myelination by oligodendrocytes ( Ishibashi et al . , 2006
exocytosis of these organelles with secretion of IL-1β and caspase-1 . Chemicalstriggersexocytosis of these organelles with secretion of IL-1β and caspase-1 . Chemicals
both the release of Ca2 + from the intracellular pool Fig .triggeredboth the release of Ca2 + from the intracellular pool Fig .
Threshold for significance(passive) was setThreshold for significance
heterogeneity within and distinction from endoplasmic reticulum Inhibition of inositol trisphosphate - induced calcium release by caffeine(passive) is prevented byheterogeneity within and distinction from endoplasmic reticulum Inhibition of inositol trisphosphate - induced calcium release by caffeine
to activation of purinergic receptorsleadingto activation of purinergic receptors
The level of significance for ANOVA(passive) was setThe level of significance for ANOVA