by the activation of C1q by IgG and IgM(passive) is causedC1q glomerular deposition
by a vascular disorder(passive) may be causedamyloid deposition
The mechanisms through whichmay influenceamyloid deposition
More than 30 proteinscan causeamyloid deposition
the neurodegeneration cascadecausingamyloid deposition
none of the neurotrophic factor alterationsinfluencedamyloid deposition
by gout(passive) caused byurate deposition
factorsinfluencingurate deposition
by the activation of C1q by IgG and IgM(passive) is caused byglomerular deposition
amyloid , hyperuricemia , or local malignancy infiltrate(passive) caused byglomerular deposition
by the activation of thrombin(passive) is ... causedfibrinogen deposition
Rapid rates of sediment inputcauseddeposition of the shales
by atherosclerosis(passive) caused byendothelial deposition
PHB(passive) caused byH3.3 deposition
such things as flood events or volcanic ash deposition(passive) caused byvarve deposition
generalized myxedemacausesdeposition of mucopolysaccharides
such things as flood events , bathymetric changes associated with lake level change , or sudden changes in sediment source(passive) caused byvarve deposition
the extracellular space(passive) caused bythe extracellular space
a soft tissue ( or bony ) masscan rarely causea soft tissue ( or bony ) mass
the adverse global pathological burden(passive) caused bythe adverse global pathological burden
enlargement of the ocular muscules(passive) is caused byenlargement of the ocular muscules
to clot formation and subsequent thrombosisleadingto clot formation and subsequent thrombosis
local inflammatory and immunologic alterations for a direct neurotoxicity with microglial recruitment and astrocyte activationcauseslocal inflammatory and immunologic alterations for a direct neurotoxicity with microglial recruitment and astrocyte activation
to microglial activation ( Meyer - Luehmann et al . , 2008 ) and the production of proinflammatory mediators that contribute to disease pathogenesisleadsto microglial activation ( Meyer - Luehmann et al . , 2008 ) and the production of proinflammatory mediators that contribute to disease pathogenesis
mechanical damage to RBCscan causemechanical damage to RBCs
the Prevention and Treatment of Formation Damage(passive) Caused bythe Prevention and Treatment of Formation Damage
to productionleadsto production
problems in wettability alterationcausesproblems in wettability alteration
to recruitment of NXF1leadsto recruitment of NXF1
to device failure or clinical complicationscan leadto device failure or clinical complications
to targeted RNA instability in ALS and FTD ... and may ultimately cause cell death by disrupting energy production and protein synthesis pathwaysleadsto targeted RNA instability in ALS and FTD ... and may ultimately cause cell death by disrupting energy production and protein synthesis pathways
the wettability reversalwill causethe wettability reversal
to mesotheliomas , which typically occurs as a group of conditions ( other than liver , spleen , or nodesmay leadto mesotheliomas , which typically occurs as a group of conditions ( other than liver , spleen , or nodes
viral pathway fate by effects on other viral gene products feeding back to the level of K - Rtamay influenceviral pathway fate by effects on other viral gene products feeding back to the level of K - Rta
in the deposition of a metal and an anodic productresultsin the deposition of a metal and an anodic product
in the deposition of a metal and an anodic product ... e.g. , nickel and nickel - phosphorusresultsin the deposition of a metal and an anodic product ... e.g. , nickel and nickel - phosphorus
in the removal of interstitial materials and the formation of reactive surface properties on , for example , compound target surfacesmay resultin the removal of interstitial materials and the formation of reactive surface properties on , for example , compound target surfaces
in the removal of interstitial materials and the formation of reactive surface properties onmay resultin the removal of interstitial materials and the formation of reactive surface properties on
in a strongly staining , continuous distribution along the epithelial - stromal junctionresultingin a strongly staining , continuous distribution along the epithelial - stromal junction
to corresponding decreases in the deposition rate and film thicknessledto corresponding decreases in the deposition rate and film thickness
damage to temperature - sensitive materials and semiconductor surfacemay causedamage to temperature - sensitive materials and semiconductor surface
to the formation of a range of landforms in the lower course of the riverleadsto the formation of a range of landforms in the lower course of the river
to particle formation or increased diffusion time for larger clusterscan leadto particle formation or increased diffusion time for larger clusters
in films with lower resistanceresultedin films with lower resistance
to changes in surface albedocan leadto changes in surface albedo
to the formation of amorphous carbonleadsto the formation of amorphous carbon
to [ [ inflammation ] ] and [ [ fibrosisleadingto [ [ inflammation ] ] and [ [ fibrosis
the formation of iron silicide layer at the interfacecausingthe formation of iron silicide layer at the interface
in soil acidificationcan resultin soil acidification