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Motor effectscan resultfrom stimulation of sensory cortex
Dr. Halassa and colleagues ... by whichcan causeattentional modulation of sensory cortex depends on the thalamus
John Carlson ’s labdiscoveredhow specific leg sensory neurons arouse
Disruption of NMDA receptor functionpreventsnormal experience - dependent homeostatic synaptic plasticity in mouse primary visual cortex
most of Capsaicin biological effectsresultfrom initial arousal of certain sensory neurons
In particular , toxins from marine animals have a preference for blocking voltage - gated sodium channels ,resultingin sensory abnormalities ( because of dysfunction of sensory neurons
how to translate those signals from the artificial skinto createthe appropriate sensations in the sensory cortex of the brain
The reticular formation also filters incoming sensory impulsesto preventthe cortex from being constantly bombarded by sensory stimulation
two of the ASIC family memberscontributeto mechanotransduction in mouse sensory neurons
I MHC genescontributesto ultrasensitive chemodetection by mouse vomeronasal sensory neurons
to the manumit of transmitters ( for standard , sub-leadsto the manumit of transmitters ( for standard , sub-
to neuronal hyperexcitabilitycontributesto neuronal hyperexcitability
to the unshackle of transmitters ( in the direction of norm , sub- standpoint P and excitatory amino acids ) that obligate to and switch pain - projection neurons in the spinal cordleadsto the unshackle of transmitters ( in the direction of norm , sub- standpoint P and excitatory amino acids ) that obligate to and switch pain - projection neurons in the spinal cord
that ocular dominance was largely preserved in the mutant , with similar proportion of cells responding to contra- or ipsilateral stimulationdiscoveredthat ocular dominance was largely preserved in the mutant , with similar proportion of cells responding to contra- or ipsilateral stimulation
to the release of transmitters ( for the sake of example , sub- posture P and excitatory amino acids ) that bind to and turn on pain - projection neurons in the spinal lineleadsto the release of transmitters ( for the sake of example , sub- posture P and excitatory amino acids ) that bind to and turn on pain - projection neurons in the spinal line
to an attenuation of the painful symptoms of hypersensitivity via the inhibition of Nav1.8 channel activitymay leadto an attenuation of the painful symptoms of hypersensitivity via the inhibition of Nav1.8 channel activity
global lowered pain threshold(passive) caused byglobal lowered pain threshold
to markedly impaired axon regenerationledto markedly impaired axon regeneration
in behavioral studiescreatedin behavioral studies
a significant increase in mitochondrial content , quantified as a higher density of stationary mitochondriacauseda significant increase in mitochondrial content , quantified as a higher density of stationary mitochondria