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Qaagi - Book of Why

Causes

Effects

levels of PAR in models of PD ... in turnresultedin less alpha synuclein aggregation

BergerdiscoveredAlpha waves[75

factorsinfluencealpha - synuclein function

of glucose and hesperidin(passive) is composedAlpha Glucosyl Hesperidin

For one , dopaminergic neurons show abnormalities in mitochondrial complex 1causingaggregation of alpha - synuclein

defects in the gene for the alpha chain of haemoglobin , which is located on chromosome 16(passive) is caused byAlpha thalassaemia

other factorscontributeto alpha - synuclein aggregation

by gene deletions(passive) Caused byAlpha Thalassaemia

by a poly(A ) site mutation(passive) caused byAlpha - thalassaemia

a poly(A ) site mutation(passive) caused byAlpha - thalassaemia

a polyadenylation signal mutation(passive) caused byAlpha - thalassaemia

by a polyadenylation signal mutation(passive) caused byAlpha - thalassaemia

that IDI1 and IDI2 may be associated with the production of cholesterol metabolites in neuronsleadingto alpha - synuclein aggregation

Lewy bodiescomposedof aggregates of the alpha - synuclein

the type - checkerwill setalpha to Unlifted

this compoundwould ... influencealpha synuclein aggregates

The teamdiscoveredalpha - synuclein clumps

SNCA gene triplication impairs neuronal differentiation and maturation in Parkinson 's patient - derived induced pluripotent stem cells(passive) caused byElevated alpha - synuclein

by SNCA gene triplication impairs neuronal differentiation and maturation in Parkinson 's patient - derived induced pluripotent stem cells(passive) caused byElevated alpha - synuclein

protein clumpscomposedof aggregated alpha - synuclein

protein clumpsmainly composedof aggregated alpha - synuclein

for the US Special Forces(passive) was inventedPolartec ALPHA

Mutation of the SNCA genecausesalpha - synuclein

cytoplasmic inclusionscomposedof aggregated alpha - synuclein

bodies insoluble cytoplasmic inclusionscomposedof aggregated alpha - synuclein

Mutations incausealpha - synuclein

insoluble cytoplasmic inclusionscomposedof aggregated alpha - synuclein

of sugars(passive) are composedAlpha glucans

Neuronal inclusions ofcontributealpha - synuclein

Tone thereinfluencedalpha - adrenergic

to provide advanced investment strategies in an ETF and represent ProShares ' further expansion within the alternative ETF space(passive) are designedAlpha ProShares

of characteristic regions that include the N - terminal head domain , the flexible hinge domain , and the ear domain(passive) are composedalpha - adaptins

using the Python programming language as an accessible and handy tool for rolling dice on IRC(passive) was specially designedAlpha - PyRC Diceroller

to cell deathcontributesto cell death

of the other form of the proteincomposedof the other form of the protein

in the immediate aftermathsetin the immediate aftermath

to the development of PDcontributesto the development of PD

Parkinson 's DiseasecausesParkinson 's Disease

the loss of motor functions in PD patientscausesthe loss of motor functions in PD patients

to neurodegeneration ofleadingto neurodegeneration of

neuronal deathcauseneuronal death

to distinct synucleinopathiesmay leadto distinct synucleinopathies

to neurotoxicityleadingto neurotoxicity

paralysis(passive) caused byparalysis

degenerationcausedegeneration

PD , in principle we can use a similar approach to knock down synuclein expressioncausesPD , in principle we can use a similar approach to knock down synuclein expression

to strange faults within the hippocampal neurons ' " communication mechanismsledto strange faults within the hippocampal neurons ' " communication mechanisms

to various cellular disorders including microtubule impairment , synaptic and mitochondrial dysfunctions , oxidative stress as well as dysregulation of Calciummay leadto various cellular disorders including microtubule impairment , synaptic and mitochondrial dysfunctions , oxidative stress as well as dysregulation of Calcium

to decreased A1AT activity inleadingto decreased A1AT activity in

mitochondrial trafficking deficiency and respiration dysfunctioncan causemitochondrial trafficking deficiency and respiration dysfunction

to the formation of harmful structures in human membranes found in Parkinson 's and Alzheimer 's patientscan leadto the formation of harmful structures in human membranes found in Parkinson 's and Alzheimer 's patients

within Lewy bodiesdiscoveredwithin Lewy bodies

to Parkinson?s diseasemight leadto Parkinson?s disease

problemscauseproblems

familial Parkinson 's diseasecausefamilial Parkinson 's disease

Parkinson 's DiseasecausesParkinson 's Disease

to substantia nigra dysfunctioncontributesto substantia nigra dysfunction

to cell deathleadsto cell death

all the problemsis causingall the problems

in cellular stresswould ... resultin cellular stress

to the pathogenesis of Parkinson 's diseasecontributesto the pathogenesis of Parkinson 's disease

mitochondrial impairment(passive) caused bymitochondrial impairment

to toxicityleadsto toxicity

the toxicity(passive) caused bythe toxicity

significant damage to neuronscausessignificant damage to neurons

diseases(passive) caused bydiseases

less transmission and damage(passive) caused byless transmission and damage

pathology in these diseasescausespathology in these diseases

familial early onsetcausefamilial early onset

to the formation of pore - like structures on membranescan leadto the formation of pore - like structures on membranes

local tissue damagecauselocal tissue damage

to dangerous structures in membranesledto dangerous structures in membranes

the immune response(passive) provoked bythe immune response

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