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the factorsinfluenceperitraumatic dissociation
a direct conformational change to APLIP1 ( JIP1causesKlc dissociation
Cultures were grown at 37 ° C ; before collecting the cells , the cultures were incubated 5 min at 37 ° C with chloramphenicol ( final concentration , 0.1 mg / mlto preventpolysome dissociation
gouty arthritis of the wrist - Case report(passive) caused byScapholunate dissociation
Blunt Trauma Damschen , Donald D.(passive) Caused byScapulothoracic Dissociation
during Forex Brokers Paignton , chronic RyR hyperphosphorylation by PKAcausesdissociation of FKBP12
legal , chronic RyR hyperphosphorylation by PKA ( Ser-2808causesdissociation of FKBP12
Select item 1915319420.Disruption of Nficcausesdissociation of odontoblasts
Electron impactalso causesFomblin - Zdol dissociation
4.77 Impact Factor Disruption of Nficcausesdissociation of odontoblasts
the possibility that activated Hep induces in APLIP1 ( JIP1 ) a direct conformational changecausesKlc dissociation
phosphorylation of the five central residuescausesPlk4 dissociation
scapholunate ligament tear which creates widening of scapholunate space(passive) is caused byScapholunate dissociation
Acta Chir De Smet L , Degreef I. Bilateral osteochondroma of the scaphoidcausingscapholunate dissociation
may be due to the lack of S408 phosphorylationpreventingp56lck dissociation
SG assembly ( 41 ) , and cycloheximidepreventspolysome dissociation
urea 1 M. Direction of the TLG development indicated by(passive) provoked byHcy dissociation
Addition of 0.15 µM FKBP12 to SR vesiclespreventedFKBP12 dissociation
the breakup of water molecules by the sun ’s ultraviolet light , and photosynthesis , the process that uses carbon dioxide and water ( H2O ) to form organic compounds while also releasing oxygen ( O2(passive) caused byphotochemical dissociation
titrating in unlabeled NCPsto preventnucleosome dissociation
The DNA ... the observation channel , which lacks free YoYo1 and contains Mg(OAccausesYoYo1 dissociation
groups on the iminesto preventimine dissociation
further unfolding of α - catenin(passive) caused byvinculin dissociation
injected ATPto triggerMDA5hSNAP dissociation
D , Exposure of endothelial cells to a rapid NO donorcausesHSF1–HSP90 dissociation
C , Exposure of endothelial cells to a slow NO donorcausesHSF1-HSP90 dissociation
elevation of cytosolic cAMP concentrations(passive) caused byHoloenzyme dissociation
Figure 5.RNAi - mediated depletion of Rab7causesVps26 dissociation
radical crosslinking of P - triblock micelle coreswill preventmicelle dissociation
DoxorubicintriggeredLIN9 dissociation
Microinjection of anti - GBF1 antibodiescauseddissociation of COPI
Neddylation of CUL1 or the presence of SKP1 and ATPcausesCAND1 dissociation
treatment of cells with PS - PKCζ in combination with TSApreventedp107 dissociation
block / acceleration dissociation and third degree atrioventricular block allmay createatrioventricular dissociation
the activation of an associated receptor(passive) is triggered byNivestopedia dissociation
1;19(9):1022 - 1027.FigureMay ... causes4.Cell dissociation
low pH. YscUC(passive) was prevented byYscUC dissociation
Only two out of four concentrated antibody preparations , 9D2 and 9D7causeddissociation of COPI
higher phosphate buffer concentrationspreventedcofactor dissociation
cleavage of the extracellular segment of Dsg1 by S. aureus ETs ... sufficientto causekeratinocyte dissociation
space(passive) created byspace
to formation of large intercellular gaps ( arrows ) and loss of Dsg 3-containing desmosomesleadingto formation of large intercellular gaps ( arrows ) and loss of Dsg 3-containing desmosomes
from severe traumaresultsfrom severe trauma
to unopposed volar flexion of the scaphoidleadsto unopposed volar flexion of the scaphoid
to Al12XH and H atom proceeds without a barrierleadingto Al12XH and H atom proceeds without a barrier
to a failure to integrate the trauma related information and prevents the realization that the trauma belongs to the past [ 13leadsto a failure to integrate the trauma related information and prevents the realization that the trauma belongs to the past [ 13
in flexion of the scaphoid and a ring sign often also seen with progressive perilunate instability , which is discussed further belowresultsin flexion of the scaphoid and a ring sign often also seen with progressive perilunate instability , which is discussed further below
only after the trauma - exposedindividual has experienced a high level of distress , and that its function isnot to protect the individual from experiencing any level of distress but toprotect them from experiencing additional ( and perhaps unbearable or overwhelming)distressis triggeredonly after the trauma - exposedindividual has experienced a high level of distress , and that its function isnot to protect the individual from experiencing any level of distress but toprotect them from experiencing additional ( and perhaps unbearable or overwhelming)distress
a significant number of oxygen atoms as well ... if UV photodissociation serves as a basis for H2O formationmust contributea significant number of oxygen atoms as well ... if UV photodissociation serves as a basis for H2O formation
to desmolysis / acantholysisleadingto desmolysis / acantholysis
in altered cristae morphology and release of Opa1 and cyt c into the cytosol of CA1 neuronsresultingin altered cristae morphology and release of Opa1 and cyt c into the cytosol of CA1 neurons
to gel structure destabilization associated with vitreous liquefaction and posterior vitreous detachment in diabetesleadingto gel structure destabilization associated with vitreous liquefaction and posterior vitreous detachment in diabetes
to the exposure of an additional lysine - binding sitemay leadto the exposure of an additional lysine - binding site
in the absence of SK2 channels and the emergence of non - inactivating BK channels [ 22,23resultingin the absence of SK2 channels and the emergence of non - inactivating BK channels [ 22,23
2017 20 Cyclic Nonrespiratory Pulse Pressure Oscillations(passive) Caused by2017 20 Cyclic Nonrespiratory Pulse Pressure Oscillations
to the Gibbs free energy change for DCU ( DCU0 ) of 128leadingto the Gibbs free energy change for DCU ( DCU0 ) of 128
from combined thoracic epidural and general anesthesiaresultingfrom combined thoracic epidural and general anesthesia
Cappato R 28008694 2017 Cyclic Nonrespiratory Pulse Pressure Oscillations(passive) Caused byCappato R 28008694 2017 Cyclic Nonrespiratory Pulse Pressure Oscillations
poor tissue oxygenation PaO2causingpoor tissue oxygenation PaO2
to a significantly higher gain of cytokeratin - positive cells compared to percentage of cytokeratin - positive cells after manual tissue disaggregationledto a significantly higher gain of cytokeratin - positive cells compared to percentage of cytokeratin - positive cells after manual tissue disaggregation
conditioningcreatesconditioning
favorable conditions for the separation of copper and leadto createfavorable conditions for the separation of copper and lead
2725 Cyclic Nonrespiratory Pulse Pressure Oscillations(passive) Caused by2725 Cyclic Nonrespiratory Pulse Pressure Oscillations
in the increasing intensity of the mass peaks corresponding to the ionized reaction products with temperatureresultedin the increasing intensity of the mass peaks corresponding to the ionized reaction products with temperature
as much as 2 % of the oxygen in our current atmospheremay have contributedas much as 2 % of the oxygen in our current atmosphere
ROCK - dependent anoikiscausesROCK - dependent anoikis
An abnormal P - R interval(passive) is caused byAn abnormal P - R interval
to downregulation of TFleadsto downregulation of TF
to the formation of intrusive memoriesleadsto the formation of intrusive memories
linker - specific phosphorylation of Smad1 in Xenopus ectodermcauseslinker - specific phosphorylation of Smad1 in Xenopus ectoderm
a 10-fold increase in the duration of ryanodine receptor channel opening in lipid bilayerscauseda 10-fold increase in the duration of ryanodine receptor channel opening in lipid bilayers
to an 18 ° head rotation ( Strunk et al . , 2011leadsto an 18 ° head rotation ( Strunk et al . , 2011
in mobile polaronsresultingin mobile polarons
to VISI deformityleadsto VISI deformity
blood accumulation within the liver and death by hypovolumic shockcausingblood accumulation within the liver and death by hypovolumic shock
the loss of the Sin3 complex from the p21 promoter a , b and c. ChIP was performed in 293 T cells treated for 10 hours with DMSO ( D ) or SAHA ( S ) d , e and f. ChIPcausesthe loss of the Sin3 complex from the p21 promoter a , b and c. ChIP was performed in 293 T cells treated for 10 hours with DMSO ( D ) or SAHA ( S ) d , e and f. ChIP
proliferation and epithelial morphogenesis ( 4 , 129influencesproliferation and epithelial morphogenesis ( 4 , 129
from rupture of the scapholunate ligamentresultingfrom rupture of the scapholunate ligament
to Dorsal or VolarIntercarlate aka Intercarpal Segmental Instability ( DISIor VISIwill leadto Dorsal or VolarIntercarlate aka Intercarpal Segmental Instability ( DISIor VISI
to reactivation of EMT gene expression ( SI Appendix , Figleadsto reactivation of EMT gene expression ( SI Appendix , Fig