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cystitis and kidney stones(passive) caused byurinary secretion
any drugsinfluencepituitary secretion
Patient ... any medicationsmay influencepituitary secretion
any medicationsmay influencepituitary secretion
any medications ... , if possibleinfluencepituitary secretion
studies on the factorsinfluencingcytokines secretion
factorsinfluencingcytokines secretion
persists in human macrophagesinfluencingcytokine secretion
The upregulation of TGM2 in the choroid plexus ( CPcould influenceCSF secretion
by caspase-1(passive) caused bycytokine secretion
Increased adrenocorticotropic hormonecould ... leadACTH ) secretion
ACTHonly minimally influencesaldosterone secretion
ACTHcan influencealdosterone secretion
1,25(oh)2 d3 impairsintestinal ca engrossment and modifys blood serum calciumcausespth secretion
a defect in the parathyroid cells that makes them resistant or hyposensitive to the suppressive effects of hypercalcemia(passive) caused byPTH ) secretion
sympathetic nerve activationresultingrenin secretion
the factorsmay influencerenin secretion
to studymay influencerenin secretion
not a stimuluscausesrenin secretion
by hypertension(passive) caused byrenin secretion
the ability of calciumto causenorepinephrine secretion
the antidiuretic hormone ... just dehydrationcausesADH secretion
by alcohol(passive) caused byADH secretion
by furosemide in rats ( 1(passive) caused byrenin secretion
by furosemide(passive) caused byrenin secretion
Most conditionscausesecretion of ADH
the factorscausesecretion of ADH
by GLP-1(passive) caused byglucagon secretion
by an incretin effect(passive) is ... influenced byglucagon secretion
an increase incausesglucagon secretion
concentrationcausesglucagon secretion
the brain or pulmonary diseasescauseadh secretion
the DIET1 gene in humansinfluencesFGF19 secretion
insulin secretion and fatscauseglucagon secretion
by epinephrine infusion(passive) caused byglucagon secretion
factorsinfluencingchemokine secretion
the process of calcium releasecausessecretion
adrenergic regulation of the sympathetic nervous system and the hypothalamic - pituitary - adrenal axis ( 1(passive) is influenced bysecretion
Activation of these receptorscould influencesecretion
Sympathetic secretion of lacrimal glandscausessecretion
peripheral vasoconstriction and water retentioncausesperipheral vasoconstriction and water retention
in sodium loss and potassium retentionresultsin sodium loss and potassium retention
increased sodium reabsorption and therefore water reabsorptioncausesincreased sodium reabsorption and therefore water reabsorption
to a reduction in the release of glucose from the liverleadingto a reduction in the release of glucose from the liver
sodium to be retained in the body by the kidneyscausessodium to be retained in the body by the kidneys
renal absorption of sodiumcausesrenal absorption of sodium
an increase in serum potassiumcausesan increase in serum potassium
sodium retentioncausessodium retention
in excess sodium and fluid retentionresultingin excess sodium and fluid retention
increased distal tubule K+ secretioncausesincreased distal tubule K+ secretion
water and salt retentioncauseswater and salt retention
to reduced hepatic glucose outputleadsto reduced hepatic glucose output
sodium ion and water retention resulting in an increase in blood volume and pressurecausessodium ion and water retention resulting in an increase in blood volume and pressure
sodium and water retentioncausessodium and water retention
to sodium and water retentionleadsto sodium and water retention
sodium ion reabsorption from the distal tubulecausessodium ion reabsorption from the distal tubule
sodium and fluid retentioncausessodium and fluid retention
to excess hepatic glucose productionleadingto excess hepatic glucose production
to reduced endogenous glucose productionleadingto reduced endogenous glucose production
sodium and fluid retention , which further increases intravascular volumecausessodium and fluid retention , which further increases intravascular volume
retention of Na+ and Watercausesretention of Na+ and Water
sodium to becausessodium to be
sodium or water retention at collecting ductcausessodium or water retention at collecting duct
sodium to be reabsorbed in the distal renal tubule in exchange for potassium and hydrogen ionscausessodium to be reabsorbed in the distal renal tubule in exchange for potassium and hydrogen ions
in small increases in serum potassiummay resultin small increases in serum potassium
in excess sodium foodresultingin excess sodium food
to salt and water retentionleadsto salt and water retention
in renal tubular damagecan resultin renal tubular damage
increased distal tubule K+ secretion ... causing the hypokalemia seen with contraction alkalosiscausesincreased distal tubule K+ secretion ... causing the hypokalemia seen with contraction alkalosis
the kidney to retain water and sodiumcausesthe kidney to retain water and sodium
increased distal tubule K+ secretion ... in turn causing the hypokalemia seen with contraction alkalosiscausesincreased distal tubule K+ secretion ... in turn causing the hypokalemia seen with contraction alkalosis
to decreased salt & water retention leads to decreased preload decreasedleadsto decreased salt & water retention leads to decreased preload decreased
to vascular damageleadsto vascular damage
to secretion of too much parathyroid hormone - > HYPERCALCAEMIA ( high serumleadingto secretion of too much parathyroid hormone - > HYPERCALCAEMIA ( high serum
to increased hepatic gluconeogenesisleadsto increased hepatic gluconeogenesis
the salt and water retension increase the plasma volume and increase the preloadcausesthe salt and water retension increase the plasma volume and increase the preload
to hyperaldosteronismleadingto hyperaldosteronism
to patients with hypotensionleadsto patients with hypotension
the renin angiotensin systeminfluencedthe renin angiotensin system
to severe stomachleadsto severe stomach