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The extracellular immune complexescauseactivation of the complement system
the presence of immune complexes(passive) triggered bycomplement system activation
antigen - antibody complexes ( the classical pathway ) or a normal slow activation(passive) may be triggered byActivation of the complement system
which in most cases presented with an antigenemia and deposition of Ig or immune complexes ,causingactivation of the complement system
initiators of complement pathways ( i.e. , molecules ... directly or indirectlytriggersthe activation of complement system
The presence of protein aggregatescan also provokecomplement system activation
a multiplicity of “ danger signals , ” such as pathogen - associated molecular patterns ( PAMPs ) , antigen / antibody complexes , and the presence of transformed cells , apoptotic cells , or cell debris(passive) is triggered byActivation of the complement system
These serveto triggeractivation of the complement system
The tau proteincan influenceactivation of complement systems
In this model , ALI is induced by intrapulmonary immune complex deposition ,causingactivation of the complement system
The acidosiscausesactivation of complement systems
protease inhibitorscan influenceactivation of the complement system
intramolecular signalleadingto complement system activation
antigen - bound Abs(passive) is triggered byActivation of the complement system
several proteins such as albumin , fibronectin , fibrinogen , and complementleadto complement system activation
in different ways(passive) can be triggeredActivation of the complement system
Macmillan Magazines Ltd. Bacterial pathogenspreventactivation of the complement system
C1s complex and causes it to dissociate from C1qthus preventingactivation of the complement system
how two proteins — CFH and FHL-1 — might be regulated in the retinato preventcomplement - system activation
the presence of 10 mM ethylene glycol tetraacetic acidto preventactivation of the complement system
heavy parasitemiacausingactivation of the complement system
RCM infusionsresultin complement system activation
when they pass through the cooler parts of the peripheral circulationultimately causingactivation of the complement system
Most CCPspreventactivation of the complement system
using drugsto preventactivation of the complement system
tissuescausingactivation of the complement system
Full - text from Publisher 's SitecontributesComplement system activation
Excess of porphyrinscauseactivation of the complement system
The accumulation of porphyrinscausesactivation of the complement system
mediatorsresultingfrom complement system activation
bacteria ... host C4-binding protein ( C4BPpreventsactivation of the complement system
both sepsis and traumamay causeactivation of the complement system
to local inflammation , tissue damage and upregulation of angiogenic factors such as vascular endothelial growth factor ( VEGFcould leadto local inflammation , tissue damage and upregulation of angiogenic factors such as vascular endothelial growth factor ( VEGF
to a variety of responses including increased vascular permeability , chemotaxis of phagocytic cells , activation of inflammatory cells , opsonization of foreign particles , direct killing of cells and tissue damageleadsto a variety of responses including increased vascular permeability , chemotaxis of phagocytic cells , activation of inflammatory cells , opsonization of foreign particles , direct killing of cells and tissue damage
in several effects that assist in defense of the body ( Fig . 22.5resultsin several effects that assist in defense of the body ( Fig . 22.5
to enhanced local inflammationleadingto enhanced local inflammation
to generation of antimicrobial compounds , release of anaphylatoxinsleadsto generation of antimicrobial compounds , release of anaphylatoxins
tumor cell destructioncausestumor cell destruction
the antigen Lysis (passive) Caused bythe antigen Lysis
to intravascular hemolysis , spontaneous hemorrhageleadsto intravascular hemolysis , spontaneous hemorrhage
to the ependymal damage induced by microbial neuraminidase | Springer for Research & Development December 2016contributesto the ependymal damage induced by microbial neuraminidase | Springer for Research & Development December 2016
to the ependymal damage induced by microbial neuraminidase | Journal of Neuroinflammation | Full Text Complement system activationcontributesto the ependymal damage induced by microbial neuraminidase | Journal of Neuroinflammation | Full Text Complement system activation
a wide range of cellular responses ranging from apoptosis to opsonization ( 27triggersa wide range of cellular responses ranging from apoptosis to opsonization ( 27
in the formation of the membrane attack complex which causes membrane pore development and subsequent cell lysisresultsin the formation of the membrane attack complex which causes membrane pore development and subsequent cell lysis
to the formation of the membrane attack complex ( MAC ) , which directly disrupts targeted pathogens via cell lysisultimately leadsto the formation of the membrane attack complex ( MAC ) , which directly disrupts targeted pathogens via cell lysis
in explosion / lysis of the bacterial cellsresultsin explosion / lysis of the bacterial cells
to local inflammation , tissue damage , and the resulting blood vessel growthcan leadto local inflammation , tissue damage , and the resulting blood vessel growth
to the formation of the MAC , which in turn causes lysisleadsto the formation of the MAC , which in turn causes lysis
to inflammation , enhanced phagocytosis , and direct lysis of some bacterialeadsto inflammation , enhanced phagocytosis , and direct lysis of some bacteria
in opsonization of pathogens , recruitment of phagocytes , and lysis of bacterial cellsresultsin opsonization of pathogens , recruitment of phagocytes , and lysis of bacterial cells
to local inflammation , tissue damage ( as in dry AMD ) and the resulting blood vessel growth ( angiogenesis in wet AMDcan leadto local inflammation , tissue damage ( as in dry AMD ) and the resulting blood vessel growth ( angiogenesis in wet AMD
any number of events such as opsonization , chemotaxis , inflammation , and lysis ... breaking ) of cellscan triggerany number of events such as opsonization , chemotaxis , inflammation , and lysis ... breaking ) of cells
lysis of foreign cells and activation and migration of nonspecific inflammation cellsthus causeslysis of foreign cells and activation and migration of nonspecific inflammation cells
in a cascade of biochemical reactions that ultimately ends in lysis and disruption of foreign or effete cellsresultsin a cascade of biochemical reactions that ultimately ends in lysis and disruption of foreign or effete cells
to the formation of membrane attack complex ( MAC ) that forms channel on the bacterial membrane , causing bacterial lysisleadsto the formation of membrane attack complex ( MAC ) that forms channel on the bacterial membrane , causing bacterial lysis
in a wide range of reactions such as lysis of various kinds of cells , bacteria and protozoa , inactivation of viruses , and the direct mediation of inflammatory processescan resultin a wide range of reactions such as lysis of various kinds of cells , bacteria and protozoa , inactivation of viruses , and the direct mediation of inflammatory processes
in a proteolytic cascade eventually forming the membrane attack complex ( MAC ) leading to cell membrane perforation , lysis , and the dissolution of cellular contents , andresultsin a proteolytic cascade eventually forming the membrane attack complex ( MAC ) leading to cell membrane perforation , lysis , and the dissolution of cellular contents , and
a sequence of biochemical reactions in which one component activates another component in a cascade fashiontriggersa sequence of biochemical reactions in which one component activates another component in a cascade fashion
an enzyme cascade , which activates further functions of the immune system and boosts the non - specific immune responsetriggersan enzyme cascade , which activates further functions of the immune system and boosts the non - specific immune response
to AD and AMD not by cytolysis but by the gradual elimination and accumulation of cellular debris from cells where the complement system is sub - lethally activatedleadsto AD and AMD not by cytolysis but by the gradual elimination and accumulation of cellular debris from cells where the complement system is sub - lethally activated
to actiavtion of C3a and C5a mediatorsleadsto actiavtion of C3a and C5a mediators
to the formation of the membrane attack complex possibly on rod photoreceptorsmay leadto the formation of the membrane attack complex possibly on rod photoreceptors
in : opsonic functionresultsin : opsonic function
to activation of the clotting and fibrinolytic systems ( 79 ) , resulting in activation of several clotting factors , including thrombin , which have C3 and C5 convertase activitiesleadsto activation of the clotting and fibrinolytic systems ( 79 ) , resulting in activation of several clotting factors , including thrombin , which have C3 and C5 convertase activities
in opsonization of the target and its interaction with cells of the RESresultsin opsonization of the target and its interaction with cells of the RES
to the release of pro - inflammatory peptidesleadsto the release of pro - inflammatory peptides
to the dopaminergic neuron damage [ 53may contributeto the dopaminergic neuron damage [ 53
to neuronal damage and deathcan leadto neuronal damage and death
to survivalcontributesto survival
in the elimination of pathogensresultsin the elimination of pathogens
to a cascade of reactions occurring on the surface of pathogensleadsto a cascade of reactions occurring on the surface of pathogens
to the formation of either soluble ( sC5b-9 ) orleadsto the formation of either soluble ( sC5b-9 ) or