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It is mediated by the binding of XNAs ( xenoreactive natural antibodies ) to the donor endotheliumcausingactivation of the human [ [ complement system
P. falciparum infectionleadsto extreme complement system activation
an integration site within the complement C2 genecould leadto mis - activation of the complement system
various genetic mutationscausinguncontrolled activation of the complement system
Its main function is thoughtto resultin the activation of the complement system
main biochemical pathways ... : the classical complement binding pathwayresultin activation of the complement system
by an immune complex(passive) caused bythe activation of the complement system
The contact of blood with the membranecan resultin activation of the complement system
conditionscausingactivation of the complement system
in turncan leadto complement system activation
complement genesresultover activation of the complement system
The processing of blood to produce transfusion productscan leadto the activation of the complement system
The antigen - antibody complexleadsto activation of complement system
Recognition of foreign or altered structures in the body by some of the soluble pattern recognition receptorsmay leadto activation of the complement system
believedto resultfrom activation of the complement system
The lattercan leadto activation of complement system
The extracellular immune complexescauseactivation of the complement system
that inhibition of VEGF with anti - VEGF agentsmay contributeto complement system activation
inhibition of VEGF with anti - VEGF agentsmay contributeto complement system activation
Anti - DNA antibodies or immune complexes which contain these antibodies ... the kidneyresultsin activation of the complement system
three main biochemical pathwaysresultin activation of the complement system
an acute inflammatory responsemostly resultingfrom activation of the complement system
C5leadsto over activation of the complement system
It triggers an acute inflammatory responsemostly resultingfrom activation of the complement system
allograft injuryresultingfrom activation of the complement system
aHUS carry mutations in complement genesresultin over activation of the complement system
The IgM and IgG then bind to the tissueleadingto activation of the complement system
IgG. The IgM and IgG then bind to the tissueleadingto activation of the complement system
three branches ... : the classical complement pathway , the alternative complement pathway , and the mannose - binding lectin pathwayleadto activation of the complement system
vessel wallsleadingto activation of the complement system
protease inhibitorscan influenceactivation of the complement system
immune complexes deposit in vessel wallsleadingto activation of the complement system
immunization or ischemia reperfusion injuryresultingin activation of the complement system
In latent KSHV - infected endothelial cells , some complement regulatory proteins are downregulatedresultingin activation of the complement system
Igg igm antibodies and complement cell bound host cells with foreign antigen drug actingcausesone complement activation
may be found located in vessel walls and organs ( eg . glomerulonephritisleadsto activation of the complement system
that anti - PEG IgM is produced by spleen cells in response to an injected dose of blank pegylated liposomes , which bind selectively to PEG present at the liposome surfacecausingthe activation of the complement system
Immune complexes as well as autoantibodies bound to antigens on cellscausecomplement activation
Antigen - antibody immune complex depositioncausingcomplement activation
Addition of patient serum containing mutant factor H to control plateletsresultedin complement activation
in lysisresultsin lysis
in opsonization of virus by complementresultsin opsonization of virus by complement
in endothelial damage , inflammation , thrombosis and necrosis of the transplantresultsin endothelial damage , inflammation , thrombosis and necrosis of the transplant
in opsonization of virusresultsin opsonization of virus
in formation of the membrane attack complex ( MAC ) on the membranes of target cellsresultsin formation of the membrane attack complex ( MAC ) on the membranes of target cells
in several effects that assist in defense of the bodyresultsin several effects that assist in defense of the body
to membrane attack complex ( MAC ) deposition on blood vessels and subsequent necrosisleadsto membrane attack complex ( MAC ) deposition on blood vessels and subsequent necrosis
to local inflammationcould leadto local inflammation
to local inflammationcan leadto local inflammation
to the formation of the MAC , which in turn causes lysis , and death of target cellsleadsto the formation of the MAC , which in turn causes lysis , and death of target cells
to the formation of the membrane attack complex that consequently lyses cellseventually leadsto the formation of the membrane attack complex that consequently lyses cells
to the formationcan leadto the formation
in cell lysisresultsin cell lysis
cell lysisresultscell lysis
to the formation of molecules with proinflammatory propertiesleadsto the formation of molecules with proinflammatory properties
to a number of mechanismsleadsto a number of mechanisms
to local inflammation , tissue damagecan leadto local inflammation , tissue damage
to initiation of important effects which include 1leadto initiation of important effects which include 1
in formation of membrane attack complexes ( MACsresultsin formation of membrane attack complexes ( MACs
in formation of membrane attack complexes ( MACsresultsin formation of membrane attack complexes ( MACs
to the formation of the membrane attack complex possibly on rod photoreceptorsmay leadto the formation of the membrane attack complex possibly on rod photoreceptors
the Lysiscausethe Lysis
reactions(passive) caused byreactions
in a cascade of interactions of these proteinsresultsin a cascade of interactions of these proteins
in generation of C5aresultsin generation of C5a
in the elimination of pathogensresultsin the elimination of pathogens
to the release of pro - inflammatory peptidesleadsto the release of pro - inflammatory peptides
to the pathogenesis of AMDmay ... contributeto the pathogenesis of AMD
in opsonizationresultsin opsonization
to cleavage of C3leadsto cleavage of C3
a cascade of reactions that assist in the immune responsecausesa cascade of reactions that assist in the immune response
chemotaxis whichleadschemotaxis which
chemotaxisleadschemotaxis
to the formation of the MAC , which in turn causes lysisleadsto the formation of the MAC , which in turn causes lysis
to intravascular hemolysisleadsto intravascular hemolysis
to four principal outcomesleadsto four principal outcomes
to opsonization of microorganisms , lysis of target cells , and release of inflammatory mediators from leukocytesleadsto opsonization of microorganisms , lysis of target cells , and release of inflammatory mediators from leukocytes
to generation of antimicrobial compounds , release of anaphylatoxinsleadsto generation of antimicrobial compounds , release of anaphylatoxins
in red blood cell lysis.[3resultsin red blood cell lysis.[3
to the cleavage of : C3 42leadsto the cleavage of : C3 42