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Our resulting chemical mechanisms ... other atmospheric physical variablesinfluencethe formation of secondary organic aerosols from aromatics
their abilityto contributeto atmospheric formation of secondary organic aerosol ( SOA
overexpression of heme - oxygenasecan preventreperfusion - mediated generation of reactive oxygen species
catalyzing O2 into H2Oto preventformation of reactive oxygen species upon exposure to oxidant
plantscausingphytotoxicity by the formation of reactive oxygen species
Progressive loss of dopaminergic neurons induced by unilateral rotenone infusion into the medial forebrain bundle Anwar Norazit , Adrian Meedeniya , Maria Nga Nguyen , Alan Mackay - Sim Rotenone , a mitochondrial complex 1 inhibitorcausesoxidative damage via production of reactive oxygen species
High glucosecausesexcess mitochondrial reactive oxygen species formation
allopurinolpreventsischemia - reperfusion generation of reactive oxygen species
Dexrazoxane is a prodrug analog of the metal chelator EDTA that likely acts by removing iron from the iron - anthracycline complexthus preventingformation of damaging reactive oxygen species
coincidental with reperfusionto preventdamage to myocardium from formation of reactive oxygen species
to large increases in viscosity ... and may be at least partially responsible for the high viscosities observed in some SOAcould leadto large increases in viscosity ... and may be at least partially responsible for the high viscosities observed in some SOA
the number of climatically relevant particles in the atmosphereinfluencesthe number of climatically relevant particles in the atmosphere
volatile species that are pumped awaycreatingvolatile species that are pumped away
to its genotoxicitycould ... contributeto its genotoxicity
further muscle damage , muscle edemacausesfurther muscle damage , muscle edema
cellular damagecan causecellular damage
to irreversible cell damage and an overall functional decline ( Harman , 1956leadsto irreversible cell damage and an overall functional decline ( Harman , 1956
bilayer damage(passive) caused bybilayer damage
to the pathophysiology of inflammationcontributeto the pathophysiology of inflammation
in bladder voiding dysfunctionresultsin bladder voiding dysfunction