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The increase in Ca2 +also contributesto the activation of phospholipase A2 ( PLA2 ) , which release the precursor arachidonic acid that is converted into the prostaglandins and leukotrienes ( Module 1
These increasespossibly resultfrom hypoxia - induced activation of cPLA2 and subsequent release of arachidonic acid and its conversion to prostaglandins
epigenetic modificationscan resultin the activation and overexpression of pro - drug resistance genes.[2
epigenetic modificationscan resultin the activation and overexpression of pro - drug resistance genes.[12
Activation of MAPK by MEKmay resultin the activation of cytosolic phospholipase A2 ( cPLA2 ) and subsequent production of arachidonic acid and prostaglandins
JNK Crizotinib molecular weight activationcontributesto gallic acid elicited p53 activation and apoptosis induction
PLD2 in antigen - stimulated mast cellsleadingto PLD2 activation and the production of the signaling molecules lysophosphatidic acid and diacylglycerol
Phosphorylates PLD2 in antigen - stimulated mast cellsleadingto PLD2 activation and the production of the signaling molecules lysophosphatidic acid and diacylglycerol
the CNTs introduction(passive) are also influenced byThe adsorption and activation of hydrogen on this particular hybrid catalyst
chronic alcohol exposureleadsto activation of Ca2 + -dependent and arachidonic acid - specific PLA2
C5a level ( C5aRleadingin a inactive histidine of the TLR and variant molecular acid activation to particle
Minor endothelial injurymay causeplatel adhesion and activation with the resultant release of PDGF
mechanismsleadingto its activation and release of arachidonic acid ( AA
parenchymal microglia and astro- cytesresultin up - regulation of adhesion molecules , activation of metalloproteinases and catabolism of arachidonic acid
CCL2 and CXCL1causedthe activation of cul tured microglia and stimulated the production of proinflammatory molecules
SSAO oxidative activity stimulates glucose transport via the translocation of GLUT4 carriers to the cell surfaceresultingfrom a potent tyrosine phosphorylation of IRS-1 and IRS-3 and phosphoinositide 3-kinase activation
the release of chemokines MCP-1 and KC , and anotherleadsto the release of uric acid and the activation of the NALP3 inflammasome
The ischemia and especially postischemic reperfusionledto activation of acid hydrolases in sarcoplasmic reticulum and of lipid peroxidation
A highly enantioselective direct Mannich - type reaction of aldimines with dialkyl malonates ... a Mg(II)-BINOLate saltwas designedas a cooperative acid - base catalyst that can activate both aldimines and malonates
interactions between the prometastatic scaffolding protein HEF1 / Cas - L / NEDD9 and the oncogenic Aurora A ( AurA ) kinase at the basal body of ciliacausesphosphorylation and activation of HDAC6 , a tubulin deacetylase ... promoting ciliary disassembly
These research resultshave leadto the incorporation of especially Caprylic , Capric and Lauric Acid
that interactions between the prometastatic scaffolding protein HEF1 / Cas - L / NEDD9 and the oncogenic Aurora A ( AurA ) kinase at the basal body of ciliacausesphosphorylation and activation of HDAC6 ... promoting ciliary disassembly
interactions between the prometastatic scaffolding protein HEF1 / Cas - L / NEDD9 and the oncogenic Aurora A ( AurA ) kinase at the basal body of ciliacausesphosphorylation and activation of HDAC6 ... promoting ciliary disassembly
Peripheral nitric oxidecontributesto both formalin- and NMDA - induced activation of nociceptors
to reduction of muscle size ... 27might leadto reduction of muscle size ... 27
to apoptosis [ 8leadingto apoptosis [ 8
in apoptosisresultsin apoptosis
to beta - arrestin phosphorylation and internalizationleadingto beta - arrestin phosphorylation and internalization
to beta - arrestin phosphorylation and internalizationleadingto beta - arrestin phosphorylation and internalization
to an increase in corticosteroid outputleadsto an increase in corticosteroid output
to apoptotic cell deathultimately leadingto apoptotic cell death
to apoptotic cell demiseleadsto apoptotic cell demise
to coughmay contributeto cough
to apoptosis ( 21leadingto apoptosis ( 21
increases in renal vascular resistance which in turn shifts renal pressure natriuresis to the right causing hypertensionwould causeincreases in renal vascular resistance which in turn shifts renal pressure natriuresis to the right causing hypertension
to beta - arrestin phosphorylation and internalizationleadingto beta - arrestin phosphorylation and internalization
to beta - arrestin phosphorylation and internalizationleadingto beta - arrestin phosphorylation and internalization
to beta-leadingto beta-
capillary permeability , vasodilation , and hypotensioncan causecapillary permeability , vasodilation , and hypotension
to apoptotic cell deathultimately leadingto apoptotic cell death
apoptosis and reduction in insulin synthesis and secretionthereby causingapoptosis and reduction in insulin synthesis and secretion
to beta - arrestin phosphorylation and internalizationleadingto beta - arrestin phosphorylation and internalization
to the metastatic behavior of human breast cancer cellsmay contributeto the metastatic behavior of human breast cancer cells
to apoptosisleadsto apoptosis
programmed cell deathto causeprogrammed cell death
a significant increase in COX-2 promoter activitycausesa significant increase in COX-2 promoter activity
leukocytecausesleukocyte
to beta-leadingto beta-
to IFN - I productionleadingto IFN - I production
to the recruitment of the Apoptosis Regulator FADDleadsto the recruitment of the Apoptosis Regulator FADD
to N - OH-3-ABAleadingto N - OH-3-ABA
polarization toward a T helper type 2 immune response via the release of IL-25 and IL-33may causepolarization toward a T helper type 2 immune response via the release of IL-25 and IL-33
insteadcould ... contributeinstead
to development of many interesting inhibitors against HDAC , human IMPDH , and T. congolense IMPDHledto development of many interesting inhibitors against HDAC , human IMPDH , and T. congolense IMPDH