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a course ... to helpseta reciprocal course
The BCR / ABL fusion proteinresultsfrom the reciprocal chromosomal translocation t(9;22
CML ... the Philadelphia ( Ph ) chromosomeresultsfrom the reciprocal chromosomal translocation t(9;22
the Philadelphia ( Ph ) chromosomeresultsfrom the reciprocal chromosomal translocation t(9;22
the translocation chromosomesmight be resultedfrom a reciprocal translocation event
the BCR / ABL1 fusion generesultsfrom the reciprocal translocation t(9;22)(q34;q11.2
CML ... the BCR / ABL1 fusion generesultsfrom the reciprocal translocation t(9;22)(q34;q11.2
The Philadelphia chromosomeresultsfrom the reciprocal translocation , t(9;22)(q34;q11.2
CML ... the bcr - abl fusion generesultsfrom the reciprocal translocation t(9;22)(q34;q11
The truncated chromosome 22resultsfrom the reciprocal translocation t(9;22)(q34;q11
who possessed a novel karyotyperesultingfrom a reciprocal balanced chromosomal translocation , t(2;7)(p13;p12
the sigma bladedoes ... causea reciprocal effect
Recent discussion of innovations in military weapons systems , strategies , and technologyprompteda reciprocal question
The molecular signature of chronic myeloid leukemia ( CML ) is the BCR - ABL fusion geneoriginatingfrom a reciprocal t(9;22
the tubular structure against the stabilizercausinga reciprocal movement
the stabilizercausinga reciprocal movement
The BCR - ABL tyrosine kinase oncogeneresultsfrom a reciprocal t(9;22
CML ... the Philadelphia ( Ph ) chromosomeresultsfrom the reciprocal chromosomal translocation
to exist between parental support and symptoms of depression(passive) was discoveredA reciprocal relationship
a US decision to do the same showing solidarity with the UK over alleged Moscow 's involvement in the poisoning of former Russian intelligence officer Sergei Skripal and his daughter(passive) prompted bya reciprocal move
different chromosomesresultingin balanced reciprocal translocation
the individual and their environmentinfluenceeach other in a reciprocal manner
fusion genesresultingfrom reciprocal chromosomal translocations
to contactto seta reciprocal link with
contactto seta reciprocal link with
the owner askingto seta reciprocal link
Abnormalitiesresultingfrom an unbalanced reciprocal translocation
therefore carries the risk the DNA ends that were not adjacent previously may be joinedcausinga chromosomal translocation
which can be donewill discovera reciprocal arrangement
e chromosomesresultingfrom the reciprocal translocation
molecular eventsmay leadto a reciprocal translocation
by breaks(passive) caused byreciprocal translocation
a genetic rearrangementresultingfrom reciprocal translocation
The origin of the chromosomal materialcontributedto the reciprocal translocation
More commonly , there is a two - way exchange of segments between the chromosomesresultingin a reciprocal translocation
genotypes including two pure - lines ( different subspecies , japonica and indica ) , a pair of reciprocal F1 hybrids parented by the two pure - lines , andresulteda pair of reciprocal tetraploids
one DSBcan causea translocation
later users as well as vice versa(passive) are influenced byreciprocal interdependence
mutual tenderness and intimacyleadingto a reciprocal avowal
the receive patternresultingfrom a reciprocal
to the fusion of the Abelson murine leukemia ( ABL ) gene on chromosome 9 with the breakpoint cluster region ( BCR ) gene on chromosome 22leadingto the fusion of the Abelson murine leukemia ( ABL ) gene on chromosome 9 with the breakpoint cluster region ( BCR ) gene on chromosome 22
in a fusion of the EWS gene to the Fli1 generesultsin a fusion of the EWS gene to the Fli1 gene
in in - frame fusion transcripts with various partner genesresultsin in - frame fusion transcripts with various partner genes
in the formation of Bcr - Abl generesultsin the formation of Bcr - Abl gene
in a gene fusion product between the RNA binding protein Ews and the transcription factor Fli1[17 , 18resultingin a gene fusion product between the RNA binding protein Ews and the transcription factor Fli1[17 , 18
to the fusion of the SS18 ( SYT ) gene to one of three SSX genes ( SSX1 , SSX2 or SSX4leadsto the fusion of the SS18 ( SYT ) gene to one of three SSX genes ( SSX1 , SSX2 or SSX4
hematopoietic stem cells(passive) caused byhematopoietic stem cells
in fusion of the Ewing sarcoma 1 ( EWS1 ) gene on chromosome 22 and WT1 on chromosome 11resultingin fusion of the Ewing sarcoma 1 ( EWS1 ) gene on chromosome 22 and WT1 on chromosome 11
one kind of leukemia a ) Uncontrolled divisions of largecauseone kind of leukemia a ) Uncontrolled divisions of large
CML ... in which a piece of chromosome 9 containing the ABL1 oncogene is translocated to chromosome 22 and fused to the BCR gene(passive) is caused byCML ... in which a piece of chromosome 9 containing the ABL1 oncogene is translocated to chromosome 22 and fused to the BCR gene
in overexpression of the CYCLIN D1 gene ( GENES , BCL-1).Lymphoma , T - Cell , Peripheralresultingin overexpression of the CYCLIN D1 gene ( GENES , BCL-1).Lymphoma , T - Cell , Peripheral
in overexpression of the CYCLIN D1 gene ( GENES , BCL-1).Image Cytometryresultingin overexpression of the CYCLIN D1 gene ( GENES , BCL-1).Image Cytometry
to the fusion of the SS18 ( SYT ) gene to one of three SSX genes ( SSX1 , SSX2 or SSX4leadsto the fusion of the SS18 ( SYT ) gene to one of three SSX genes ( SSX1 , SSX2 or SSX4
in the formation of Bcr - Abl generesultsin the formation of Bcr - Abl gene
to ETV6-NTRK3 fusion productleadsto ETV6-NTRK3 fusion product
in a fusion of the PML protein to the retinoic acid receptor resultingin a fusion of the PML protein to the retinoic acid receptor
to duplication of chromosomal regionsleadingto duplication of chromosomal regions
in an in - frame fusion with the retinoic acid receptor alpha ( RARA ) generesultedin an in - frame fusion with the retinoic acid receptor alpha ( RARA ) gene
Ewing 's sarcoma(passive) is caused byEwing 's sarcoma
to the generation of EWS - ETSleadingto the generation of EWS - ETS
The transformation to CML(passive) is caused byThe transformation to CML
the introduction of an oncogene through either the activation of a proto - oncogene or the introduction of a fusion genemay causethe introduction of an oncogene through either the activation of a proto - oncogene or the introduction of a fusion gene
to fusion of the BCR / ABL oncogenesleadingto fusion of the BCR / ABL oncogenes
in the expression of a TrkC fusion proteinresultsin the expression of a TrkC fusion protein
in ETV6-NTRK3 gene fusionresultsin ETV6-NTRK3 gene fusion
to the fusion geneleadingto the fusion gene
in fusion of the anaplastic lymphoma kinase ( ALK ) to nucleophosmin ( NPMcould resultin fusion of the anaplastic lymphoma kinase ( ALK ) to nucleophosmin ( NPM
in the fusion protein TLSresultsin the fusion protein TLS
in an in - frame fusion oncogene between the N - terminus of MLLresultsin an in - frame fusion oncogene between the N - terminus of MLL
a fusion of the C8orf34 genecausinga fusion of the C8orf34 gene
in BCR JAK2 fusionresultingin BCR JAK2 fusion
disruption or activation of genescausesdisruption or activation of genes
a fusion of the C8orf34 gene with the MET protooncogenecausinga fusion of the C8orf34 gene with the MET protooncogene
3 - 4 % of the cases of Down Syndrome(passive) are caused by3 - 4 % of the cases of Down Syndrome
to leukemia , or cancer of the blood cellsleadsto leukemia , or cancer of the blood cells
partial T18causespartial T18
in a 1would resultin a 1
in a 1resultin a 1
probablycontributesprobably
in their deletion ( 13resultingin their deletion ( 13